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pro vyhledávání: '"P. G. Natali"'
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Autor:
Luigi Maria Larocca, Mauro Piantelli, G. Castrilli, Nicola Maggiano, P. G. Natali, F. Savini, F. O. Ranelletti, D. Tatone
Publikováno v:
Melanoma Research. 11:469-476
Hyperthermia produces regression of human cancer. Because hyperthermia has produced only limited results, attention has focused on searching for substances able to sensitize tumour cells to the effects of hyperthermia. The flavonoid quercetin has bee
Autor:
P. G. Natali, Giovanni Scambia, Anna Fagotti, Gabriella Ferrandina, Francesco Maneschi, A De Pasqua, Pierluigi Benedetti-Panici, Maria Giovanna Salerno, M Mottolese, Salvatore Mancuso
Publikováno v:
British Journal of Cancer
The aim of this study was to assess the association of p53 status with primary cytoreduction, response to chemotherapy and outcome in stage III–IV primary ovarian cancer patients. Immunohistochemical analysis of p53 was performed on formalin-fixed,
Autor:
Annamaria Biroccio, Francesca Spinella, Erica Salvati, Roberta Cianfrocca, V Di Castro, P. G. Natali, Anna Bagnato, Laura Rosanò, Francesca Spadaro, Piera Tocci
Despite the fundamental pathophysiological importance of β-catenin in tumor progression, the mechanism underlying its final transcriptional output has been partially elucidated. Here, we report that β-arrestin-1 (β-arr1) is an epigenetic regulator
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::99566c90c8c8632b2559a98c15379802
http://hdl.handle.net/11573/1132979
http://hdl.handle.net/11573/1132979
Publikováno v:
British Journal of Cancer
In vitro studies have demonstrated that fibronectin (FN) can deliver a mitogenic signal to quiescent human melanoma cells and that the alpha 5/beta 1-integrin receptor mediates this stimulus. In view of this finding we have analysed the in vivo expre
Autor:
Paola Nisticò, PG De Berardinis, C. Buono, T Alonzi, I Venturo, P. G. Natali, Stefania Morrone
Publikováno v:
Journal of Clinical Investigation. 94:1426-1431
Cell-mediated immune response to breast tumor has only been marginally investigated. To gain insight into this issue we have developed two clones of distinct phenotype, CD3+ alpha/beta, CD4+, CD8-, CD16-, and CD3+ alpha/beta, CD4-, CD8+, CD16-, respe