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Autor:
P-Y, Scarabin
Publikováno v:
La Revue de medecine interne. 24(4)
Autor:
P, Marques-Vidal, M, Montaye, B, Haas, A, Bingham, A, Evans, I, Juhan-Vague, J, Ferrières, G, Luc, P, Amouyel, D, Arveiler, J, Yarnell, J B, Ruidavets, P Y, Scarabin, P, Ducimetière
Publikováno v:
Atherosclerosis. 157(2)
The relationships between alcoholic beverages and cardiovascular risk factors were assessed in 6730 men living in France or Northern Ireland. In France, all alcoholic beverages were significantly correlated with body mass index (BMI), systolic (SBP)
Autor:
P Y, Scarabin
Publikováno v:
La Revue de medecine interne. 21
Autor:
E, Oger, P Y, Scarabin
Publikováno v:
Annales d'endocrinologie. 60(3)
Hormone replacement therapy relieves climacteric symptom and prevents postmenopausal osteoporosis. A protective effect of estrogen against coronary heart disease remains debatable and inconclusive results have been reported with respect to the risk o
Publikováno v:
Thrombosis and haemostasis. 82(3)
Among users of low-dose oral contraceptives (OC), cardiovascular diseases occur mainly in smokers. The mechanisms by which OC and smoking increase the risk for arterial thrombotic risk have not been adequately explained. Epidemiological evidence sugg
Autor:
I, Behague, O, Poirier, V, Nicaud, A, Evans, D, Arveiler, G, Luc, J P, Cambou, P Y, Scarabin, L, Bara, F, Green, F, Cambien
Publikováno v:
Circulation. 93(3)
Polymorphisms of the beta fibrinogen gene have been shown to affect plasma fibrinogen levels and the risk of peripheral arterial disease. We now present the results of a detailed analysis of the beta fibrinogen gene in relation to plasma fibrinogen a
Publikováno v:
Thrombosis and haemostasis. 74(3)
Several studies have suggested a dose-response relation between the oestrogen content of oral contraceptive (OC) and the risk of both venous thrombosis and arterial disease, when oestrogen doses were higher than 50 micrograms. However, there is no cl
Publikováno v:
Thrombosis and haemostasis. 64(2)
Many case-control studies have suggested that increased platelet aggregation (PA) could be involved in the pathogenesis of diabetic microangiopathy. However, longitudinal data are needed to support this hypothesis. We consider here such an approach i
Akademický článek
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