Zobrazeno 1 - 10
of 28
pro vyhledávání: '"P J, Chowienczyk"'
Autor:
Emma Rezel-Potts, Abdel Douiri, Xiaohui Sun, Phillip J. Chowienczyk, Ajay M. Shah, Martin C. Gulliford
Publikováno v:
PLoS Medicine, Vol 19, Iss 7 (2022)
Background Acute Coronavirus Disease 2019 (COVID-19) has been associated with new-onset cardiovascular disease (CVD) and diabetes mellitus (DM), but it is not known whether COVID-19 has long-term impacts on cardiometabolic outcomes. This study aimed
Externí odkaz:
https://doaj.org/article/2fd31ec39ce84231afdfe55017e1439f
Autor:
Franca Morselli, Luca Faconti, Charlotte E Mills, Steve Morant, Philip J Chowienczyk, J Kennedy Cruickshank, Andrew J Webb, Alessandro Cavarape
Publikováno v:
Artery Research, Vol 26, Iss Supplement 1 (2020)
Background: Epidemiological and animal studies suggest the potential of dietary nitrate (NO 3−) to inhibit atherogenesis. Spironolactone may improve arterial stiffness. We tested if 6 months’ intervention with dietary nitrate and spironolactone c
Externí odkaz:
https://doaj.org/article/01b56ca579314f9baec26f47366b2e2b
Publikováno v:
Cardiovascular Ultrasound, Vol 16, Iss 1, Pp 1-7 (2018)
Abstract Background Interactions between the left ventricular (LV) and the arterial system, (ventricular-arterial coupling) are key determinants of cardiovascular function. However, most of studies covered multiple cardiovascular risk factors, which
Externí odkaz:
https://doaj.org/article/42175349944d4844af2da13bcc56033a
Publikováno v:
Artery Research, Vol 24 (2018)
Objective: To test if arterial stiffness (AS) can be modulated by the autonomic nervous system (ANS) independently of blood pressure (BP) in hypertensive patients. Material and methods: AS was measured by carotid-femoral pulse wave velocity (cfPWV)
Externí odkaz:
https://doaj.org/article/d59f21ff906142f698350e432df85922
Publikováno v:
Artery Research, Vol 24 (2018)
Objective: The autonomic nervous system (ANS) plays an important role in regulating blood pressure (BP), but its action on arterial stiffness (AS) is still debated. Here we examine if device-guided paced breathing (DGB) 1, via its action on ANS, can
Externí odkaz:
https://doaj.org/article/5daae0e1cc0c4f6583506909ab372e4f
Publikováno v:
Artery Research, Vol 12 (2015)
Background: Arterial calcification may lead to regional variation in distension imposing stresses on the arterial wall that predispose to plaque rupture. The objective of this study was to use a novel speckle tracking method to investigate regional m
Externí odkaz:
https://doaj.org/article/98da9668aca54be9915228b392e83651
Autor:
Michael V Holmes, Benyu Jiang, Karen McNeill, Melinda Wong, Stephen P Oakley, Bruce Kirkham, Phil J Chowienczyk
Publikováno v:
PLoS ONE, Vol 5, Iss 4, p e10242 (2010)
Within the general population, levels of C-reactive protein (CRP) are positively associated with atherosclerotic cardiovascular disease (CVD). Whether CRP is causally implicated in atherogenesis or is the results of atherosclerosis is disputed. A rol
Externí odkaz:
https://doaj.org/article/f18e8a06a48c43f380831dab0a7cf71c
Autor:
Ian B. Wilkinson, Carmel M. McEniery, Giuseppe Schillaci, Pierre Boutouyrie, Patrick Segers, Anne Donald, Philip J. Chowienczyk
Publikováno v:
Artery Research, Vol 4, Iss 2 (2010)
The last 15 years have seen an explosion of interest in large artery haemodynamics, and arterial stiffness in particular. A number of devices are now available to assess arterial stiffness, and although many are in routine use, they do not necessaril
Externí odkaz:
https://doaj.org/article/11ea4061e55245799cb070444780d00b
Autor:
M, Dawes, P J, Chowienczyk
Publikováno v:
Best practiceresearch. Clinical obstetricsgynaecology. 15(6)
Pharmacokinetics describes the handling of a drug by the body - how the drug is absorbed, distributed and eliminated and how these processes determine plasma concentrations of the drug. Changes in maternal physiology during pregnancy influence pharma
Publikováno v:
British journal of pharmacology. 134(5)
1. N(G)-monomethyl-L-arginine (L-NMMA) constricts human forearm resistance vasculature and selectively attenuates vasodilator responses to endothelium-dependent vasodilators. Incomplete inhibition of such responses could be due to an inadequate dose