Zobrazeno 1 - 10
of 34
pro vyhledávání: '"P Boerkmann"'
Publikováno v:
European Respiratory Journal. 5:810-814
Eight weeks after a single intravenous injection of trypsin, more than half of 26 treated rats showed pulmonary emphysema, as demonstrated by a significant increase of the mean linear intercept (MLI = 107 microns) in comparison with 11 controls (69 +
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f36f5f9ec403b97c70c200e4993a58cd
https://doi.org/10.1183/09031936.93.05070810
https://doi.org/10.1183/09031936.93.05070810
Publikováno v:
Comptes rendus de l'Academie des sciences. Serie III, Sciences de la vie. 319(5)
Injection of trypsin triggers the delayed appearance of a lung emphysema. Classically, emphysema is attributed to elastase, more particularly to leucocyte elastase. Indeed the acute phase of this experiment is characterized by a granulocyte sequestra
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::0e78f8822ba18ec39eeb308478bf60ed
https://pubmed.ncbi.nlm.nih.gov/8763737
https://pubmed.ncbi.nlm.nih.gov/8763737
Publikováno v:
Journal of Pathology
Journal of Pathology, Wiley, 1996, 178 (2), pp.215-220. ⟨10.1002/(sici)1096-9896(199602)178:23.0.co;2-k⟩
Journal of Pathology, Wiley, 1996, 178 (2), pp.215-220. ⟨10.1002/(sici)1096-9896(199602)178:23.0.co;2-k⟩
International audience; Intravenous injection of trypsin in the rat induces early lung leucostasis and emphysema of delayed onset. This report confirms that this emphysema is not rat-specific and that the leucostasis is not related to the presence of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1fb666d28dd87664d400d5ca5280ed5b
https://hal-ineris.archives-ouvertes.fr/ineris-03166482
https://hal-ineris.archives-ouvertes.fr/ineris-03166482
Publikováno v:
The Journal of pathology. 178(2)
Intravenous injection of trypsin in the rat induces early lung leucostasis and emphysema of delayed onset. This report confirms that this emphysema is not rat-specific and that the leucostasis is not related to the presence of contaminating endotoxin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::12e3e8e2d0a5ec1462f4012aa974e28b
https://pubmed.ncbi.nlm.nih.gov/8683392
https://pubmed.ncbi.nlm.nih.gov/8683392
Publikováno v:
Respiratory medicine. 88(9)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bd09f4a61a7eb0263b7452dd4c833f22
https://pubmed.ncbi.nlm.nih.gov/7809445
https://pubmed.ncbi.nlm.nih.gov/7809445
Autor:
E, Reichart, P, Boerkmann
Publikováno v:
Pathologie-biologie. 39(10)
Intrapleural administration of elastase was found to induce pulmonary emphysema identical to that seen after an endotracheal instillation of elastase in 33% of cats and 14% of rats given the treatment [15]. This experiment was repeated using hamsters
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::15fbfe898d60260daf675b3ff4cdf803
https://pubmed.ncbi.nlm.nih.gov/1805139
https://pubmed.ncbi.nlm.nih.gov/1805139
Publikováno v:
Comptes rendus de l'Academie des sciences. Serie III, Sciences de la vie. 310(13)
The injection of trypsin provokes a decrease of the circulating antiproteases. Its effect on an elastase-induced emphysema is negligible. Paradoxically, emphysema is quantitatively demonstrated in rats treated with trypsin alone: an influx of polymor
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::4de1e333b45c4ce004f9ab11292f2df6
https://pubmed.ncbi.nlm.nih.gov/2114197
https://pubmed.ncbi.nlm.nih.gov/2114197
Publikováno v:
Experimental Lung Research; 1992, Vol. 18 Issue: 1 p45-53, 9p
Publikováno v:
Pathologie-biologie. 35(4)
The endotracheal deposition of pancreatic porcine elastase (EPP) at 40 u X kg-1 body weight provokes a typical pulmonary emphysema (alveoli disruption) in rats. This emphysema is significant (p less than 0.001) when quantitatively compared to a place
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::8f78901475bd50b2cf7b0afff1c48355
https://pubmed.ncbi.nlm.nih.gov/3035467
https://pubmed.ncbi.nlm.nih.gov/3035467
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