Zobrazeno 1 - 10
of 199
pro vyhledávání: '"Oxygen/*pharmacology"'
Autor:
Marie-Jeanne Richard, Laurent Argaud, Sandrine Lablanche, Cécile Cottet-Rousselle, Thierry Berney, Pierre-Yves Benhamou, Camille Laporte, Eric Fontaine, Frédéric Lamarche
Publikováno v:
Biochimica et Biophysica Acta, Vol. 1847, No 6-7 (2015) pp. 629-639
BBA-Biochimica et Biophysica Acta
BBA-Biochimica et Biophysica Acta, Elsevier, 2015, 1847 (6-7), pp.629-39. ⟨10.1016/j.bbabio.2015.04.002⟩
BBA-Biochimica et Biophysica Acta
BBA-Biochimica et Biophysica Acta, Elsevier, 2015, 1847 (6-7), pp.629-39. ⟨10.1016/j.bbabio.2015.04.002⟩
International audience; Deficit in oxygen and energetic substrates delivery is a key factor in islet loss during islet transplantation. Permeability transition pore (PTP) is a mitochondrial channel involved in cell death. We have studied the respecti
Publikováno v:
Pires, I M, Bencokova, Z, McGurk, C & Hammond, E M 2010, ' Exposure to acute hypoxia induces a transient DNA damage response which includes Chk1 and TLK1 ', Cell cycle (Georgetown, Tex.), vol. 9, no. 13, pp. 2502-2507 . https://doi.org/10.4161/cc.9.13.12059
Severe hypoxia has been demonstrated to induce a replication arrest which is associated with decreased levels of nucleotides. Chk1 is rapidly phosphorylated in response to severe hypoxia and in turn deactivates TLK1 through phosphorylation. Loss of C
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7a8205d10c797ce9a84a3f68a84c5684
https://doi.org/10.4161/cc.9.13.12059
https://doi.org/10.4161/cc.9.13.12059
Publikováno v:
Cell and Tissue Research
Regenerative medicine-based approaches for the repair of damaged cartilage rely on the ability to propagate cells while promoting their chondrogenic potential. Thus, conditions for cell expansion should be optimized through careful environmental cont
Autor:
Michel Ovize, Delphine Baetz, L. Ferreras, M. Strina, C. Crola-Da-Silva, Camille Villedieu, Abdallah Gharib, Bruno Pillot, Muhammad Rizwan Alam, Hélène Thibault, Thomas Bochaton
Publikováno v:
Journal of Molecular and Cellular Cardiology
Journal of Molecular and Cellular Cardiology, Elsevier, 2015, 84, pp.61-9. ⟨10.1016/j.yjmcc.2015.03.017⟩
Journal of Molecular and Cellular Cardiology, Elsevier, 2015, 84, pp.61-69. ⟨10.1016/j.yjmcc.2015.03.017⟩
Journal of Molecular and Cellular Cardiology, Elsevier, 2015, 84, pp.61-9. ⟨10.1016/j.yjmcc.2015.03.017⟩
Journal of Molecular and Cellular Cardiology, Elsevier, 2015, 84, pp.61-69. ⟨10.1016/j.yjmcc.2015.03.017⟩
International audience; RATIONALE: How ischemic postconditioning can inhibit opening of the mitochondrial permeability transition pore (PTP) and subsequent cardiac myocytes death at reperfusion remains unknown. Recent studies have suggested that de-a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a582b704e0200515a77d42e876a455c7
https://hal.archives-ouvertes.fr/hal-01850547
https://hal.archives-ouvertes.fr/hal-01850547
Autor:
Changchun Chen, Fausto Arellano-Carbajal, Zoltan Soltesz, Patrick Laurent, Geoffrey M. Nelson, Emmanuel D. Levy, Mario de Bono
Publikováno v:
eLife
eLife, Vol 4 (2015)
eLife, 4
eLife, Vol 4 (2015)
eLife, 4
Brains organize behavior and physiology to optimize the response to threats or opportunities. We dissect how 21% O2, an indicator of surface exposure, reprograms C. elegans' global state, inducing sustained locomotory arousal and altering expression
Decomposition of endoperoxide containing molecules is an attractive approach for the delayed release of singlet oxygen under mild reaction conditions. Here we describe a new method for the adaptation of the corresponding decay times by controlling th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d5482d1efdfa57fb4f9172a35e5c09aa
https://epub.uni-regensburg.de/30686/
https://epub.uni-regensburg.de/30686/
Publikováno v:
American Journal of Physiology. Heart and Circulatory Physiology, Vol. 288, No 4 (2005) pp. H1611-H1619
Whereas previous studies have shown that opening of the mitochondrial ATP-sensitive K+ (mitoKATP) channel protects the adult heart against ischemia-reperfusion injury, it remains to be established whether this mechanism also operates in the developin
Autor:
Hoy, Andrew James.
Publikováno v:
Access electronically.
Thesis (M.Sc.)--University of Wollongong, 2004.
Typescript. Includes bibliographical references: leaf 101-126.
Typescript. Includes bibliographical references: leaf 101-126.
Externí odkaz:
http://ro.uow.edu.au/theses/232
Autor:
Rafael Selgas, Elia Aguado-Fraile, Ignacio Blanco-Sánchez, Manuel López-Cabrera, Belen Ponte, Laura Alegre, Angel Ordoñez, Ana Saiz, Edurne Ramos, David Sáenz-Morales, Fernando Liaño, Luis del Peso, Carlos Jiménez, Elisa Conde, José Antonio Sánchez-Tomero, Manuel O. Landázuri, María Laura García-Bermejo
Publikováno v:
PLoS ONE, Vol 7, Iss 3, p e33258 (2012)
Digital.CSIC. Repositorio Institucional del CSIC
instname
PLOS ONE, Vol. 7, No 3 (2012) P. e33258
Biblos-e Archivo. Repositorio Institucional de la UAM
PLoS ONE
Digital.CSIC. Repositorio Institucional del CSIC
instname
PLOS ONE, Vol. 7, No 3 (2012) P. e33258
Biblos-e Archivo. Repositorio Institucional de la UAM
PLoS ONE
This is an open-access article distributed under the terms of the Creative Commons Attribution License.-- et al.
Acute tubular necrosis (ATN) caused by ischemia/reperfusion (I/R) during renal transplantation delays allograft function. Identifica
Acute tubular necrosis (ATN) caused by ischemia/reperfusion (I/R) during renal transplantation delays allograft function. Identifica
Publikováno v:
Scopus-Elsevier
Using the human hepatoma cell line Hep G2, we have studied a possible role of protein kinase C (PKC) activity for regulation of erythropoietin (EPO) production. During a 72-h incubation, EPO production by the cells was stimulated sevenfold by exposur