Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Oskar, McWilliam"'
Publikováno v:
Ugeskrift for laeger. 184(50)
Introduction Motivation is important when administering cognitive tests. Routine cognitive testing may become trivial both for the examiner and the test subject when using tests that only incorporate neutral items. We hypothesized that a Christmas th
Autor:
Oskar McWilliam, Helle Bach Søndergaard, Marina Rode von Essen, Finn Sellebjerg, Annette Bang Oturai, Eva Rosa Petersen, Lars Börnsen, Cecilie Ammitzbøll, Rikke Ratzer, Jeppe Romme Christensen
Publikováno v:
Journal of Autoimmunity. 97:114-121
Smoking is a risk factor for the development and progression of multiple sclerosis (MS); however, the pathogenic effects of smoking are poorly understood. We studied the smoking-associated chemokine receptor-like molecule GPR15 in relation to relapsi
Autor:
Oskar McWilliam, Nicole Elisabeth Ørsted Schultz, Flemming Fryd Johansen, Rune Skovgaard Rasmussen, Nina Vindegaard, Helle K. Iversen, Henrik Hasseldam
Publikováno v:
Neurological Research. 41:289-297
In this clinical case-control study, we investigated statin treatment in stroke patients on a range of inflammatory effectors in peripheral blood. We focus on RhoA GTPase and its downstream effectors as a future inflammatory target in stroke treatmen
Autor:
Hanne Vibeke Marquart, Peter Damm, Rikke Holm Hansen, Finn Sellebjerg, Eva Rosa Petersen, Oskar McWilliam, Cecilie Ammitzbøll, Marina Rode von Essen
Publikováno v:
Brain. 142:120-132
With the discovery that the highly effective anti-CD20 antibody therapies developed to deplete CD20+ B cells deplete CD20+ T cells equally well, a great interest in the biological properties of CD20+ T cells has emerged. In this study we show that CD
Publikováno v:
Nersesjan, V, McWilliam, O, Krarup, L H & Kondziella, D 2021, ' Autoimmune Encephalitis Related to Cancer Treatment With Immune Checkpoint Inhibitors : A Systematic Review ', Neurology, vol. 97, no. 2, pp. e191-e202 . https://doi.org/10.1212/WNL.0000000000012122
ObjectiveTo determine the clinical and laboratory features of immune checkpoint inhibitor (ICPI)–associated autoimmune encephalitis (ICPI-AIE), an increasingly recognized adverse event with ICPI treatment.MethodsWe searched PubMed, The Cochrane Lib
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bcee11111a4bc37158c1c25ff47ea3cc
https://curis.ku.dk/ws/files/276276312/2020.08.31.20185009v1.full.pdf
https://curis.ku.dk/ws/files/276276312/2020.08.31.20185009v1.full.pdf
Autor:
Oskar McWilliam, Marina Rode von Essen, Finn Sellebjerg, Cecilie Ammitzbøll, Jeppe Romme Christensen, Alexander Cuculiza Henriksen, Eva Rosa Petersen
Publikováno v:
Multiple sclerosis and related disorders. 52
Natalizumab treatment increases the frequencies of B cells in blood but reduces IgG in blood and CSF. Plasmablasts are important in the production of IgG, and the development of plasmablasts is CD49d dependent.We hypothesized that natalizumab treatme
BACKGROUNDImmune checkpoint inhibitors (ICPI) are a game changer in the treatment of various metastasized cancers, but emerging reports of adverse events, including ICPI-associated autoimmune encephalitis (ICPI-AIE), are concerning. We aimed to colle
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9dab67d4694a31ead06ac9afbafa4acd
https://doi.org/10.1101/2020.08.31.20185009
https://doi.org/10.1101/2020.08.31.20185009
Autor:
Finn Sellebjerg, Marina Rode von Essen, Rikke Holm Hansen, Helene Højsgaard Chow, Cecilie Ammitzbøll, Oskar McWilliam
Publikováno v:
Journal of Neuroimmunology. 339:577117
In patients with multiple sclerosis (MS) and healthy controls (HC) we studied circulating MAIT cells and MAIT cell subtypes expressing CXCR3 and CCR6 by flow cytometry. Absolute numbers of MAIT cells and specifically Tc17-like MAIT cells were lower i
Autor:
Cecilie, Ammitzbøll, Marina R, von Essen, Lars, Börnsen, Eva Rosa, Petersen, Oskar, McWilliam, Rikke, Ratzer, Jeppe, Romme Christensen, Annette B, Oturai, Helle B, Søndergaard, Finn, Sellebjerg
Publikováno v:
Journal of autoimmunity. 97
Smoking is a risk factor for the development and progression of multiple sclerosis (MS); however, the pathogenic effects of smoking are poorly understood. We studied the smoking-associated chemokine receptor-like molecule GPR15 in relation to relapsi