Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Oreth Montero-Ruíz"'
Autor:
José Antonio Palma-Jacinto, Edgar López-López, José Luis Medina-Franco, Oreth Montero-Ruíz, Isela Santiago-Roque
Publikováno v:
Adipocyte, Vol 13, Iss 1 (2024)
Insulin resistance is caused by the abnormal secretion of proinflammatory cytokines in adipose tissue, which is induced by an increase in lipid accumulation in adipocytes, hepatocytes, and myocytes. The inflammatory pathway involves multiple targets
Externí odkaz:
https://doaj.org/article/93f142c9164744b2bec2bd5f30d4b78a
Autor:
Oreth Montero-Ruiz, Carlos A. Gallardo-Hernández, José A. Palma-Jacinto, Isela Santiago-Roque
Publikováno v:
Anales de Radiología, México, Vol 23, Iss 3 (2024)
Objetivo: Analizar las evidencias científicas del efecto de la radiación ionizante sobre leucocitos de personal ocupacionalmente expuesto y el uso del ensayo cometa como método diagnóstico del daño a ADN. Método: Se realizó una revisión siste
Externí odkaz:
https://doaj.org/article/1e6b0555666e41f59490224436502fb5
Autor:
Oreth Montero-Ruíz, Patricia Pérez-Vera, Sara Frías, Roberto Rivera-Luna, Gloria Dehesa, Consuelo Salas, Berenice Jarquín
Publikováno v:
Cancer Genetics and Cytogenetics. 184:94-98
We evaluated the prevalence of BCR/ABL, MLL, and ETV6/RUNX1 rearrangements as well as CDKN2A (alias p16) deletion in a group of Mexican children with acute lymphoblastic leukemia (ALL) to determine whether the changes coexist, and to compare the inci
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::90941ea7d37c06c8d40b0063cf9374fb
https://doi.org/10.1016/j.cancergencyto.2008.04.003
https://doi.org/10.1016/j.cancergencyto.2008.04.003
Autor:
Oreth Montero Ruíz
Publikováno v:
Universidad Autónoma Metropolitana
UAM
Repositorio Institucional de la UAM Iztapalapa
UAM
Repositorio Institucional de la UAM Iztapalapa
Bone marrow samples from children with acute lymphoblastic leukemia were analyzed for the expresión of RUNX1a, RUNX1b and RUNX1c isoforms. Obtained patterns were associated with genetic abnormalities and the expression of the RUNX1 regulated gene BL
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0fab43fde682a19d38d356754d073f30
Autor:
Patricia Pérez-Vera, Humberto González-Márquez, Miguel Angel Alcántara-Ortigoza, Oreth Montero-Ruíz, Miguel Betancourt, Rocío Juárez-Velázquez
Publikováno v:
Leukemia research. 36(9)
Bone marrow samples from children with acute lymphoblastic leukemia were analyzed for the expression of RUNX1a/b/c isoforms. Obtained patterns were associated with genetic abnormalities and the expression of the RUNX1 regulated gene BLK. RUNX1c was p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f71154bb2929730fc48553f1a45ec2fc
https://pubmed.ncbi.nlm.nih.gov/22748822
https://pubmed.ncbi.nlm.nih.gov/22748822
Autor:
Sara Frías, Diego Arenas, Alessandra Carnevale, Rogelio Paredes-Aguilera, Roberto Rivera-Luna, Adán Valladares, Oreth Montero-Ruíz, Patricia Pérez-Vera
Publikováno v:
Cancer genetics and cytogenetics. 180(2)
RUNX1 over-representation is present in children with acute lymphoblastic leukemia. Although these cases have been related with poor outcome, not all reports describe patient follow-up. To understand its associated clinical features and prognosis, we
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1c782c7e815f914f224b3b485537defd
https://pubmed.ncbi.nlm.nih.gov/18206538
https://pubmed.ncbi.nlm.nih.gov/18206538
Autor:
Sara Frías, Verónica Ulloa-Avilés, Oreth Montero-Ruíz, Patricia Pérez-Vera, Rogelio Paredes-Aguilera, Rocío Cárdenas-Cardós, Roberto Rivera-Luna, Alessandra Carnevale
Publikováno v:
Cancer genetics and cytogenetics. 162(2)
The t(12;21) produces the gene fusion ETV6/RUNX1 and is a frequent rearrangement in childhood ALL, associated with a good prognosis. In Mexico its prevalence has not been reported. This study evaluated a group of consecutive Mexican children with new
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::501df4f5ff68a37ddafc559464ee37d8
https://pubmed.ncbi.nlm.nih.gov/16213362
https://pubmed.ncbi.nlm.nih.gov/16213362