Zobrazeno 1 - 10
of 69
pro vyhledávání: '"Onafuwa-Nuga A"'
Autor:
Zaikos, Thomas D. 1, Terry, Valeri H. 2, Sebastian Kettinger, Nadia T. 3, 4, 9, Lubow, Jay 1, Painter, Mark M. 6, Virgilio, Maria C. 3, Neevel, Andrew 2, Taschuk, Frances 2, 10, Onafuwa-Nuga, Adewunmi 2, 11, McNamara, Lucy A. 1, 12, Riddell, James, IV 5, Bixby, Dale 7, Markowitz, Norman 8, Collins, Kathleen L. 1, 2, 3, 4, 5, 6, 13, ∗
Publikováno v:
In Cell Reports 26 December 2018 25(13):3759-3773
Autor:
Thomas D. Zaikos, Valeri H. Terry, Nadia T. Sebastian Kettinger, Jay Lubow, Mark M. Painter, Maria C. Virgilio, Andrew Neevel, Frances Taschuk, Adewunmi Onafuwa-Nuga, Lucy A. McNamara, James Riddell, IV, Dale Bixby, Norman Markowitz, Kathleen L. Collins
Publikováno v:
Cell Reports, Vol 25, Iss 13, Pp 3759-3773.e9 (2018)
Summary: Long-lived reservoirs of persistent HIV are a major barrier to a cure. CD4+ hematopoietic stem and progenitor cells (HSPCs) have the capacity for lifelong survival, self-renewal, and the generation of daughter cells. Recent evidence shows th
Externí odkaz:
https://doaj.org/article/1396126f930f4b8b9aac16b70f1728b2
Autor:
Nadia T Sebastian, Thomas D Zaikos, Valeri Terry, Frances Taschuk, Lucy A McNamara, Adewunmi Onafuwa-Nuga, Ryan Yucha, Robert A J Signer, James Riddell, Dale Bixby, Norman Markowitz, Sean J Morrison, Kathleen L Collins
Publikováno v:
PLoS Pathogens, Vol 13, Iss 7, p e1006509 (2017)
Latent HIV infection of long-lived cells is a barrier to viral clearance. Hematopoietic stem and progenitor cells are a heterogeneous population of cells, some of which are long-lived. CXCR4-tropic HIVs infect a broad range of HSPC subtypes, includin
Externí odkaz:
https://doaj.org/article/7587686bdff1460f9e8cbb0bac1907c0
Autor:
McNamara, Lucy A., Onafuwa-Nuga, Adewunmi, Sebastian, Nadia T., Riddell, James, Bixby, Dale, Collins, Kathleen L.
Publikováno v:
The Journal of Infectious Diseases, 2013 Jun . 207(12), 1807-1816.
Externí odkaz:
http://dx.doi.org/10.1093/infdis/jit118
Autor:
Carter, Christoph C. 1, 2, 8, McNamara, Lucy A. 3, 4, 8, Onafuwa-Nuga, Adewunmi 5, Shackleton, Mark 6, Riddell, James, IV 5, Bixby, Dale 5, Savona, Michael R. 5, 9, Morrison, Sean J. 5, 6, 7, Collins, Kathleen L. 1, 2, 3, 5, ∗
Publikováno v:
In Cell Host & Microbe 17 March 2011 9(3):223-234
Autor:
V.H. Terry, N.T. Sebastian, R. Yucha, F. Taschuk, L.A. McNamara, A. Onafuwa-Nuga, J. Riddell, IV, D. Bixby, K.L. Collins
Publikováno v:
Journal of Virus Eradication, Vol 1, Iss , Pp 34- (2015)
Externí odkaz:
https://doaj.org/article/4e293a3fad0a46df98dd682c51fd8975
Autor:
N. Sebastian, V. Terry, F. Taschuk, L.A. McNamara, A. Onafuwa-Nuga, J. Riddell, D. Bixby, K.L. Collins
Publikováno v:
Journal of Virus Eradication, Vol 1, Iss , Pp 33-34 (2015)
Externí odkaz:
https://doaj.org/article/721b65baf64d4da7a3bfbdab164f06cc
Autor:
Norman Markowitz, Robert A.J. Signer, Frances Taschuk, Thomas D. Zaikos, Dale L. Bixby, Nadia T. Sebastian, Valeri H. Terry, Kathleen L. Collins, James Riddell, Adewunmi Onafuwa-Nuga, Sean J. Morrison, Lucy A. McNamara, Ryan E. Yucha
Publikováno v:
PLoS Pathogens, Vol 13, Iss 7, p e1006509 (2017)
PLoS Pathogens
PLoS pathogens, vol 13, iss 7
PLoS Pathogens
PLoS pathogens, vol 13, iss 7
Latent HIV infection of long-lived cells is a barrier to viral clearance. Hematopoietic stem and progenitor cells are a heterogeneous population of cells, some of which are long-lived. CXCR4-tropic HIVs infect a broad range of HSPC subtypes, includin
Autor:
Carter, Christoph C.1,2, Onafuwa-Nuga, Adewunmi3, McNamara, Lucy A.4,5, Riddell, IV, James3, Bixby, Dale3, Savona, Michael R.6, Collins, Kathleen L.1,2,3,4 klcollin@umich.edu
Publikováno v:
Nature Medicine. Apr2010, Vol. 16 Issue 4, p446-451. 6p. 1 Diagram, 5 Graphs.
Autor:
Lucy A. McNamara, Michael Mashiba, Estelle Chiari-Fort, Jason M. Norman, Kathleen L. Collins, Adewunmi Onafuwa-Nuga, Wenwen Shen
Publikováno v:
Nature immunology
APOBEC3G (A3G) is an intrinsic antiviral factor that inhibits the replication of human immunodeficiency virus (HIV) by deaminating cytidine residues to uridine. This causes guanosine-to-adenosine hypermutation in the opposite strand and results in in