Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Omer Fleissig"'
Publikováno v:
Journal of Periodontology. 91:1067-1075
Background The aim of the study is to examine bone healing following augmentation with allograft or β-tricalcium phosphate (β-TCP) and evaluate orthodontic tooth movement (OTM) into the augmented sites. Methods The study included two parts. Part I
Autor:
Yehuda Klein, Dror Aizenbud, Stella Chaushu, Omer Fleissig, Yankel Gabet, Avraham Zini, Hagai Hazan-Molina
Publikováno v:
European journal of orthodontics. 43(6)
Introduction Basic research in orthodontics is commonly conducted in rodents. However, experimental studies on orthodontic tooth movement (OTM) lack a standard method to examine OTM and periodontal changes. This study describes a unifying protocol fo
Publikováno v:
Journal of dental research. 101(1)
Orthodontic tooth movement (OTM) is generated by a mechanical force that induces an aseptic inflammatory response in the periodontal tissues and a subsequent coordinated process of bone resorption and apposition. In this review, we critically summari
Autor:
Omer Fleissig, Stella Chaushu, Sharon Wald, O Barel, Yasmin Saba, Immo Prinz, Inga Sandrock, Avi-Hai Hovav, Noam Koren, A Leibowitz, Yuval Aizenbud, Dror Aizenbud, Anneke Wilharm, O. Heyman, Y Tal, Khaled Zubeidat
Publikováno v:
Journal of dental research. 100(7)
Sustained mechanical forces applied to tissue are known to shape local immunity. In the oral mucosa, mechanical stress, either naturally induced by masticatory forces or externally via mechanical loading during orthodontic tooth movement (OTM), is tr
Autor:
Stella Chaushu, A. Maimon, S. Shani-Kdoshim, Ayala Stabholz, O. Levin-Talmor, Omer Fleissig, Y. Meirow, David Polak, J. Garber-Berkstein, Yehuda Klein, A. Leibovich
Publikováno v:
Journal of dental research. 99(7)
The current study aimed at investigating the long-term biological mechanisms governing bone regeneration in osseous defects filled with bovine bone (BB). Tooth extraction sockets were filled with BB or left unfilled for natural healing in a C57BL/6 m
Autor:
Ofer Mandelboim, David Polak, Omer Fleissig, Yehuda Klein, Stella Chaushu, Yechezkel Barenholz
Publikováno v:
Scientific Reports
Scientific Reports, Vol 10, Iss 1, Pp 1-10 (2020)
Scientific Reports, Vol 10, Iss 1, Pp 1-10 (2020)
Orthodontic tooth movement (OTM) is a “sterile” inflammatory process. The present study aimed to reveal the underlying biological mechanisms, by studying the force associated-gene expression changes, in a time-dependent manner. Ni-Ti springs were
Publikováno v:
Journal of periodontology. 90(2)
Background The aim of this study was to investigate the biological mechanisms underlying alveolar bone regeneration (ABR) and orthodontic tooth movement into bovine bone (BB) regenerated sites. Methods Two mouse models were established in C57BL/6 mic
Publikováno v:
American journal of orthodontics and dentofacial orthopedics : official publication of the American Association of Orthodontists, its constituent societies, and the American Board of Orthodontics. 154(5)
Introduction Force application (FA) during orthodontic tooth movement is mediated through periodontal ligament (PDL) fibroblasts. FA on deciduous teeth has an inherent risk of root resorption, which is less in permanent teeth. Currently, the root res
Autor:
Ronit Naor, Dror Mevorach, Omer Fleissig, Rachel Yamin, Ofer Mandelboim, Ariella Glasner, Jawad Abed, Chamutal Gur, Zvi Granot, Shunit Coppenhagen-Glazer, Yotam Bar-On, Gilad Bachrach, Jonatan Enk, Shilo Rosenberg
Publikováno v:
Cell Host & Microbe
SummaryUropathogenic Escherichia coli (UPEC) are a common cause of urinary tract infections (UTIs) in humans. While the importance of natural killer (NK) cells in innate immune protection against tumors and viral infections is well documented, their
Autor:
Rinat Tzach-Nahman, Asaf Wilensky, Omer Fleissig, Lior Shapira, Rizan Nashef, Gabriel Nussbaum, A. Palmon
Publikováno v:
Scientific Reports
Scientific Reports, Vol 7, Iss 1, Pp 1-11 (2017)
Scientific Reports, Vol 7, Iss 1, Pp 1-11 (2017)
Tissue damage in chronic periodontal disease is driven by the host response to a dysbiotic microbiota, and not by bacteria directly. Among chronic inflammatory diseases of the oral cavity, inflammation and tissue damage around dental implants (peri-i