Zobrazeno 1 - 10
of 31
pro vyhledávání: '"Olufolake Adisa"'
Autor:
Samit Ghosh, Fang Tan, Tianwei Yu, Yuhua Li, Olufolake Adisa, Mario Mosunjac, Solomon F Ofori-Acquah
Publikováno v:
PLoS ONE, Vol 6, Iss 3, p e18399 (2011)
Sickle cell disease (SCD) is characterized by hemolysis, vaso-occlusion and ischemia reperfusion injury. These events cause endothelial dysfunction and vasculopathies in multiple systems. However, the lack of atherosclerotic lesions has led to the id
Externí odkaz:
https://doaj.org/article/344572c54fa7475dbbf0b6dabb63ba7f
Autor:
Janet Figueroa, Harold K. Simon, Reshika D. Mendis, Claudia R. Morris, Adina Alazraki, Robert Hagbom, Sam Friedman, Shaminy Manoranjithan, Courtney McCracken, Stephen M. Russell, Stephanie G. Cohen, Olufolake Adisa, Zayir M Malik
Publikováno v:
Annals of Emergency Medicine. 76:S46-S55
Study objective Acute chest syndrome is a leading cause of mortality in patients with sickle cell disease (SCD). Because early detection of acute chest syndrome is directly tied to prognosis, young patients with SCD undergo countless chest radiograph
Autor:
Miranda Bailey, Santosh L. Saraf, Olufolake Adisa, Laura Pastor, Wally R. Smith, Stephen Brown, Nicholas Ramscar, Ashley Bonner, Kenneth I. Ataga
Publikováno v:
Blood. 136:32-33
OBJECTIVES: Vaso-occlusion is a hallmark feature of sickle cell disease (SCD) that promotes ischemia-reperfusion injury and leads to acute pain episodes, known as vaso-occlusive crises (VOCs). VOCs are disabling and frequently impact on the ability o
Autor:
Rodney Theodore, Vaughn Barry, Robert Clark Brown, Maa-Ohui Quarmyne, Wei Dong, Clinton H. Joiner, James Bost, Sonia Anand, Iris Buchanan, Olufolake Adisa, Peter A. Lane
Publikováno v:
American Journal of Hematology. 92:77-81
The clinical efficacy of hydroxyurea in patients with sickle cell anemia (SCA) has been well established. However, data about its clinical effectiveness in practice is limited. We evaluated the clinical effectiveness of hydroxyurea in a large pediatr
Publikováno v:
Annals of the American Thoracic Society. 14:475-477
Autor:
Amanda Watt, Deepika S. Darbari, Michael R. DeBaun, Barbara Speller-Brown, Zalaya K. Ivy, Olufolake Adisa, Shaina M. Willen, Suvankar Majumdar, Leah Vance Utset, Leann Schilling, Stefanie Pepper, Chantel L. Jenkins, Mark Rodeghier
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9426fb65915ebabc383907bac58cfad5
https://europepmc.org/articles/PMC5842125/
https://europepmc.org/articles/PMC5842125/
Autor:
Samit Ghosh, Kesmic A Jackson, Prasanthi Chappa, Fang Tan, David R. Archer, Solomon F. Ofori-Acquah, Olufolake Adisa
Publikováno v:
Journal of Clinical Investigation. 123:4809-4820
The prevention and treatment of acute chest syndrome (ACS) is a major clinical concern in sickle cell disease (SCD). However, the mechanism underlying the pathogenesis of ACS remains elusive. We tested the hypothesis that the hemolysis byproduct hemi
Publikováno v:
Respiratory Medicine ISBN: 9783319419107
Acute chest syndrome (ACS) is the major lung complication and leading cause of death in sickle cell disease (SCD). It is characterized by a rapid disease onset, pulmonary infiltration, hypoxemia, and several other clinical indications. Postmortem ACS
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::50d53af14ba1e1998ac0b4838b3830d8
https://doi.org/10.1007/978-3-319-41912-1_3
https://doi.org/10.1007/978-3-319-41912-1_3
Autor:
Sarah G, Lazarus, Michael, Kelleman, Olufolake, Adisa, April R, Zmitrovich, Robert, Hagbom, Stephanie, Cohen, Courtney E, McCracken, Claudia R, Morris
Publikováno v:
American journal of hematology. 91(8)
Alloimmunization to transfused HOD red blood cells is not increased in mice with sickle cell disease
Autor:
Solomon F. Ofori-Acquah, Eldad A. Hod, Leslie S. Kean, James C. Zimring, Steven L. Spitalnik, Olufolake Adisa, David R. Archer, Jeanne E. Hendrickson, Samit Ghosh, Prasanthi Chappa, Jennifer Perry
Publikováno v:
Transfusion. 52:231-240
BACKGROUND: Increased rates of red blood cell (RBC) alloimmunization in patients with sickle cell disease may be due to transfusion frequency, genetic predisposition, or immune dysregulation. To test the hypothesis that sickle cell pathophysiology in