Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Olof Joakim Pettersson"'
Publikováno v:
Endocrine Connections, Vol 10, Iss 4, Pp 422-431 (2021)
Background: Monitoring of pancreatic neuroendocrine tumors (PanNET) undergoing peptide receptor radionuclide therapy (PRRT) with 177Lu-DOTATATE depends on changes in tumor size, which often occur late. Tumor growth rate (TGR) allows for quantitative
Externí odkaz:
https://doaj.org/article/f7414d7e54eb474ebc977a0c310f1cee
Publikováno v:
Nucleic Acids Research
Pettersson, O J, Aagaard, L, Jensen, T G & Damgaard, C K 2015, ' Molecular mechanisms in DM1-a focus on foci ', Nucleic Acids Research, vol. 43, no. 4, pp. 2433-2441 . https://doi.org/10.1093/nar/gkv029
Pettersson, O J, Aagaard, L, Jensen, T G & Damgaard, C K 2015, ' Molecular mechanisms in DM1-a focus on foci ', Nucleic Acids Research, vol. 43, no. 4, pp. 2433-2441 . https://doi.org/10.1093/nar/gkv029
Myotonic dystrophy type 1 is caused by abnormal expansion of a CTG-trinucleotide repeat in the gene encoding Dystrophia Myotonica Protein Kinase (DMPK), which in turn leads to global deregulation of gene expression in affected individuals. The transc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::72b2674c2b7bd639d856372026f42110
https://pubmed.ncbi.nlm.nih.gov/25605794
https://pubmed.ncbi.nlm.nih.gov/25605794
Autor:
Diana Andrejeva, Christian Kroun Damgaard, Lars Aagaard, Thomas G. Jensen, Olof Joakim Pettersson, Rune Thomsen
Publikováno v:
Pettersson, O J, Aagaard, L, Andrejeva, D, Thomsen, R, Jensen, T G & Damgaard, C K 2014, ' DDX6 regulates sequestered nuclear CUG-expanded DMPK-mRNA in dystrophia myotonica type 1 ', Nucleic Acids Research, vol. 42, no. 11, pp. 7186-7200 . https://doi.org/10.1093/nar/gku352
Nucleic Acids Research
Nucleic Acids Research
Myotonic dystrophy type 1 (DM1) is caused by CUG triplet expansions in the 3′ UTR of dystrophia myotonica protein kinase (DMPK) messenger ribonucleic acid (mRNA). The etiology of this multi-systemic disease involves pre-mRNA splicing defects elicit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2048ad59f783bafc2b4860f7fd01a3e3
https://pure.au.dk/portal/da/publications/ddx6-regulates-sequestered-nuclear-cugexpanded-dmpkmrna-in-dystrophia-myotonica-type-1(1d7f1b5b-2ddb-4351-8cc7-aaff9f138486).html
https://pure.au.dk/portal/da/publications/ddx6-regulates-sequestered-nuclear-cugexpanded-dmpkmrna-in-dystrophia-myotonica-type-1(1d7f1b5b-2ddb-4351-8cc7-aaff9f138486).html
Autor:
Rune Thomsen, Maria Jakobsen, Thomas J. Corydon, Thomas G. Jensen, Christina Bøg Pedersen, Niels Gregersen, Jens Michael Hertz, Olof Joakim Pettersson, Jan Larsen
Publikováno v:
BMC Research Notes, Vol 4, Iss 1, p 490 (2011)
BMC Research Notes
Larsen, J, Pettersson, O J, Jakobsen, M, Thomsen, R, Pedersen, C B, Hertz, J M, Gregersen, N, Corydon, T J & Jensen, T G 2011, ' Myoblasts generated by lentiviral mediated MyoD transduction of myotonic dystrophy type 1 (DM1) fibroblasts can be used for assays of therapeutic molecules ', BMC Research Notes, vol. 4, pp. 490 . https://doi.org/10.1186/1756-0500-4-490
BMC Research Notes
Larsen, J, Pettersson, O J, Jakobsen, M, Thomsen, R, Pedersen, C B, Hertz, J M, Gregersen, N, Corydon, T J & Jensen, T G 2011, ' Myoblasts generated by lentiviral mediated MyoD transduction of myotonic dystrophy type 1 (DM1) fibroblasts can be used for assays of therapeutic molecules ', BMC Research Notes, vol. 4, pp. 490 . https://doi.org/10.1186/1756-0500-4-490
Background Myotonic dystrophy type 1 (DM1) is the most common muscle dystrophy in adults. The disease is caused by a triplet expansion in the 3'end of the myotonic dystrophy protein kinase (DMPK) gene. In order to develop a human cell model for inves
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9ac9c6e10afcc505bb2194ebe7105b07
http://www.biomedcentral.com/1756-0500/4/490
http://www.biomedcentral.com/1756-0500/4/490