Výsledky vyhledávání - "Olivier D. Loudig"

Supplemental methods from An Akt3 Splice Variant Lacking the Serine 472 Phosphorylation Site Promotes Apoptosis and Suppresses Mammary Tumorigenesis

Autor: Rachel B. Hazan, Richard N. Kitsis, Larry Norton, Susan Fineberg, Yihong Wang, Jeffrey Segall, Greg R. Phillips, Wendy M. McKimpson, Dulguun Amgalan, Olivier D. Loudig, Outhiriaradjou Benard, Huizhi Liang, Jiahong Yao, Kimita Suyama

Supplemental methods

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9cf47f9924a434c6c4df68bac2baf488
https://doi.org/10.1158/0008-5472.22410386.v1

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Supplemental Figure. 4 from An Akt3 Splice Variant Lacking the Serine 472 Phosphorylation Site Promotes Apoptosis and Suppresses Mammary Tumorigenesis

Effects of Akt3/-S472 knockout and Akt3/+S472 or Akt1 overexpression on apoptosis

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5c5984c52fee6131d638704114434418
https://doi.org/10.1158/0008-5472.22410398.v1

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Supplemental Figure.1 from An Akt3 Splice Variant Lacking the Serine 472 Phosphorylation Site Promotes Apoptosis and Suppresses Mammary Tumorigenesis

Next generation sequencing of genomic DNA isolated from Akt3/+S472 and Akt3/-S472 knockout cell lines

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3981b86f7ab7e26c95d44029f68ab3f2
https://doi.org/10.1158/0008-5472.22410389

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Supplementary Figure legends from An Akt3 Splice Variant Lacking the Serine 472 Phosphorylation Site Promotes Apoptosis and Suppresses Mammary Tumorigenesis

Supplementary Figure legends

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::2209a20870fa0e906c62b27af7b3ec0e
https://doi.org/10.1158/0008-5472.22410383.v1

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Supplemental Figure. 5 from An Akt3 Splice Variant Lacking the Serine 472 Phosphorylation Site Promotes Apoptosis and Suppresses Mammary Tumorigenesis

Akt3/-S472 overexpression in MDA-MB-436 cells suppresses tumor growth by inducing Bim expression and tumor apoptosis

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9ec09e084c85a616d7d2101d58a03f00
https://doi.org/10.1158/0008-5472.22410395

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Supplementary Figure legends from An Akt3 Splice Variant Lacking the Serine 472 Phosphorylation Site Promotes Apoptosis and Suppresses Mammary Tumorigenesis

Supplementary Figure legends

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::25dc13f851ee222fda74d8f4c2c0648f
https://doi.org/10.1158/0008-5472.22410383

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Supplemental Figure. 2 from An Akt3 Splice Variant Lacking the Serine 472 Phosphorylation Site Promotes Apoptosis and Suppresses Mammary Tumorigenesis

Next generation sequencing of genomic DNA isolated from Akt3/+S472 and Akt3/-S472 knockout 3475 tumors

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1339364b6e0a8108ab6729b45da77da9
https://doi.org/10.1158/0008-5472.22410404

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Supplemental Figure. 3 from An Akt3 Splice Variant Lacking the Serine 472 Phosphorylation Site Promotes Apoptosis and Suppresses Mammary Tumorigenesis

CRISPR mediated knockout of Akt3/-S472 in MDA-MB-231 cells increases mammary tumorigenesis

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::158b1253b524a0b226aaad70b59d3c5e
https://doi.org/10.1158/0008-5472.22410401.v1

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Data from An Akt3 Splice Variant Lacking the Serine 472 Phosphorylation Site Promotes Apoptosis and Suppresses Mammary Tumorigenesis

The Akt pathway is a well-known promoter of tumor malignancy. Akt3 is expressed as two alternatively spliced variants, one of which lacks the key regulatory serine 472 phosphorylation site. Whereas the function of full-length Akt3 isoform (Akt3/+S472

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5e0a0d0a04e37edc3850c268538cf1d1
https://doi.org/10.1158/0008-5472.c.6507983.v1

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