Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Oliver Pelz-Ackermann"'
Autor:
Thomas Ernst, Franz X. Gruber, Oliver Pelz-Ackermann, Jacqueline Maier, Markus Pfirrmann, Martin C. Müller, Ingvild Mikkola, Kimmo Porkka, Dietger Niederwieser, Andreas Hochhaus, Thoralf Lange
Publikováno v:
Haematologica, Vol 94, Iss 9 (2009)
Background Various techniques have been employed to detect BCR-ABL kinase domain mutations in patients with chronic myeloid leukemia who are resistant to imatinib. This has led to different reported frequencies of mutations and the finding of a heter
Externí odkaz:
https://doaj.org/article/8780bb4cfcb644fb88c8f91f1b727968
Publikováno v:
DMW - Deutsche Medizinische Wochenschrift. 131:2390-2394
Autor:
Insa S. Schroeder, Bernadette Harwardt, Matthias Jung, Sabine Foja, Dagmar Riemann, Oliver Pelz-Ackermann
Publikováno v:
Cellular reprogramming. 15(1)
Pluripotency is characterized by specific transcription factors such as OCT4, NANOG, and SOX2, but also by pluripotency-associated microRNAs (miRs). Somatic cells can be reprogrammed by forced expression of these factors leading to induced pluripoten
Autor:
Thoralf Lange, Thomas Ernst, Andreas Hochhaus, Martin Müller, Markus Pfirrmann, Franz Gruber, Kimmo Porkka, Oliver Pelz-Ackermann, Dietger Niederwieser, Jacqueline Maier, Ingvild Mikkola
Background Various techniques have been employed to detect BCR-ABL kinase domain mutations in patients with chronic myeloid leukemia who are resistant to imatinib. This has led to different reported frequencies of mutations and the finding of a heter
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0484d34a300c0db21dc61b2d11a8e618
https://hdl.handle.net/10037/3181
https://hdl.handle.net/10037/3181
Autor:
Thoralf Lange, Heike Pfeifer, Michael W. Deininger, Dietger Niederwieser, Oliver Pelz-Ackermann, Haifa Kathrin Al-Ali, Song-Yau Wang, Michael Cross
Publikováno v:
Leukemia. 22(12)
Highly sensitive and quantitative detection of BCR-ABL kinase domain mutations by ligation PCR
Autor:
Rüdiger Alt, Franziska Wilhelm, Oliver Pelz-Ackermann, Dietger Niederwieser, Dietmar Egger, Michael Cross
Publikováno v:
Experimental hematology. 37(2)
Objective The ABCG2 transporter has been identified as a determinant of the side population (SP) in murine bone marrow and a potential marker for primitive stem cells. To assess the potential of the SP phenotype and ABCG2 expression to identify stem
Autor:
Jörg Petersen, Marc von Mach, Amelie Lupp, Walter Beerheide, Manja Kamprad, Wiebke Schormann, Carolin Elsner, Andreas K. Nussler, Jan G. Hengstler, Dimitry Spitkovsky, Henryk Zulewski, Jeanett Edelmann, Friedrich Jakob Hammersen, Matthias Hermes, Lars Christian Horn, Oliver Pelz-Ackermann, Alexander Bauer, Marc Brulport, Wolfgang Härtig
Publikováno v:
Hepatology (Baltimore, Md.). 46(3)
In recent years, a large number of groups studied the fate of human stem cells in livers of immunodeficient animals. However, the interpretation of the results is quite controversial. We transplanted 4 different types of human extrahepatic precursor
Autor:
Rothe K, Philipp Brandmaier, Ralf B. Tröbs, Ruediger Alt, Gabriele Scholz, Grigore Cernaianu, Oliver Pelz Ackermann, Helmut Witzigmann, Michael Cross
Publikováno v:
Journal of pediatric surgery. 43(7)
Therapies aiming at inducing differentiation or apoptosis of neuroblastoma (NB) are an important research topic. Although retinoic acid showed promising antitumoral results, its effects against refractory disease are limited. Putative candidates for
Autor:
Dietger Niederwieser, Oliver Pelz-Ackermann, Michael W. Deininger, Michael Cross, Haifa Kathrin Al-Ali, Rainer Krahl, Song-Yau Wang, Kathrin Wildenberger, Thoralf Lange
Publikováno v:
Blood. 110:2918-2918
Objectives: Acquired resistance to imatinib (IM) in patients with chronic myeloid leukaemia (CML) is frequently due to mutations of the BCR-ABL kinase domain (KD). Current thinking holds that this results from the selection of pre-existing mutant clo
Autor:
Thomas Ernst, Ingvild Mikkola, Martin C. Mueller, Kimmo Porkka, Oliver Pelz-Ackermann, Andreas Hochhaus, Thoralf Lange, Franz Gruber, Dietger Niederwieser
Publikováno v:
Blood. 110:321-321
Major cause of acquired resistance to imatinib in patients (pts) with chronic myeloid leukemia (CML) are BCR-ABL kinase domain (KD) mutations. Various techniques to detect KD mutations have been employed, resulting in different frequencies of mutatio