Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Oliver Lieven"'
Publikováno v:
PLoS ONE, Vol 9, Iss 12, p e115442 (2014)
Besides the active Wnt signalling itself, the extracellular inhibition by Dkk1 is important for various embryonic developmental processes, such as optic vesicle differentiation and facial outgrowth. Although a feedback crosstalk of the active Wnt/β-
Externí odkaz:
https://doaj.org/article/84361477904144b7a286e3bb4d34dd66
Autor:
Nadine Nagy, Oliver Lieven, Stephanie Pick, Jens W. Fischer, Vu Thao-Vi Dao, Tatsiana Suvorava, Ulrich Rüther, Martina Weber, Georg Kojda
Publikováno v:
Antioxidants & Redox Signaling, 23(9), 711-723. Mary Ann Liebert Inc.
Aims: Vascular oxidative stress generated by endothelial NO synthase (eNOS) was observed in experimental and clinical cardiovascular disease, but its relative importance for vascular pathologies is unclear. We investigated the impact of eNOS-dependen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3d367b502e7110f4f8e34d99b79798b3
https://doi.org/10.1089/ars.2014.6059
https://doi.org/10.1089/ars.2014.6059
Autor:
Anant Mamidi, Jenny Johansson, Henrik Semb, Zarah Löf Öhlin, Thomas U. Greiner, Gokul Kesavan, Silvia Lommel, Wan Chun Li, Oliver Lieven
Publikováno v:
Development. 141:685-696
Delamination plays a pivotal role during normal development and cancer. Previous work has demonstrated that delamination and epithelial cell movement within the plane of an epithelium are associated with a change in cellular phenotype. However, how t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::07525167d0cf51c0e42aaf524e350187
https://doi.org/10.1242/dev.100297
https://doi.org/10.1242/dev.100297
Autor:
Oliver Lieven, Sabine Maasen
Publikováno v:
GAIA - Ecological Perspectives for Science and Society. 16:35-40
Transdisciplinary research creates a specific site of negotiation ("Gestaltungsöffentlichkeit") in which both scientific and extra-scientific forms of knowledge are debated. Our empirical analysis indicates that creating this site requires "boundary
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::dbf6cb9db418d1a9a44de7eba1e31b22
https://doi.org/10.14512/gaia.16.1.11
https://doi.org/10.14512/gaia.16.1.11
Publikováno v:
PLoS ONE, Vol 9, Iss 12, p e115442 (2014)
PLoS ONE
PLoS ONE
Besides the active Wnt signalling itself, the extracellular inhibition by Dkk1 is important for various embryonic developmental processes, such as optic vesicle differentiation and facial outgrowth. Although a feedback crosstalk of the active Wnt/β-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1743a5281bf9400983f1302564e289d9
http://europepmc.org/articles/PMC4278905?pdf=render
http://europepmc.org/articles/PMC4278905?pdf=render
Autor:
Henning Bier, Julia Reifenberger, Oliver Lieven, Vera Balz, Andreas Knopf, Christian Gwosdz, Kathrin Scheckenbach
Publikováno v:
International journal of cancer. 118(3)
The abrogation of the function of the "gatekeeper of the genome", p53, is the most prevalent molecular alteration in solid human tumors. Regarding melanomas the involvement of p53 alterations is discussed controversially to date. In order to evaluate
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1ff4ed2f08033a7f3caa5caed1e676d1
https://pubmed.ncbi.nlm.nih.gov/16094622
https://pubmed.ncbi.nlm.nih.gov/16094622
Autor:
Kathrin Scheckenbach, Oliver Lieven, Karl Götte, Ulrike Bockmühl, Rainer Zotz, Henning Bier, Vera Balz
Publikováno v:
Cancer epidemiology, biomarkersprevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology. 13(11 Pt 1)
A polymorphism at codon 72 of the human tumor suppressor p53 determines translation into either arginine or proline. Yet, the impact of this amino acid variability on the risk to develop malignant tumors, particularly carcinomas associated with human
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::295b82e96156c93e5940f512e8e55232
https://pubmed.ncbi.nlm.nih.gov/15533911
https://pubmed.ncbi.nlm.nih.gov/15533911