Zobrazeno 1 - 10
of 109
pro vyhledávání: '"Olga O. Favorova"'
Autor:
Maria V. Pisklova, Natalia M. Baulina, Ivan S. Kiselev, Dmitry A. Zateyshchikov, Olga O. Favorova, Olga S. Chumakova
Publikováno v:
Терапевтический архив, Vol 95, Iss 4, Pp 302-308 (2023)
Background. Hypertrophic cardiomyopathy (HCM) is the most common inherited heart disease; it is characterized by left ventricular (LV) hypertrophy that cannot be explained by hemodynamic causes. It is believed that sarcomere dysfunction underlies the
Externí odkaz:
https://doaj.org/article/0a6bd92b65ef4224a850846c98b8e648
Publikováno v:
Терапевтический архив, Vol 94, Iss 4, Pp 463-466 (2022)
In recent decades, advances in molecular biology have led to a change in understanding the inheritance mechanisms and development of cardiological diseases of predominantly genetic origin, such as hypertrophic and dilated cardiomyopathies, familial h
Externí odkaz:
https://doaj.org/article/7283c178c430450aa303ed1273994c94
Multiplex Analysis of Serum Cytokine Profiles in Systemic Lupus Erythematosus and Multiple Sclerosis
Autor:
Mark M. Melamud, Evgeny A. Ermakov, Anastasiia S. Boiko, Daria A. Kamaeva, Alexey E. Sizikov, Svetlana A. Ivanova, Natalia M. Baulina, Olga O. Favorova, Georgy A. Nevinsky, Valentina N. Buneva
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 22, p 13829 (2022)
Changes in cytokine profiles and cytokine networks are known to be a hallmark of autoimmune diseases, including systemic lupus erythematosus (SLE) and multiple sclerosis (MS). However, cytokine profiles research studies are usually based on the analy
Externí odkaz:
https://doaj.org/article/725253afbfbc479795dbe2168b09c1a6
Publikováno v:
Acta Naturae
The epigenetic mechanisms of gene expression regulation are a group of the key cellular and molecular pathways that lead to inherited alterations in genes activity without changing their coding sequence. DNA methylation at the C5 position of cytosine
Autor:
Natalia Baulina, A N Boyko, Olga O. Favorova, M. S. Kozin, G. V. Pavlova, Olga Kulakova, Ivan Kiselev
Publikováno v:
Молекулярная биология. 55:956-964
There is increasing evidence that the interaction of the mitochondrial and nuclear genomes substantially affects the risk of neurodegenerative diseases. The role of mitonuclear interactions in the development of multiple sclerosis, a severe chronic n
Autor:
Philipp Kopylov, D. G. Gognieva, Anna S. Akselrod, Natalia М. Baulina, Andrey N. Rozhkov, N. A. Matveeva, Olga O. Favorova, E. S. Tebenkova, Dmitriy Yu. Shchekochikhin
Publikováno v:
Annals of the Russian academy of medical sciences. 75:283-291
Rationale. Cardiovascular diseases remain the leading cause of human death in the world. Studying the role of regulatory non-coding RNAs, which include short single-stranded miRNA molecules, allows a more detailed understanding of the pathological pr
Publikováno v:
Nevrologiâ, Nejropsihiatriâ, Psihosomatika, Vol 12, Iss 1S, Pp 15-19 (2020)
Multiple sclerosis (MS) is a severe chronic CNS disease characterized by autoimmune inflammation, demyelination, and neurodegeneration. The interaction of mitochondrial and nuclear genomes is shown to be important in the formation of a predisposition
Autor:
Olga Kulakova, A N Boyko, G. V. Pavlova, Ivan Kiselev, M. S. Kozin, Olga O. Favorova, Natalia Baulina
Publikováno v:
Nevrologiâ, Nejropsihiatriâ, Psihosomatika, Vol 12, Iss 1S, Pp 4-8 (2020)
Multiple sclerosis (MS) is a chronic autoimmune disease, in the pathogenesis of which the concurrence of demyelination of central nervous system (CNS) axons and neurodegeneration plays a role and which is accompanied by progressive neurological dysfu
Publikováno v:
Молекулярная биология. 54:699-704
Myocardial infarction (MI), one of the most common manifestations of cardiovascular system aging, is often fatal. The vast majority of studies on genetic susceptibility to age-dependent diseases are carried out using the case-control study design. Ho
Publikováno v:
Молекулярная биология. 54:596-602
Recently, it has been shown that dysfunction of mitochondria is an important component of the molecular mechanisms of the development of many neurodegenerative diseases. These include multiple sclerosis, a chronic autoimmune and neurodegenerative dis