Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Ole Jørgen Kaasbøll"'
Autor:
Maria Belland Olsen, Xiang Yi Kong, Mieke C. Louwe, Knut H. Lauritzen, Ylva Schanke, Ole Jørgen Kaasbøll, Håvard Attramadal, Jonas Øgaard, Sverre Holm, Pål Aukrust, Liv Ryan, Terje Espevik, Maria Yurchenko, Bente Halvorsen
Publikováno v:
Frontiers in Immunology, Vol 15 (2024)
Acute myocardial infarction (MI) results in tissue damage to affected areas of the myocardium. The initial inflammatory response is the most damaging for residual cardiac function, while at later stages inflammation is a prerequisite for proper heali
Externí odkaz:
https://doaj.org/article/ce016b2ea7294e4086757b857055d202
CTGF/CCN2 Postconditioning Increases Tolerance of Murine Hearts towards Ischemia-Reperfusion Injury.
Autor:
Ole Jørgen Kaasbøll, Ingvild Tronstad Moe, Mohammad Shakil Ahmed, Espen Stang, Else Marie Valbjørn Hagelin, Håvard Attramadal
Publikováno v:
PLoS ONE, Vol 11, Iss 2, p e0149000 (2016)
BACKGROUND AND PURPOSE:Previous studies of ischemia-reperfusion injury (IRI) in hearts from mice with cardiac-restricted overexpression of CCN2 have shown that CCN2 increases tolerance towards IRI. The objectives of this study were to investigate to
Externí odkaz:
https://doaj.org/article/477267d77ca8411d812f4adf71f2331b
Autor:
Jørgen Gravning, Stein Ørn, Ole Jørgen Kaasbøll, Vladimir N Martinov, Cord Manhenke, Kenneth Dickstein, Thor Edvardsen, Håvard Attramadal, Mohammed Shakil Ahmed
Publikováno v:
PLoS ONE, Vol 7, Iss 12, p e52120 (2012)
AIMS: Myocardial CCN2/CTGF is induced in heart failure of various etiologies. However, its role in the pathophysiology of left ventricular (LV) remodeling after myocardial infarction (MI) remains unresolved. The current study explores the role of CTG
Externí odkaz:
https://doaj.org/article/523b9b3e5bf44ade8481f9b2e4873574
Autor:
Sima Zolfaghari, Ole Jørgen Kaasbøll, Vivi T. Monsen, Bojana Sredic, Else Marie V. Hagelin, Håvard Attramadal
Publikováno v:
The Journal of biological chemistry.
Cellular Communication Network (CCN) proteins have multimodular structures important for their roles in cellular responses associated with organ development and tissue homeostasis. CCN2 has previously been reported to be secreted as a preproprotein t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1b581ee8045e589ba464908305cb8e10
https://pubmed.ncbi.nlm.nih.gov/36529291
https://pubmed.ncbi.nlm.nih.gov/36529291
Autor:
Else Marie Valbjørn Hagelin, Fabian A. Line, Vivi T. Monsen, M. Shakil Ahmed, Håvard Attramadal, Ole Jørgen Kaasbøll, Sima Zolfaghari
Publikováno v:
Journal of cell communication and signaling. 16(3)
CCN5 is a divergent member of the cellular communication network factor (CCN) family in that it lacks the carboxyl terminal cystine knot domain common to the other CCN family members. CCN5 has been reported to antagonize the profibrotic actions of CC
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b15a805e771ea7ff0e8e3fa62269f3a5
https://pubmed.ncbi.nlm.nih.gov/34854055
https://pubmed.ncbi.nlm.nih.gov/34854055
Autor:
Else Marie Valbjørn Hagelin, Jianhua Wang, Ashish K. Gadicherla, Ole Jørgen Kaasbøll, Vivi Talstad Monsen, Håvard Attramadal, Meng-Qiu Dong
Publikováno v:
Journal of Biological Chemistry. 293:17953-17970
Connective tissue growth factor (CTGF; now often referred to as CCN2) is a secreted protein predominantly expressed during development, in various pathological conditions that involve enhanced fibrogenesis and tissue fibrosis, and in several cancers
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7c9186b6feac03579c1ae7f7cfaeb250
https://doi.org/10.1074/jbc.ra118.004559
https://doi.org/10.1074/jbc.ra118.004559
Autor:
Davi de Miranda Fonseca, Tuula A. Nyman, Maria Belland Olsen, E. Schrumpf, Håvard Attramadal, Kuan Yang, Animesh Sharma, Bente Halvorsen, Azita Rashidi, Arne Yndestad, Espen Melum, Magnar Bjørås, Pål Aukrust, Jan Magnus Aronsen, Christen P. Dahl, Katrine Alfsnes, Maria Cornelia Louwe, Ole Jørgen Kaasbøll, Mingyi Yang, Ivar Sjaastad, Linn E. Fosshaug, Jonas Øgaard, Muhammad Shakil Ahmed, Thor Ueland, Vidar Magne Skulberg
Publikováno v:
JACC: Basic to Translational Science
An inflammatory response is required for tissue healing after a myocardial infarction (MI), but the process must be balanced to prevent maladaptive remodeling. This study shows that improved survival and cardiac function following MI, in mice deficie
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::781d815e558215b95d7e9ffdb49518a6
https://pubmed.ncbi.nlm.nih.gov/33427823
https://pubmed.ncbi.nlm.nih.gov/33427823
Autor:
Trine Ranheim, Erhe Gao, Arne Yndestad, Leif Erik Vinge, Ingrid Kristine Ohm, Håvard Attramadal, Øystein Sandanger, Azita Rashidi, Marte Bliksøen, Katrine Alfsnes, Pål Aukrust, Ole Jørgen Kaasbøll, Ståle H. Nymo
Publikováno v:
Biochemical and Biophysical Research Communications. 469:1012-1020
Background The innate immune receptor NLRP3 recognizes tissue damage and initiates inflammatory processes through formation multiprotein complexes with the adaptor protein ASC and caspase-1, i.e. NLRP3 inflammasomes, which through cleavage of pro-IL-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::584d3af565c485445079136ea1086162
https://doi.org/10.1016/j.bbrc.2015.12.051
https://doi.org/10.1016/j.bbrc.2015.12.051
Autor:
Egil Lien, Pål Aukrust, May-Kristin Torp, Trine Ranheim, Håvard Attramadal, Ole Jørgen Kaasbøll, Azita Rashidi, Arne Yndestad, Maria Belland Olsen, Alexandra Vanessa Finsen, Katrine Alfsnes, Linn E. Fosshaug, Marina Sokolova, Lars Eide, Øystein Sandanger, Leif Erik Vinge
Publikováno v:
Biochimica et biophysica acta. Molecular and cell biology of lipids. 1862(2)
Palmitate triggers inflammatory responses in several cell types, but its effects on cardiac fibroblasts are at present unknown. The aims of the study were to (1) assess the potential of palmitate to promote inflammatory signaling in cardiac fibroblas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3812de176c5300dfab106d8b523c5dce
https://pubmed.ncbi.nlm.nih.gov/27845246
https://pubmed.ncbi.nlm.nih.gov/27845246
Autor:
Else Marie Valbjørn Hagelin, Mohammad Shakil Ahmed, Ole Jørgen Kaasbøll, Håvard Attramadal, Ingvild Tronstad Moe, Espen Stang
Publikováno v:
PLoS ONE
PLoS ONE, Vol 11, Iss 2, p e0149000 (2016)
PLoS ONE, Vol 11, Iss 2, p e0149000 (2016)
BACKGROUND AND PURPOSE:Previous studies of ischemia-reperfusion injury (IRI) in hearts from mice with cardiac-restricted overexpression of CCN2 have shown that CCN2 increases tolerance towards IRI. The objectives of this study were to investigate to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::72926e6aa3910a4571bb74b474120030
http://europepmc.org/articles/PMC4752337
http://europepmc.org/articles/PMC4752337