Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Oktay F. Rifki"'
Publikováno v:
Journal of Molecular and Cellular Cardiology. 59:128-138
Recent work has demonstrated that autophagy, a phylogenetically conserved, lysosome-mediated pathway of protein degradation, is a key participant in pathological cardiac remodeling. One common feature of cell growth and autophagy is membrane biogenes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::355663338dea93cc8d8f4c264169f1b5
https://doi.org/10.1016/j.yjmcc.2013.02.015
https://doi.org/10.1016/j.yjmcc.2013.02.015
Autor:
Michelle A. Theobald, Richard M. Weinshilboum, Bianca A. Thomae, Eric D. Wieben, Oktay F. Rifki, Bruce W. Eckloff
Publikováno v:
Journal of Neurochemistry. 87:809-819
Sulfotransferase (SULT) 1A3 catalyzes the sulfate conjugation of catecholamines and structurally related drugs. As a step toward studies of the possible contribution of inherited variation in SULT1A3 to the pathophysiology of human disease and/or var
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::48bfb6a5b6f1a7dffa2f4c76e1d4b4aa
https://doi.org/10.1046/j.1471-4159.2003.02027.x
https://doi.org/10.1046/j.1471-4159.2003.02027.x
Autor:
Joseph A. Hill, Oktay F. Rifki
Publikováno v:
Journal of cardiovascular pharmacology. 60(3)
Stress-induced hypertrophic growth of the myocardium is a pathogenetic milestone in the progression of heart failure. Some evidence suggests that suppression of pathological cardiac hypertrophy per se is a viable target for therapeutic intervention,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0e3029200215faac27286174764edcc4
https://pubmed.ncbi.nlm.nih.gov/22743635
https://pubmed.ncbi.nlm.nih.gov/22743635
Autor:
Viviana Moresi, Rhonda Bassel-Duby, Chad E. Grueter, Michele Carrer, Oktay F. Rifki, John M. Shelton, James A. Richardson, Eric N. Olson
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America. 109(5)
Maintenance of skeletal muscle structure and function requires efficient and precise metabolic control. Autophagy plays a key role in metabolic homeostasis of diverse tissues by recycling cellular constituents, particularly under conditions of calori
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7ab37f4307f09674572602e71ba787ee
https://pubmed.ncbi.nlm.nih.gov/22307625
https://pubmed.ncbi.nlm.nih.gov/22307625
Publikováno v:
Circulation Research. 109
Background: Recent work has demonstrated that autophagy, a phylogenetically conserved, lysosome-mediated pathway of protein degradation, is a key participant in pathological cardiac remodeling. One common feature of cell growth and autophagy is membr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::696ea71844a10daf1d63a9861811f751
https://doi.org/10.1161/res.109.suppl_1.ap056
https://doi.org/10.1161/res.109.suppl_1.ap056
Autor:
Mikael Oscarson, Ulrich M. Zanger, Kathrin Klein, Urs A. Meyer, Oktay F. Rifki, Michel Eichelbaum
Publikováno v:
Clinical pharmacology and therapeutics. 80(5)
Background The antiepileptic drug carbamazepine (CBZ) is a potent inducer of human drug metabolism, resulting in serious interactions with many commonly prescribed drugs. The molecular mechanisms underlying this response are not well understood, howe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6e2a7155378ac1bf926167fa5e70b6b9
https://pubmed.ncbi.nlm.nih.gov/17112801
https://pubmed.ncbi.nlm.nih.gov/17112801
Autor:
Bianca A, Thomae, Oktay F, Rifki, Michelle A, Theobald, Bruce W, Eckloff, Eric D, Wieben, Richard M, Weinshilboum
Publikováno v:
Journal of neurochemistry. 87(4)
Sulfotransferase (SULT) 1A3 catalyzes the sulfate conjugation of catecholamines and structurally related drugs. As a step toward studies of the possible contribution of inherited variation in SULT1A3 to the pathophysiology of human disease and/or var
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::887775d12a75d0246d51fa2785fb1323
https://pubmed.ncbi.nlm.nih.gov/14622112
https://pubmed.ncbi.nlm.nih.gov/14622112