Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Oktavia Tarjanyi"'
Autor:
Akos Hegyi, Oktavia Tarjanyi, Tibor T. Glant, Katalin Mikecz, Tibor A. Rauch, Ferenc Boldizsár, Katalin Olasz
Publikováno v:
Cardiovascular Pathology. 25:353-361
Although dilated cardiomyopathy (DCM) is often caused by viral infections, it frequently involves autoimmune mechanisms associated with particular HLA-DR and DQ alleles. Our homozygous HLA-DQ8Ab(0) transgenic mice in the BALB/c background (HLA-DQ8(BA
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::760afa177eecb40581875fa894164dd3
https://doi.org/10.1016/j.carpath.2016.05.003
https://doi.org/10.1016/j.carpath.2016.05.003
Autor:
Bálint Balogh, Mónika Vecsernyés, Alexandra Stayer‐Harci, Gergely Berta, Oktávia Tarjányi, György Sétáló Jr
Publikováno v:
FEBS Open Bio, Vol 13, Iss 5, Pp 818-832 (2023)
Corticotropin‐releasing factor (CRF) stimulates adrenocorticotropic hormone (ACTH) secretion from the pituitary gland and is an essential regulator of the hypothalamic–pituitary–adrenocortical axis. Isoforms of CRF receptor are known to mediate
Externí odkaz:
https://doaj.org/article/d62f8476191e4df3820cf0ddf9656f21
Publikováno v:
International Immunology
Proteoglycan (PG) aggrecan-induced arthritis (PGIA) is a murine model of rheumatoid arthritis (RA). Although BALB/c and DBA/2 mice share the same MHC (H-2d) haplotype, the BALB/c strain is susceptible to PGIA, while DBA/2 mice are resistant. Therefor
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ef0a4442d8173b34bf77cb76819f9db1
https://doi.org/10.1093/intimm/dxp018
https://doi.org/10.1093/intimm/dxp018
Autor:
Oktávia Tarjányi, Julian Haerer, Mónika Vecsernyés, Gergely Berta, Alexandra Stayer-Harci, Bálint Balogh, Kornélia Farkas, Ferenc Boldizsár, József Szeberényi, György Sétáló
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-14 (2022)
Abstract Rat pheochromocytoma (PC12) cells were treated with the proteasome inhibitor MG-132 and morphological changes were recorded. Initially, neuronal differentiation was induced but after 24 h signs of morphological deterioration became apparent.
Externí odkaz:
https://doaj.org/article/ebb0e18ea15f423fafa627ca78a178af
Autor:
Péter Németh, Katalin Olasz, Oktavia Tarjanyi, Katalin Mikecz, Tibor T. Glant, Anita Hanyecz, Ferenc Boldizsár
Publikováno v:
BioMed Research International
BioMed Research International, Vol 2014 (2014)
BioMed Research International, Vol 2014 (2014)
Rheumatoid arthritis (RA) is a systemic autoimmune disease and its targeting of the joints indicates the presence of a candidate autoantigen(s) in synovial joints. Patients with RA show immune responses in their peripheral blood to proteoglycan (PG)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::91d9b64165d45c4457bbcec948cd4a3d
https://pubmed.ncbi.nlm.nih.gov/24605340
https://pubmed.ncbi.nlm.nih.gov/24605340
Autor:
Mónika Vecsernyés, Oktavia Tarjanyi, Gergely Berta, József Szeberényi, Marianna Pap, András Balogh, György Sétáló, Alexandra Harci
Publikováno v:
Brain research. 1520
In this work we tried to identify mechanisms that could explain how chemical inhibition of heat-shock protein 90 reduces nerve growth factor signaling in rat pheochromocytoma PC12 cells. Geldanamycin is an antibiotic originally discovered based on it
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f961d4c0cda57a7a0326a8d3739d5c7a
https://pubmed.ncbi.nlm.nih.gov/23701727
https://pubmed.ncbi.nlm.nih.gov/23701727
Autor:
Marianna Pap, Gergely Berta, József Szeberényi, Oktavia Tarjanyi, Alexandra Harci, Eszter B. Bacsa, György Sétáló, Borbála Stark
Publikováno v:
Neurochemistry international. 63(5)
The PC12 (rat pheochromocytoma) cell line is a popular model system to study neuronal differentiation. Upon prolonged nerve growth factor (NGF) exposure these tumor cells stop to divide, become polygonal, grow projections and start to look and behave
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::65baf51220c8dbe1f83f5a0487d14a99
https://pubmed.ncbi.nlm.nih.gov/23911694
https://pubmed.ncbi.nlm.nih.gov/23911694
Autor:
Katalin Olasz, Akos Hegyi, W. van Eden, Tibor T. Glant, Katalin Mikecz, Ferenc Boldizsár, Tibor A. Rauch, Katalin Kis-Toth, Oktavia Tarjanyi
Publikováno v:
Clinical and experimental immunology. 167(2)
Summary T cell receptor transgenic (TCR-Tg) mice specific for the arthritogenic 5/4E8 epitope in the G1 domain of cartilage proteoglycan were generated and back-crossed into arthritis-prone BALB/c background. Although more than 90% of CD4+ T cells of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c06ef7a561b465c4fa81dd603059fa54
https://pubmed.ncbi.nlm.nih.gov/22236012
https://pubmed.ncbi.nlm.nih.gov/22236012
Autor:
Katalin Olasz, Akos Hegyi, Katalin Mikecz, Oktavia Tarjanyi, Katalin Kis-Toth, Tibor T. Glant, Ferenc Boldizsár
Publikováno v:
Arthritis and rheumatism. 62(10)
Objective To investigate whether genetic preponderance of a T cell receptor (TCR) recognizing an arthritogenic peptide of human cartilage proteoglycan (PG) is sufficient for development of arthritis. Methods We performed a longitudinal study using BA
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cafce742cb5e4eea373f3f36d4ba20d4
https://pubmed.ncbi.nlm.nih.gov/20564001
https://pubmed.ncbi.nlm.nih.gov/20564001
Autor:
Ferenc Boldizsár, Alison Finnegan, Beata Tryniszewska, Balint Farkas, Anna Laszlo, Gabor Hutas, Gyorgy Nagyeri, Holly L. Rosenzweig, Aaron R. Mangold, Simon Lin, Tibor T. Glant, Katalin Mikecz, Oktavia Tarjanyi
Publikováno v:
Arthritis Research & Therapy
Introduction The major histocompatibility complex (H-2d) and non-major histocompatibility complex genetic backgrounds make the BALB/c strain highly susceptible to inflammatory arthritis and spondylitis. Although different BALB/c colonies develop prot
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3188d9b9e56dfe3d2c2d1b7182f220e7
http://europepmc.org/articles/PMC2688253
http://europepmc.org/articles/PMC2688253