Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Oksana Chernushevich"'
Autor:
Amina Abdul Qayum, Andrew J. Spence, Jamie Josephine Avila McLeod, Carole A. Oskeritzian, Brian O. Barnstein, Daniel Abebayehu, Gurjas S. Bajaj, Bianca Baker, Alena P. Chumanevich, Anuya Paranjape, Marcela T. Taruselli, Oksana Chernushevich, John J. Ryan
Mast cells are critical effectors of allergic disease and can be activated by IL-33, a proinflammatory member of the IL-1 cytokine family. IL-33 worsens the pathology of mast cell–mediated diseases, but therapies to antagonize IL-33 are still forth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c5a689213148feb9994e9ede4bc91b86
https://europepmc.org/articles/PMC5109997/
https://europepmc.org/articles/PMC5109997/
Autor:
Victoria A. Metelskaya, Nadezhda G. Gumanova, N. Gavrilova, Alexander Y. Kots, Oksana Chernushevich
Publikováno v:
Biomarkers : biochemical indicators of exposure, response, and susceptibility to chemicals. 22(3-4)
Noninvasive diagnostics of early stages of coronary artery disease and discrimination between various extents of vascular lesions in patients is an important clinical problem especially considering wide spread use of cholesterol lowering drugs that a
Publikováno v:
The Journal of Immunology. 194:49.16-49.16
Dexamethasone, a glucocorticosteroid known for its anti-inflammatory and immunosuppressive activities, has been shown to inhibit IgE-mediated mast cell activation. The present research investigates the role of this drug in suppressing IL-33-mediated
Publikováno v:
The Journal of Immunology. 192:54.19-54.19
Cyclosporine A (CsA), a potent calcineurin antagonist and immunosuppressant, is known to inhibit IgE-mediated mast cell activation. The present research investigates the role of this drug in suppressing IL-33-mediated mast cell activation. IL-33 is a
Publikováno v:
The Journal of Immunology. 192:54.21-54.21
Dexamethasone, a potent synthetic member of the glucocorticoid class of steroid drugs is known for its immunosuppressant and anti-inflammatory effects. The present research investigates the role of this anti-allergic drug in suppressing IL-33-mediate