Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Odra Santander"'
Autor:
Martina Valencia, Odra Santander, Eloísa Torres, Natali Zamora, Fernanda Muñoz, Rodrigo Pascual
Publikováno v:
AIMS Neuroscience, Vol 9, Iss 3, Pp 320-344 (2022)
During prenatal life, exposure to synthetic glucocorticoids (SGCs) can alter normal foetal development, resulting in disease later in life. Previously, we have shown alterations in the dendritic cytoarchitecture of Purkinje cells in adolescent rat pr
Externí odkaz:
https://doaj.org/article/854ca80187944a09841ebc63ddb4a893
Autor:
Francisca García-Rojas, Carolina Flores-Muñoz, Odra Santander, Pamela Solis, Agustín D. Martínez, Álvaro O. Ardiles, Marco Fuenzalida
Publikováno v:
Biomolecules, Vol 13, Iss 6, p 887 (2023)
Pannexin-1 (Panx1) hemichannel is a non-selective transmembrane channel that may play important roles in intercellular signaling by allowing the permeation of ions and metabolites, such as ATP. Although recent evidence shows that the Panx1 hemichanne
Externí odkaz:
https://doaj.org/article/8ecaa2306d264fb7b9d02fe0f0e00e12
Autor:
Carolina Flores-Muñoz, Francisca García-Rojas, Miguel A. Pérez, Odra Santander, Elena Mery, Stefany Ordenes, Javiera Illanes-González, Daniela López-Espíndola, Arlek M. González-Jamett, Marco Fuenzalida, Agustín D. Martínez, Álvaro O. Ardiles
Publikováno v:
Cells, Vol 11, Iss 22, p 3646 (2022)
Enhanced activity and overexpression of Pannexin 1 (Panx1) channels contribute to neuronal pathologies such as epilepsy and Alzheimer’s disease (AD). The Panx1 channel ablation alters the hippocampus’s glutamatergic neurotransmission, synaptic pl
Externí odkaz:
https://doaj.org/article/b2ec816e17904dd487b66dbd7a5d8337
Autor:
Miguel Ángel Pérez, Camila Morales, Odra Santander, Francisca García, Isabel Gómez, Valentín Peñaloza-Sancho, Pablo Fuentealba, Alexies Dagnino-Subiabre, Pablo R. Moya, Marco Fuenzalida
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 13 (2019)
Schizophrenia (SZ) is associated with changes in the structure and function of several brain areas. Several findings suggest that these impairments are related to a dysfunction in γ-aminobutyric acid (GABA) neurotransmission in brain areas such as t
Externí odkaz:
https://doaj.org/article/239005d8197d42ec83e796f39b6f1259
Autor:
Carolina Flores-Muñoz, Francisca García-Rojas, Miguel A. Perez, Odra Santander, Elena Mery, Daniela Lopez-Espíndola, Arlek M. Gonzalez-Jamett, Marco Fuenzalida, Agustín D. Martinez, Alvaro Oscar Ardiles
Enhanced activity and overexpression of Pannexin 1 (PANX1) channels contribute to neuronal pathologies, such as epilepsy and Alzheimer’s disease (AD). In the hippocampus, the PANX1 channel ablation alters glutamatergic neurotransmission, synaptic p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f0a87b890cc329f5b41d4d2f7286a402
https://doi.org/10.21203/rs.3.rs-1242197/v1
https://doi.org/10.21203/rs.3.rs-1242197/v1
Publikováno v:
Clinical Pediatric Endocrinology
Previous animal studies have indicated that excessive prenatal circulating glucocorticoid (GC) levels induced by the antenatal administration of synthetic GC (sGC) significantly alter neuronal development in the cerebellar and hippocampal neurons of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fb48270c1bc37a2ee9dff37d3384b76e
https://doi.org/10.1297/cpe.26.9
https://doi.org/10.1297/cpe.26.9
Publikováno v:
Romanian journal of morphology and embryology = Revue roumaine de morphologie et embryologie. 58(1)
Several studies have indicated that abnormal prenatal changes in the circulating glucocorticoids (GCs), induced by either maternal stress or exogenous GC administration, significantly alter the development of Purkinje cells (PCs). Among the suggested
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::e47ce8570be9d3d61a9d9af65ca45217
https://pubmed.ncbi.nlm.nih.gov/28523300
https://pubmed.ncbi.nlm.nih.gov/28523300
Publikováno v:
Journal of Neurology & Neurophysiology.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8496c4ba127dbdc3256164866e6c3093
https://doi.org/10.4172/2155-9562.c1.044
https://doi.org/10.4172/2155-9562.c1.044