Zobrazeno 1 - 10
of 115
pro vyhledávání: '"Odile, Kellermann"'
Autor:
Luiz W. Ribeiro, Mathéa Pietri, Hector Ardila-Osorio, Anne Baudry, François Boudet-Devaud, Chloé Bizingre, Zaira E. Arellano-Anaya, Anne-Marie Haeberlé, Nicolas Gadot, Sonja Boland, Stéphanie Devineau, Yannick Bailly, Odile Kellermann, Anna Bencsik, Benoit Schneider
Publikováno v:
Particle and Fibre Toxicology, Vol 19, Iss 1, Pp 1-22 (2022)
Abstract Background Epidemiological emerging evidence shows that human exposure to some nanosized materials present in the environment would contribute to the onset and/or progression of Alzheimer’s disease (AD). The cellular and molecular mechanis
Externí odkaz:
https://doaj.org/article/39138cafd6da479a9d8511efe7d61aa2
Autor:
Hélène Arnould, Vincent Baudouin, Anne Baudry, Luiz W Ribeiro, Hector Ardila-Osorio, Mathéa Pietri, Cédric Caradeuc, Cynthia Soultawi, Declan Williams, Marjorie Alvarez, Carole Crozet, Fatima Djouadi, Mireille Laforge, Gildas Bertho, Odile Kellermann, Jean-Marie Launay, Gerold Schmitt-Ulms, Benoit Schneider
Publikováno v:
PLoS Pathogens, Vol 17, Iss 10, p e1009991 (2021)
Corruption of cellular prion protein (PrPC) function(s) at the plasma membrane of neurons is at the root of prion diseases, such as Creutzfeldt-Jakob disease and its variant in humans, and Bovine Spongiform Encephalopathies, better known as mad cow d
Externí odkaz:
https://doaj.org/article/e0f84de0ab084344b839c7190a6a9010
Autor:
Juliette Ezpeleta, Vincent Baudouin, Zaira E. Arellano-Anaya, François Boudet-Devaud, Mathéa Pietri, Anne Baudry, Anne-Marie Haeberlé, Yannick Bailly, Odile Kellermann, Jean-Marie Launay, Benoit Schneider
Publikováno v:
Nature Communications, Vol 10, Iss 1, Pp 1-13 (2019)
Aβ plaques have been detected in brains of patients with prion diseases. Here, using mice, the authors show that prion infection enhances Aβ production via a PDK1-TACE mechanism and that brain deposition of Aβ induced by Aβ seeds co-transmitted w
Externí odkaz:
https://doaj.org/article/1e4e707388e247a5aac2217f5c04838e
Autor:
Benoit Schneider, Anne Baudry, Mathéa Pietri, Aurélie Alleaume-Butaux, Chloé Bizingre, Pierre Nioche, Odile Kellermann, Jean-Marie Launay
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 15 (2021)
Amyloid-based neurodegenerative diseases such as prion, Alzheimer's, and Parkinson's diseases have distinct etiologies and clinical manifestations, but they share common pathological events. These diseases are caused by abnormally folded proteins (pa
Externí odkaz:
https://doaj.org/article/b12abf07f59a4a9a925fce07329b3a9c
Autor:
Juliette Ezpeleta, François Boudet-Devaud, Mathéa Pietri, Anne Baudry, Vincent Baudouin, Aurélie Alleaume-Butaux, Nathalie Dagoneau, Odile Kellermann, Jean-Marie Launay, Benoit Schneider
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-13 (2017)
Abstract Although cellular prion protein PrPC is well known for its implication in Transmissible Spongiform Encephalopathies, its functions remain elusive. Combining in vitro and in vivo approaches, we here show that PrPC displays the intrinsic capac
Externí odkaz:
https://doaj.org/article/98b23f663c354ca58b1295e144d129b8
Publikováno v:
Frontiers in Neuroscience, Vol 13 (2019)
Serotonin transporter, SERT (SLC64A for solute carrier family 6, member A4), is a twelve transmembrane domain (TMDs) protein that assumes the uptake of serotonin (5-HT) through dissipation of the Na+ gradient established by the electrogenic pump Na/K
Externí odkaz:
https://doaj.org/article/e40116cf5a6648ba8d04b223891d6f73
Autor:
Hélène Arnould, Anne Baudry, Mathéa Pietri, Odile Kellermann, Jean-Marie Launay, Benoit Schneider
Publikováno v:
médecine/sciences
médecine/sciences, 2022, 38 (8-9), pp.643-645. ⟨10.1051/medsci/2022094⟩
médecine/sciences, 2022, 38 (8-9), pp.643-645. ⟨10.1051/medsci/2022094⟩
International audience; No abstract available
Autor:
Hélène, Arnould, Anne, Baudry, Mathéa, Pietri, Odile, Kellermann, Jean-Marie, Launay, Benoit, Schneider
Publikováno v:
Medecine sciences : M/S. 38(8-9)
Publikováno v:
Neurobiology of Disease, Vol 46, Iss 2, Pp 272-278 (2012)
With an estimated life-time prevalence of 15 to 17% and an incapacitating illness in 50% of cases, depressive spectrum disorders represent a heavy public health burden. Despite considerable efforts to underpin the molecular and cellular changes assoc
Externí odkaz:
https://doaj.org/article/72f17c6d31454beda97adc9ef84d5524
Autor:
Luiz W. Ribeiro, Mathéa Pietri, Hector Ardila-Osorio, Anne Baudry, François Boudet-Devaud, Chloé Bizingre, Zaira E. Arellano-Anaya, Anne-Marie Haeberlé, Nicolas Gadot, Sonja Boland, Stéphanie Devineau, Yannick Bailly, Odile Kellermann, Anna Bencsik, Benoit Schneider
Publikováno v:
Particle and Fibre Toxicology
Particle and Fibre Toxicology, 2022, 19 (1), pp.48. ⟨10.1186/s12989-022-00490-x⟩
Particle and Fibre Toxicology, BioMed Central, 2022, 19 (1), pp.48. ⟨10.1186/s12989-022-00490-x⟩
Particle and Fibre Toxicology, 2022, 19 (1), pp.48. ⟨10.1186/s12989-022-00490-x⟩
Particle and Fibre Toxicology, BioMed Central, 2022, 19 (1), pp.48. ⟨10.1186/s12989-022-00490-x⟩
Background Epidemiological emerging evidence shows that human exposure to some nanosized materials present in the environment would contribute to the onset and/or progression of Alzheimer’s disease (AD). The cellular and molecular mechanisms whereb