Zobrazeno 1 - 10
of 72
pro vyhledávání: '"O Tatsuzawa"'
Publikováno v:
Blood. 88:1841-1845
HRONIC GRANULOMATOUS disease (CGD) is a rare inherited disorder whereby phagocytes cannot generate superoxide anion and its derivatives to other active oxygen species that may be used for killing infectious agents. Patients with this disease have rec
Autor:
Y, Date, N, Seki, S, Kamizono, T, Higuchi, T, Hirata, K, Miyata, M, Ohkuni, O, Tatsuzawa, S, Yokota, K, Joo, K, Ueda, T, Sasazuki, A, Kimura, K, Itoh, H, Kato
Publikováno v:
Arthritis and rheumatism. 42(12)
To study polymorphisms in the 5'-flanking promoter/enhancer region of the tumor necrosis factor alpha (TNFalpha) gene and in the coding regions of HLA class I and class II genes, in order to better understand the genetic background of juvenile rheuma
Autor:
S, Kobayashi, O, Tatsuzawa
Publikováno v:
Ryoikibetsu shokogun shirizu. (25 Pt 3)
Autor:
T, Fujita, O, Tatsuzawa
Publikováno v:
Ryoikibetsu shokogun shirizu. (25 Pt 3)
Autor:
Y, Wada, N, Wada, M, Kubo, M, Nagata, O, Tatsuzawa, M, Okazaki, T, Oishi, K, Joh, T, Okabe, S, Kohno
Publikováno v:
Ryumachi. [Rheumatism]. 38(3)
We compared clinical findings in 12 cases of systemic lupus erythematosus (SLE) in boys with those in 49 cases in girls. The age at which SLE developed in boys was consistent with that of infantile SLE and there was no age specificity. Momy cases in
Autor:
T, Futatani, T, Miyawaki, S, Tsukada, S, Hashimoto, T, Kunikata, S, Arai, M, Kurimoto, Y, Niida, H, Matsuoka, Y, Sakiyama, T, Iwata, S, Tsuchiya, O, Tatsuzawa, K, Yoshizaki, T, Kishimoto
Publikováno v:
Blood. 91(2)
The B-cell defect in X-linked agammaglobulinemia (XLA) is caused by mutations in the gene for Bruton's tyrosine kinase (BTK). Using the anti-BTK monoclonal antibody (48-2H), a flow cytometric analysis of intracytoplasmic BTK protein expressed in mono
Publikováno v:
Blood. 88(5)
The cytosolic 67-kD protein in phagocytes (p67-phox) and B lymphocytes is one of essential components of the superoxide-generating system in these cells, and its defect causes an autosomal recessive type of chronic granulomatous disease (CGD). We per
Publikováno v:
Biochemical and biophysical research communications. 199(3)
Cytosolic components of human neutrophils, p47phox and p67phox, deficiencies of which lead to chronic granulomatous disease (CGD), potentiate respiratory burst oxidase translocating from cytosol to membrane upon cell stimulation. In this report we de
Autor:
S, Kobayashi, N, Wada, K, Fukunaga, Y, Saito, O, Tatsuzawa, T, Okabe, N, Kojima, K, Joh, M, Kubo
Publikováno v:
Ryumachi. [Rheumatism]. 32(2)
Thirty-seven patients with systemic JRA were analyzed. Fifty four per cent of patients had mono-cyclic systemic type. Age at onset ranged from 6.0-6.8 years (median 6.4). Boys were more affected than girls (24/11). Cardiac involvement occurred in 10
Publikováno v:
Archives of Disease in Childhood. 59:1016-1019
Two Japanese outbreaks of erythema infectiosum were investigated for evidence of human parvovirus infection by a solid phase antibody capture radioimmunoassay based on a monoclonal antibody to human parvovirus. Specific IgM and high concentrations of