Zobrazeno 1 - 7
of 7
pro vyhledávání: '"O O, Gnedina"'
Publikováno v:
Cell and Tissue Biology. 16:547-554
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::be86f42b5742a9da1a1256cf523d0a73
https://doi.org/10.1134/s1990519x22060037
https://doi.org/10.1134/s1990519x22060037
Autor:
O. O. Gnedina, M. V. Igotti
Publikováno v:
Cell and Tissue Biology. 15:236-247
To establish the mechanisms of transformed cells resistance to the histone deacetylase inhibitors (HDACi), we compared the changes of the main proliferative signaling cascades activities in cells that are sensitive or resistant to HDACi-induced apopt
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::598c5f75f3ad37276f4032c19f60a218
https://doi.org/10.1134/s1990519x21030020
https://doi.org/10.1134/s1990519x21030020
Publikováno v:
Cell and Tissue Biology. 11:42-50
Histone deacetylase inhibitors (HDIs) cause an irreversible cell cycle arrest in G1 phase and senescence of E1A + Ras transformed fibroblasts. The modulation of the antiproliferative action of the RNA synthesis inhibitor actinomycin D (AMD) with sodi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::36afbd6034e09ead7a6455240f42f78c
https://doi.org/10.1134/s1990519x17010059
https://doi.org/10.1134/s1990519x17010059
Publikováno v:
Tsitologiia. 58(10)
In the present article we have studied the combined effect of actinomycin D (AMD) with HDI sodium butyrate (NaBut) on proliferation of transformed cells. It has been shown that NaBut increased the cytotoxic effect of low concentrations of AMD (8 nM).
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::0764463132fa636149d466a86bca2751
https://pubmed.ncbi.nlm.nih.gov/30198696
https://pubmed.ncbi.nlm.nih.gov/30198696
Publikováno v:
Cell and Tissue Biology. 9:348-355
Histone deacetylase inhibitors (HDIs) cause irreversible cell cycle arrest and senescence of transformed E1A + Ras cells. The HDI sodium butyrate (NaB) causes a violation of DNA repair and, as a consequence, phosphorylation of DNA breaks marker histo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::af6880be93987119f2ceb74ccbce0be4
https://doi.org/10.1134/s1990519x15050065
https://doi.org/10.1134/s1990519x15050065
Autor:
E. A. Filippova, O. O. Gnedina, A.K. Pojidaeva, Valery A. Pospelov, S. B. Svetlikova, M.V. Igotti Abramova
Publikováno v:
The International Journal of Biochemistry & Cell Biology. 51:102-110
HDAC inhibitors (HDIs) induce irreversible cell cycle arrest and senescence in E1A+Ras expressing cells. Furthermore, HDIs activate Gadd45α/NF-κB signaling pathway to suppress apoptosis thereby promoting the cell survival. Here, to clarify the role
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7d72cf368e10ce693bf8be0eb9b942c1
https://doi.org/10.1016/j.biocel.2014.03.031
https://doi.org/10.1016/j.biocel.2014.03.031
Publikováno v:
Tsitologiia. 57(4)
Histone deacetylase inhibitors (HDIs) causes irreversible cell cycle arrest and senescence of E1A+Ras transformed cells. HDI sodium butyrate (NaB) affects the DNA repair and consequently modulates the phosphorylation of DNA breaks marker histone H2AX
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::6086c2766793dd866dbd3cc1f907ab3a
https://pubmed.ncbi.nlm.nih.gov/26349241
https://pubmed.ncbi.nlm.nih.gov/26349241