Zobrazeno 1 - 10
of 76
pro vyhledávání: '"O'Regan, D.J."'
Autor:
Hemmings, K.E., Riches-Suman, Kirsten, Bailey, M.A., O'Regan, D.J., Turner, N.A., Porter, K.E.
Yes
Increased cardiovascular morbidity and mortality in individuals with type 2 diabetes (T2DM) is a significant clinical problem. Despite advancements in achieving good glycaemic control, this patient population remains susceptible to macrovasc
Increased cardiovascular morbidity and mortality in individuals with type 2 diabetes (T2DM) is a significant clinical problem. Despite advancements in achieving good glycaemic control, this patient population remains susceptible to macrovasc
Externí odkaz:
http://hdl.handle.net/10454/18477
Autor:
Alshanwani, A.R., Riches-Suman, Kirsten, O'Regan, D.J., Wood, I.C., Turner, N.A., Porter, K.E.
Yes
Cardiovascular disease is a leading cause of morbidity and mortality. Smooth muscle cells (SMC) comprising the vascular wall can switch phenotypes from contractile to synthetic, which can promote the development of aberrant remodelling and i
Cardiovascular disease is a leading cause of morbidity and mortality. Smooth muscle cells (SMC) comprising the vascular wall can switch phenotypes from contractile to synthetic, which can promote the development of aberrant remodelling and i
Externí odkaz:
http://hdl.handle.net/10454/16246
Autor:
Riches-Suman, Kirsten, Huntriss, J., Keeble, C., Wood, I.C., O'Regan, D.J., Turner, N.A., Porter, K.E.
Yes
Type 2 diabetes mellitus prevalence is growing globally, and the leading cause of mortality in these patients is cardiovascular disease. Epigenetic mechanisms such as microRNAs (miRs) and DNA methylation may contribute to complications of ty
Type 2 diabetes mellitus prevalence is growing globally, and the leading cause of mortality in these patients is cardiovascular disease. Epigenetic mechanisms such as microRNAs (miRs) and DNA methylation may contribute to complications of ty
Externí odkaz:
http://hdl.handle.net/10454/11032
Autor:
Riches-Suman, Kirsten, Alshanwani, A.R., Warburton, P., O'Regan, D.J., Ball, S.G., Wood, I.C., Turner, N.A., Porter, K.E.
Yes
Type 2 diabetes (T2DM) promotes premature atherosclerosis and inferior prognosis after arterial reconstruction. Vascular smooth muscle cells (SMC) respond to patho/physiological stimuli, switching between quiescent contractile and activated
Type 2 diabetes (T2DM) promotes premature atherosclerosis and inferior prognosis after arterial reconstruction. Vascular smooth muscle cells (SMC) respond to patho/physiological stimuli, switching between quiescent contractile and activated
Externí odkaz:
http://hdl.handle.net/10454/10084
Autor:
Sedgwick, B., Riches-Suman, Kirsten, Bageghni, S.A., O'Regan, D.J., Porter, K.E., Turner, N.A.
Yes
Introduction Type 2 diabetes mellitus (T2DM) promotes adverse myocardial remodeling and increased risk of heart failure; effects that can occur independently of hypertension or coronary artery disease. As cardiac fibroblasts (CFs) are key ef
Introduction Type 2 diabetes mellitus (T2DM) promotes adverse myocardial remodeling and increased risk of heart failure; effects that can occur independently of hypertension or coronary artery disease. As cardiac fibroblasts (CFs) are key ef
Externí odkaz:
http://hdl.handle.net/10454/10085
Yes
Background/purpose Coronary heart disease is the leading cause of morbidity in patients with type 2 diabetes mellitus (T2DM), frequently resulting in a requirement for coronary revascularization using the internal mammary artery (IMA) or sap
Background/purpose Coronary heart disease is the leading cause of morbidity in patients with type 2 diabetes mellitus (T2DM), frequently resulting in a requirement for coronary revascularization using the internal mammary artery (IMA) or sap
Externí odkaz:
http://hdl.handle.net/10454/10086
Autor:
Roberts, A.C., Gohil, J., Hudson, L., Connolly, K., Warburton, P., Suman, R., O'Toole, P., O'Regan, D.J., Turner, N.A., Riches-Suman, Kirsten, Porter, K.E.
Yes
Type 2 diabetes (T2DM) confers increased risk of endothelial dysfunction, coronary heart disease, and vulnerability to vein graft failure after bypass grafting, despite glycaemic control. This study explored the concept that endothelial cell
Type 2 diabetes (T2DM) confers increased risk of endothelial dysfunction, coronary heart disease, and vulnerability to vein graft failure after bypass grafting, despite glycaemic control. This study explored the concept that endothelial cell
Externí odkaz:
http://hdl.handle.net/10454/10083
Periodical
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