Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Nuru Eltrich"'
Publikováno v:
Frontiers in Immunology, Vol 15 (2024)
IntroductionThe atypical chemokine receptor 2 (ACKR2) is a chemokine scavenger receptor, which limits inflammation and organ damage in several experimental disease models including kidney diseases. However, potential roles of ACKR2 in reducing inflam
Externí odkaz:
https://doaj.org/article/d789c1bfa10a4ea4928b6e3f20fb6c24
Publikováno v:
PLoS ONE, Vol 8, Iss 7, p e68167 (2013)
TNF is an important mediator of glomerulonephritis. The two TNF-receptors TNFR1 and TNFR2 contribute differently to glomerular inflammation in vivo, but specific mechanisms of TNFR-mediated inflammatory responses in glomeruli are unknown. We investig
Externí odkaz:
https://doaj.org/article/1377c766040b407d9f994ad3e2e111bf
Autor:
Andrei Bideak, Martin B. Müller, Maja T. Lindenmeyer, John M. Hoppe, Volker Vielhauer, Nuru Eltrich, Susanna Müller, Moritz Lux, Bernhard Ryffel
Publikováno v:
Kidney International
Kidney International, Nature Publishing Group, 2019, 95 (1), pp.75-93. ⟨10.1016/j.kint.2018.08.012⟩
Kidney International, Nature Publishing Group, 2019, 95 (1), pp.75-93. ⟨10.1016/j.kint.2018.08.012⟩
Tumor necrosis factor-α (TNF) is a cytokine mediating inflammatory kidney diseases such as immune complex glomerulonephritis. Its two receptors, TNFR1 and TNFR2, play distinct roles in this process, with TNFR2 strongly required for induction of dise
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::73b504717c4ddbaff16533df08e054ba
https://hal-cnrs.archives-ouvertes.fr/hal-02126385
https://hal-cnrs.archives-ouvertes.fr/hal-02126385
Autor:
Maja T. Lindenmeyer, Volker Vielhauer, Anne M Mühe, Nuru Eltrich, Bruno Luckow, Anja Wilkening, Julia Krappe
Publikováno v:
Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association. 35(2)
Background Glomerulosclerosis and tubulointerstitial fibrosis are hallmarks of chronic kidney injury leading to end-stage renal disease. Inflammatory mechanisms contribute to glomerular and interstitial scarring, including chemokine-mediated recruitm
Autor:
Nuru Eltrich, Volker Vielhauer, Martin B. Müller, Hermann Josef Gröne, Moritz Lux, Alexander Blaut, Andrei Bideak, Massimo Locati, John M. Hoppe
Publikováno v:
The American journal of pathology. 189(2)
Following renal ischemia-reperfusion injury (IRI), resolution of inflammation allows tubular regeneration, whereas ongoing inflammatory injury mediated by infiltrating leukocytes leads to nephron loss and renal fibrosis, typical hallmarks of chronic
Autor:
Nuru Eltrich, Massimo Locati, Martin B. Müller, Alexander Blaut, John M. Hoppe, Andrei Bideak, Hermann Josef Gröne, Giuseppina Federico, Volker Vielhauer
Publikováno v:
Kidney international. 93(4)
The atypical chemokine receptor 2 (ACKR2), also named D6, regulates local levels of inflammatory chemokines by internalization and degradation. To explore potential anti-inflammatory functions of ACKR2 in glomerulonephritis, we induced autologous nep
Autor:
Ramanjaneyulu Allam, Nuru Eltrich, Anela Taubitz, Volker Vielhauer, Martin Schwarz, Shrikant R. Mulay
Publikováno v:
Kidney International. 84:116-129
Renal dendritic cells (DCs) form an interstitial network contributing to inflammatory and adaptive immune responses in the kidney. The presence and functional role of DC-like glomerular CD11c(+) mononuclear phagocytes is a matter of debate. Using com
Autor:
Hans-Joachim Anders, Santhosh V. Kumar, Nicolai Burzlaff, Martin Hrabě de Angelis, Bernd Hoppe, Volker Vielhauer, Martin Herrmann, John R. Asplin, Wolfgang Hans, Jyaysi Desai, Shrikant R. Mulay, Maciej Lech, Andrew P. Evan, Nuru Eltrich, Marc Weidenbusch, Julian A. Marschner, Melissa Grigorescu, Lisa Müller, Jonathan N. Eberhard
Publikováno v:
J. Am. Soc. Nephrol. 28, 761-768 (2016)
Intrarenal crystals trigger inflammation and renal cell necroptosis, processes that involve TNF receptor (TNFR) signaling. Here, we tested the hypothesis that TNFRs also have a direct role in tubular crystal deposition and progression of hyperoxaluri
Publikováno v:
Kidney international. 86(5)
Interleukin (IL)-1β contributes to renal injury in immune complex glomerulonephritis. However, production of mature IL-1β depends on activation of the inflammasome that cleaves pro-IL-1β into its secretable form. A functional role of the NLRP3-con
Publikováno v:
PLoS ONE, Vol 8, Iss 7, p e68167 (2013)
PLoS ONE
PLoS ONE
TNF is an important mediator of glomerulonephritis. The two TNF-receptors TNFR1 and TNFR2 contribute differently to glomerular inflammation in vivo, but specific mechanisms of TNFR-mediated inflammatory responses in glomeruli are unknown. We investig
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::72ad8c25bfaeaa29d40a25111c62da0e
https://doi.org/10.5167/uzh-92775
https://doi.org/10.5167/uzh-92775