Zobrazeno 1 - 10
of 26
pro vyhledávání: '"Noriko Hamada"'
Autor:
Noriko Hamada-Kawaguchi, Beston F. Nore, Rula Zain, Ylva Engström, C. I. Edvard Smith, Daisuke Yamamoto
Publikováno v:
Frontiers in Bioscience-Landmark, Vol 28, Iss 6, p 124 (2023)
Background: Bruton’s tyrosine kinase (BTK) is a non-receptor type tyrosine kinase originally identified as the genetic signature responsible for X-linked agammaglobulinemia (XLA) when mutated. Its functional form is required for B lymphocyte matura
Externí odkaz:
https://doaj.org/article/43bb95b004f24c748b0d467d9b16ba40
Publikováno v:
PLoS ONE, Vol 10, Iss 3, p e0121484 (2015)
Drosophila Btk29A is the ortholog of mammalian Btk, a Tec family nonreceptor tyrosine kinase whose deficit causes X-linked agammaglobulinemia in humans. The Btk29AficP mutation induces multiple abnormalities in oogenesis, including the growth arrest
Externí odkaz:
https://doaj.org/article/0f219e2c799a4c899a13e2a5b776b52b
Autor:
Hossain M Nawaz, Per Kylsten, Noriko Hamada, Daisuke Yamamoto, C I Edvard Smith, Jessica M Lindvall
Publikováno v:
PLoS ONE, Vol 7, Iss 5, p e35640 (2012)
BACKGROUND: A central question within biology is how intracellular signaling pathways are maintained throughout evolution. Btk29A is considered to be the fly-homolog of the mammalian Bruton's tyrosine kinase (Btk), which is a non-receptor tyrosine-ki
Externí odkaz:
https://doaj.org/article/fe568bf33bae40ddb5db832133d95f46
Publikováno v:
Genesis (New York, N.Y. : 2000). 55(8)
Drosophila Btk29A is a Tec family nonreceptor tyrosine kinase, the ortholog of which causes X-linked agammaglobulinemia in humans when mutant. In Btk29A
Publikováno v:
Science. 343:294-297
Wnt–β-Catenin in Germ Cells The Wnt–β-catenin pathway contributes to many signaling mechanisms during organismal development and carcinogenesis by regulating both transcription and cell adhesion. Hamada-Kawaguchi et al. (p. 294 ) demonstrate th
Autor:
Mitsuhiro Wada, Akihiro Hamada, Kenichiro Nakashima, Choichiro Miyazaki, Yuichi Ohwaki, Rie Ikeda, Ryuji Akiyoshi, Noriko Hamada, Tetsuya Hamada, Jun Ishii
Publikováno v:
Iryo Yakugaku (Japanese Journal of Pharmaceutical Health Care and Sciences). 37:203-208
Calcium channel blockers (CCBs) decrease lower esophageal sphincter pressure,which may result in gastrointestinal disease.In a previous study,we observed an increased risk of gastrointestinal disease following treatment with CCBs in elderly patients.
Autor:
Masami Suganuma, Noriko Hamada, Yoshinori Asakawa, Yasuyo Nakashiba, Masakazu Sono, Hirota Fujiki, Motoo Tori, Yoko Sono, Sachiko Okabe
Publikováno v:
Tetrahedron Letters. 41:3095-3098
Isocitric acid methyl esters and their derivatives have been synthesized and their biological activities, superoxide release inhibition and tumor necrosis factor-alpha (TNFα)’s release inhibition, have been studied. Linoleic and linolenic acid der
Autor:
Masakazu Sono, Noriko Hamada, Yoko Sono, Yoshinori Asakawa, Katsuyuki Nakashima, Toshihiro Hashimoto, Motoo Tori, Mayumi Ishikawa
Publikováno v:
Tetrahedron Letters. 41:3099-3102
An anti-tumor promoter, cryptoporic acid A methyl ester, has been synthesized by 1,4-addition of the enolate derived from the methoxycarbonylmethyl ether of albicanol to methyl maleate or fumarate as well as three other diastereoisomers.
Autor:
C. I. Edvard Smith, Per Kylsten, Daisuke Yamamoto, Noriko Hamada, Jessica M. Lindvall, Hossain M. Nawaz
Publikováno v:
PLoS ONE, Vol 7, Iss 5, p e35640 (2012)
PLoS ONE
PLoS ONE
Background A central question within biology is how intracellular signaling pathways are maintained throughout evolution. Btk29A is considered to be the fly-homolog of the mammalian Bruton’s tyrosine kinase (Btk), which is a non-receptor tyrosine-k
Autor:
Yasushi Fujiwara, Wataru Matsubara, Yuji Hara, Taishi Kuwahara, Noriko Hamada, Tadafumi Jo, Ryoichi Nomoto, Koji Kodama, Hiroyuki Kawada
Publikováno v:
Nippon Ronen Igakkai Zasshi. Japanese Journal of Geriatrics. 30:130-137
A 78-year-old woman was admitted to our hospital because of disorientation and fever on January 21, 1992. Two days before admission she experienced vomiting, anorexia and general malaise. Laboratory examinations on admission disclosed a hemoglobin le