Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Noore J. Ali"'
Autor:
Noore J. Ali, Richard W. Olsen
Publikováno v:
Journal of Neurochemistry. 79:1100-1108
Functional and behavioral tolerance to chronic benzodiazepine (BZ) exposure has been associated with an uncoupling of the BZ and GABA binding sites. As in rats exposed to BZ for periods of a week or longer, recombinant GABA(A) receptors (GABARs) expr
Autor:
Michael Levine, Noore J. Ali
Publikováno v:
Developmental Neuroscience. 28:230-238
A leading hypothesis of the cause of neuronal death in Huntington’s disease (HD) is excitotoxicity, in which subpopulations of striatal neurons are hypersensitive to glutamate release due to changes in postsynaptic N-methyl-D-aspartate receptors (N
Autor:
Blake Middleton, Bonnie Wong, Nathalie Escande-Beillard, Hui Zhong, Hoa Dang, Jing Yong, Sharon Zdunowski, V. Reggie Edgerton, Daniel L. Kaufman, Niranjala J.K. Tillakaratne, Tina Bilousova, Maia Boudzinskaia, Zhongqi-Phyllis Wu, M. Selvan Joseph, Lorraine Washburn, Noore J. Ali
Publikováno v:
Journal of neuroscience research. 89(3)
Mice that are deficient in classical major histocompatibility complex class I (MHCI) have abnormalities in synaptic plasticity and neurodevelopment and have more extensive loss of synapses and reduced axon regeneration after sciatic nerve transection
Autor:
Noore J. Ali, Nik J.L. London, Windyanne Khristy, Niranjala J.K. Tillakaratne, Arlene B. Bravo, Ray D. de Leon, V. Reggie Edgerton, Hui Zhong, Roland R. Roy
GABA signaling plays an important role in the spinal cord response to injury and subsequent motor training. Since benzodiazepines are commonly used to treat muscle spasticity in spinal cord injured subjects and the gamma2 subunit of the GABA(A) recep
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::29c10bcf81613dfd1edbed3289eb4059
https://europepmc.org/articles/PMC2700157/
https://europepmc.org/articles/PMC2700157/
Autor:
Noore J, Ali, Michael S, Levine
Publikováno v:
Developmental neuroscience. 28(3)
A leading hypothesis of the cause of neuronal death in Huntington's disease (HD) is excitotoxicity, in which subpopulations of striatal neurons are hypersensitive to glutamate release due to changes in postsynaptic N-methyl-D-aspartate receptors (NMD