Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Nohela B. Arévalo"'
Autor:
Nohela B. Arévalo, Cristian M. Lamaizon, Viviana A. Cavieres, Patricia V. Burgos, Alejandra R. Álvarez, María J. Yañez, Silvana Zanlungo
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 15 (2022)
Gaucher disease (GD) is an inherited disorder caused by recessive mutations in the GBA1 gene that encodes the lysosomal enzyme β-glucocerebrosidase (β-GC). β-GC hydrolyzes glucosylceramide (GluCer) into glucose and ceramide in the lysosome, and th
Externí odkaz:
https://doaj.org/article/eda9792f0a444733a7fc9a4585bc809b
Autor:
Patricio Manque, Nohela B Arévalo, Maria I. Behrens, Ute Woehlbier, Carol D. SanMartín, Esteban Leyton, Paola Murgas, Claudia Duran-Aniotz, Bastián I Cortés, Sandra Espinoza, Wileidy Gomez, Diego Matus, José Matías Benitez, Melissa Nassif, Claudio Hetz
Publikováno v:
Journal of Alzheimer's Disease. 82:S163-S178
Background: Disturbances in the autophagy/endolysosomal systems are proposed as early signatures of Alzheimer’s disease (AD). However, few studies are available concerning autophagy gene expression in AD patients. Objective: To explore the differen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a0b5e1b155b19cfd6abb30af23427f8f
https://doi.org/10.3233/jad-201264
https://doi.org/10.3233/jad-201264
Autor:
Daniela Castillo, Gonzalo A. Farías, Carol D. SanMartín, Nicole Rogers, Carolina Delgado, Nohela B Arévalo, Maria I. Behrens
Publikováno v:
Alzheimer's & Dementia. 16
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8e7e45dbe54c904df622dca30e6dbd01
https://doi.org/10.1002/alz.045564
https://doi.org/10.1002/alz.045564
Autor:
Maria I. Behrens, Gonzalo A. Farías, Nohela B Arévalo, Luisa Herrera, Carolina Delgado, Daniela P Castillo-Godoy, Nicole K Rogers, Italo Espinoza-Fuenzalida, Carol D. SanMartín, Mauricio Henriquez
Publikováno v:
Journal of Alzheimer's disease : JAD. 82(s1)
Background: Amyloid-β peptide (Aβ) deposition in Alzheimer’s disease (AD) is due to an imbalance in its production/clearance rate. Aβ is transported across the blood-brain barrier by LRP1 and P-gp as efflux transporters and RAGE as influx transp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::62159e372f08e892c230aa5f1fe24494
https://pubmed.ncbi.nlm.nih.gov/33216035
https://pubmed.ncbi.nlm.nih.gov/33216035