Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Nobukazu Takizawa"'
Autor:
Taishin Akiyama, Takuma Shiraishi, Junwen Qin, Hiroyasu Konno, Nobuko Akiyama, Miho Shinzawa, Maki Miyauchi, Nobukazu Takizawa, Hiromi Yanai, Hiroyuki Ohashi, Etsuko Miyamoto-Sato, Hiroshi Yanagawa, Weidong Yong, Weinian Shou, Jun-Ichiro Inoue
Publikováno v:
PLoS ONE, Vol 9, Iss 5, p e95992 (2014)
Virus-derived double-stranded RNAs (dsRNAs) are sensed in the cytosol by retinoic acid-inducible gene (RIG)-I-like receptors (RLRs). These induce the expression of type I IFN and proinflammatory cytokines through signaling pathways mediated by the mi
Externí odkaz:
https://doaj.org/article/f1776162c5cc4135bc17b28a1096c26c
Autor:
Ken-ichi HOSHI, Nobukazu TAKIZAWA, Michiharu YAMADA, Toshio KURASHIMA, Shinichirou UCHIKAWA, Takashi MIURA, Masaaki JITSUHARA, Yoshikiyo KOBAYASHI
Publikováno v:
Japanese Journal of Thrombosis and Hemostasis. 33:667-674
Autor:
Taishin Akiyama, Guoying Wu, Masaaki Kurihara, Yasuhiro Kobayashi, Riko Yoshinaga, Nobukazu Takizawa, Shintaro Yagi, Mitsuru Matsumoto, Ryosuke Tateishi, Hisataka Yasuda, Yosuke Demizu, Nobuaki Yoshida, Nobuko Akiyama, Jun-ichiro Inoue, Reiko Sakamoto, Josef M. Penninger, Hiromi Yanai, Maki Miyauchi, Miho Shinzawa
Publikováno v:
The Journal of Experimental Medicine
mTEC progenitors differentiate to mature Aire+ mTECs through a pathway that initiates with RANK and LtβR signaling via the nonclassical NF-κB, followed by TRAF-6–driven maturation.
Medullary thymic epithelial cells (mTECs) expressing autoimm
Medullary thymic epithelial cells (mTECs) expressing autoimm
Autor:
Etsuko Miyamoto-Sato, Hiroyuki Ohashi, Takuma Shiraishi, Nobukazu Takizawa, Miho Shinzawa, Weidong Yong, Hiromi Yanai, Taishin Akiyama, Maki Miyauchi, Junwen Qin, Hiroshi Yanagawa, Nobuko Akiyama, Hiroyasu Konno, Weinian Shou, Jun-ichiro Inoue
Publikováno v:
PLoS ONE
PLoS ONE, Vol 9, Iss 5, p e95992 (2014)
PLoS ONE, Vol 9, Iss 5, p e95992 (2014)
Virus-derived double-stranded RNAs (dsRNAs) are sensed in the cytosol by retinoic acid-inducible gene (RIG)-I-like receptors (RLRs). These induce the expression of type I IFN and proinflammatory cytokines through signaling pathways mediated by the mi