Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Noa Kalay"'
Autor:
David E. Ochayon, Boris M. Baranovski, Peter Malkin, Ronen Schuster, Noa Kalay, Rotem Ben-Hamo, Ido Sloma, Justin Levinson, Jared Brazg, Sol Efroni, Eli C. Lewis, Uri Nevo
Publikováno v:
Cell Transplantation, Vol 25 (2016)
Immune tolerance toward “self” is critical in multiple immune disorders. While there are several mechanisms to describe the involvement of immune cells in the process, the role of peripheral tissue cells in that context is not yet clear. The theo
Externí odkaz:
https://doaj.org/article/8c074e7b554446628cf5b9dae397d835
Publikováno v:
Cell Transplantation, Vol 22 (2013)
Pancreatic islets are a highly vascularized entity, and their transplantation into diabetic individuals requires optimal revascularization. In addition, β-cells in islets are extremely sensitive to inflammation. α-1-Antitrypsin (AAT), a circulating
Externí odkaz:
https://doaj.org/article/4eb58508c0a64ecd8b422452742a818c
Autor:
Boris M. Baranovski, Omer Nisim, Ronen Schuster, Nofar Bahar, Eli C. Lewis, Galit Shahaf, Noa Kalay, Pablo Cal, Eran Schenker, Eyal Ozeri, David E. Ochayon, Mark Mizrahi, Pnina Strauss
Publikováno v:
The Journal of Pharmacology and Experimental Therapeutics
Lifelong weekly infusions of human α1-antitrypsin (hAAT) are currently administered as augmentation therapy for patients with genetic AAT deficiency (AATD). Several recent clinical trials attempt to extend hAAT therapy to conditions outside AATD, in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b04d8a1471437b9fe905b744d4cf9592
https://doi.org/10.1124/jpet.116.236067
https://doi.org/10.1124/jpet.116.236067
Autor:
Ido Sloma, Eli C. Lewis, Rotem Ben-Hamo, David E. Ochayon, Noa Kalay, Jared Brazg, Peter Malkin, Sol Efroni, Justin Levinson, Uri Nevo, Ronen Schuster, Boris M. Baranovski
Publikováno v:
Cell Transplantation, Vol 25 (2016)
Immune tolerance toward “self” is critical in multiple immune disorders. While there are several mechanisms to describe the involvement of immune cells in the process, the role of peripheral tissue cells in that context is not yet clear. The theo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::48e05b7446adc48b4039fa2dd0528a04
https://doi.org/10.3727/096368916x690809
https://doi.org/10.3727/096368916x690809
Autor:
Boris M. Baranovski, Nofar Bahar, Ido Brami, Ronen Schuster, Noa Kalay, David E. Ochayon, Mark Mizrahi, Gabriella S. Freixo-Lima, Ofer Guttman, Ziv Kaner, Eli C. Lewis
Summary One would assume that the anti-inflammatory activity of α1-anti-trypsin (AAT) is the result of inhibiting neutrophil enzymes. However, AAT exhibits tolerogenic activities that are difficult to explain by serine-protease inhibition or by redu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c704d4f3287b82b376773f5cce44aea3
https://europepmc.org/articles/PMC4298394/
https://europepmc.org/articles/PMC4298394/
Publikováno v:
Cell Transplantation, Vol 22 (2013)
Pancreatic islets are a highly vascularized entity, and their transplantation into diabetic individuals requires optimal revascularization. In addition, β-cells in islets are extremely sensitive to inflammation. α-1-Antitrypsin (AAT), a circulating
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7b03b2489b5cea42206b6149d1012b01
https://pubmed.ncbi.nlm.nih.gov/23050776
https://pubmed.ncbi.nlm.nih.gov/23050776
Autor:
Avishag Abecassis, Eli C. Lewis, Mark Mizrahi, Noa Kalay, David Sabag, Eyal Ozeri, Galit Shahaf, Efrat Ashkenazi, David E. Ochayon
Publikováno v:
Cytokine. 52:6
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::33f57a4a0f978abbd006eac07abd24a1
https://doi.org/10.1016/j.cyto.2010.07.029
https://doi.org/10.1016/j.cyto.2010.07.029
Publikováno v:
Cytokine. 52:18
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::50aff3c71a06b621a358c0a9a4e30f3a
https://doi.org/10.1016/j.cyto.2010.07.080
https://doi.org/10.1016/j.cyto.2010.07.080