Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Noa Albelda"'
Publikováno v:
Mindfulness. 9:1723-1735
A growing number of publications have been exploring the possible effects of mindfulness-based interventions on teachers’ well-being and their professional lives. Notwithstanding promising results in this domain, this paper identifies some difficul
Publikováno v:
Brain, Behavior, and Immunity. 63:35-49
Early immune activation (IA) in rodents, prenatal through the mother or early postnatal directly to the neonate, is widely used to produce behavioral endophenotypes relevant to schizophrenia and depression. Given that maternal immune response plays a
Autor:
Noa Albelda, Liron Gitter, Michal Arad, Keren Weltman, Shlomit Flaisher-Grinberg, Daphna Joel
Publikováno v:
Hormones and Behavior. 55:356-365
Life events related to the female hormonal cycle may trigger the onset of obsessive-compulsive disorder (OCD) or exacerbate symptoms in women already suffering from it. These observations suggest a possible role for ovarian hormones in the course of
Publikováno v:
Journal of Visualized Experiments : JoVE
In the signal attenuation rat model of obsessive-compulsive disorder (OCD), lever-pressing for food is followed by the presentation of a compound stimulus which serves as a feedback cue. This feedback is later attenuated by repeated presentations of
Autor:
Noa Albelda, Daphna Joel
Publikováno v:
Neuroscience. 211
During the last 30 years there have been many attempts to develop animal models of obsessive compulsive disorder (OCD), in the hope that they may provide a route for furthering our understanding and treatment of this disorder. The present review prov
Autor:
Daphna Joel, Noa Albelda
Publikováno v:
Neuroscience and biobehavioral reviews. 36(1)
During the last 30 years there have been many attempts to develop animal models of obsessive-compulsive disorder (OCD). Most models have not been studied further following the original publication, and in the past few years, most papers present studi
Publikováno v:
Psychopharmacology. 210(1)
In recent years, an increasing body of evidence points to the involvement of the glutamatergic system and specifically the glutamatergic ionotropic N-methyl-d-aspartate (NMDA) receptor in the pathophysiology of obsessive–compulsive disorder (OCD).