Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Nivia Canales"'
Autor:
Liam Kitson, Anne A. M. J. Becker, Katrin Hartmann, Michèle Bergmann, Paulina Sepulveda-Garcia, Nivia Canales, Ananda Muller
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-13 (2024)
Abstract This study aimed to evaluate the blood bacterial microbiota in healthy and febrile cats. High-quality sequencing reads from the 16S rRNA gene variable region V3-V4 were obtained from genomic blood DNA belonging to 145 healthy cats, and 140 f
Externí odkaz:
https://doaj.org/article/5022d2c5e401423cadc231e36062ddb9
Autor:
Ezequiel Hidalgo-Hermoso, Paulina Sepúlveda-García, Javier Cabello, Sebastian Celis, Carola Valencia, Carolina Ortiz, Ignacio Kemec, Dario Moreira-Arce, Miguel Orsola, Nivia Canales, Antonio Garnham, Frank Vera, Ananda Muller
Publikováno v:
Frontiers in Veterinary Science, Vol 10 (2023)
IntroductionRecent evidence shows a high diversity of infectious agents in wildlife that represent a threat to human, domestic, and wild animal health. In Chile, wild populations of the most common cervid species, pudu (Pudu puda), have been reported
Externí odkaz:
https://doaj.org/article/e7df7a76898e45aab19d348217e952eb
Autor:
Nivia Canales, Hedie Bustamante, Jennifer Wilson-Welder, Cristian Thomas, Emilio Ramirez, Miguel Salgado
Publikováno v:
Pathogens, Vol 11, Iss 5, p 510 (2022)
Digital dermatitis (DD) is a highly contagious and infectious disease in cattle which has a considerable negative economic impact worldwide, and adversely affects animal welfare. Members of the genus Treponema are the only bacterial agents for which
Externí odkaz:
https://doaj.org/article/4e8d2fec30b949bf9346baa62961d741
Autor:
Nivia Canales, Nicolas Maldonado, Cristopher Stepke, Fernanda Fredericksen, Carola Otth, Melina Villalba, Víctor H. Olavarría, Valentina Arriagada, Pablo Garcés
Publikováno v:
Developmental & Comparative Immunology. 77:23-29
Viruses have developed cellular strategies to ensure progeny survival. One of the most interesting is immune camouflage, where the virus triggers a controlled-intensity immune response that prevents total destruction of the infected cell, thus “win