Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Nithianandan Selliah"'
Autor:
Maciej Cabanski, Jennifer J. Stewart, Nithianandan Selliah, Alessandra Vitaliti, Virginia Litwin, Cherie Green, Teri Oldaker, Steve Eck
Publikováno v:
Cytometry. Part B, Clinical cytometryREFERENCES. 100(1)
As with many aspects of the validation and monitoring of flow cytometric methods, the method transfer processes and acceptance criteria described for other technologies are not fully applicable. This is due to the complexity of the highly configurabl
Autor:
Yoav Peretz, Christèle Gonneau, Leanne Flye-Blakemore, Ajay Grover, Nithianandan Selliah, Alan Lackey, Sriram Ramanan
Publikováno v:
Methods in Pharmacology and Toxicology ISBN: 9781071601709
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::44fe509abfb6d3f534554d2b0aa12882
https://doi.org/10.1007/978-1-0716-0171-6_11
https://doi.org/10.1007/978-1-0716-0171-6_11
Autor:
Steven Eck, Cherie Green, Virginia Litwin, Jennifer J. Stewart, Nithianandan Selliah, Teri Oldaker, Alessandra Vitaliti
Publikováno v:
Current Protocols in Cytometry. 87:e53
Analytical method validation provides a means to ensure that data are credible and reproducible. This unit will provide a brief introduction to analytical method validation as applied to cellular analysis by flow cytometry. In addition, the unit will
Autor:
Randy Q. Cron, Sara White, Philip W. Zoltick, Mingce Zhang, Bassel E. Sawaya, Nithianandan Selliah, Terri H. Finkel
Publikováno v:
Virology. 381(2):161-167
FOXP3 is a necessary transcription factor for the development and function of CD4+ regulatory T-cells (Tregs). The role of Tregs in HIV-1 infection remains unclear. Here, we show that expression of FOXP3 in primary human CD4 T-cells significantly inh
Autor:
Xiao Fang Yu, Jason M. Shackelford, Karuppiah Muthumani, Dana Gabuzda, Jiangfang Wang, Debra K. Shivers, Nithianandan Selliah, Eduardo O’Neill, J. Victor Garcia, Terri H. Finkel, David B. Weiner
Publikováno v:
DNA and Cell Biology. 27:267-277
Prior work has implicated viral protein R (Vpr) in the arrest of human immunodeficiency virus type 1 (HIV-1)-infected cells in the G2 phase of the cell cycle, associated with increased viral replication and host cell apoptosis. We and others have rec
Publikováno v:
Journal of Immunological Methods. 312:1-11
RNA interference (RNAi) is an evolutionarily conserved cellular defense mechanism that protects cells from hostile genes and regulates the function of normal genes during growth and development. In this study, we established proof of principle of sma
Autor:
Mingce Zhang, Hellen Kim, Hallgeir Rui, Michael Brunner, Terri H. Finkel, Dennis DeSimone, Randy Q. Cron, Nithianandan Selliah, Richard F. Ittenbach
Publikováno v:
Virology. 344:283-291
Although HIV-1 (HIV) replicates poorly in non-dividing CD4 lymphocytes, resting T cells contribute to the latent reservoir. The gammac-related cytokines reverse this block to HIV infection; however, the molecular mechanisms controlling this process a
Autor:
Randy Q. Cron, Nithianandan Selliah, Greta E. Weiss, Zhengyu Ma, Mingce Zhang, Anna Genin, Terri H. Finkel
Gene transfer into primary human CD4 T lymphocytes is a critical tool in studying the mechanism of T cell-dependent immune responses and human immunodeficiency virus-1 (HIV-1) infection. Nucleofection® is an electroporation technique that allows eff
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3155e5e6504482c35f59f581c4bc40d0
https://europepmc.org/articles/PMC4120863/
https://europepmc.org/articles/PMC4120863/
Autor:
Terri H. Finkel, Nithianandan Selliah
Publikováno v:
Cell Death & Differentiation. 8:127-136
1 Division of Rheumatology, The Children's Hospital of Philadelphia, 3516 Civic Center Blvd., Philadelphia, PA 19104, USA 2 Department of Pediatrics, University of Pennsylvania, Philadelphia, PA 19104, USA * Corresponding author: TH Finkel, Division
Autor:
Nithianandan Selliah, Terri H. Finkel
Publikováno v:
The Journal of Immunology. 160:5697-5701
In early HIV disease, immunodeficiency is characterized by the inability of CD4+ T cells to produce a critical cytokine, IL-2, and to express the receptor for IL-2 (IL-2R) in response to antigenic or mitogenic stimulation. The shared common γ-chain