Výsledky vyhledávání - "Nissinen, A. E. (Antti E.)"

Salivary IgA antibody to malondialdehyde–acetaldehyde associates with mild periodontal pocket depth

Autor: Akhi, R. (Ramin), Nissinen, A. E. (Antti E.), Wang, C. (Chunguang), Kyrklund, M. (Mikael), Paju, S. (Susanna), Mäntylä, P. (Päivi), Buhlin, K. (Kåre), Sinisalo, J. (Juha), Pussinen, P. J. (Pirkko J.), Hörkkö, S. (Sohvi)

Objective: Oxidized epitopes such as malondialdehyde–acetaldehyde (MAA) play a crucial role in the progression of atherosclerosis through activation of the humoral immune response. The exact mechanism of the association between atherosclerosis and

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______2423::8653c0e6bf8ff2a20440ffde39b9603c
http://urn.fi/urn:nbn:fi-fe2023051243948

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Compensatory IgM to the rescue:patients with selective IgA deficiency have increased natural IgM antibodies to MAA–LDL and no changes in oral microbiota

Autor: Mella, M. A. (Miia A.), Lavrinienko, A. (Anton), Akhi, R. (Ramin), Hindström, R. (Rasmus), Nissinen, A. E. (Antti E.), Wang, C. (Chunguang), Kullaa, A. (Arja), Salo, T. (Tuula), Auvinen, J. (Juha), Koskimäki, J. J. (Janne J.), Hörkkö, S. (Sohvi)

IgA is the most abundant Ab in the human body. However, most patients with selective IgA deficiency (SIgAD) are asymptomatic. IgM, and to lesser extent IgG Abs, are generally presumed to compensate for the lack of IgA in SIgAD by multiplying and adop

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______2423::0c29399389a6ac436fba262abf9bf7a6
http://urn.fi/urn:nbn:fi-fe2021062439902

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