Zobrazeno 1 - 10
of 23
pro vyhledávání: '"Ningfeng Tang"'
Publikováno v:
Pediatric Research. 92:1325-1331
Hypoxic-ischemic encephalopathy (HIE) is a devastating disease with lifelong disabilities. Hypothermia is currently the only treatment. At term, the neonatal cerebellum may be particularly vulnerable to the effects of HIE. At this time, many developm
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6c1fde7ebc98c2ea60cb041586abb999
https://doi.org/10.1038/s41390-022-01974-4
https://doi.org/10.1038/s41390-022-01974-4
Publikováno v:
Pediatric research
Background Bilirubin is produced by the breakdown of hemoglobin and is normally catabolized and excreted. Neurotoxic accumulation of serum bilirubin often occurs in premature infants. The homozygous Gunn rat lacks uridine diphosphate glucuronosyltran
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::750b49f3c5353ba0c4f14cc93e933d12
http://europepmc.org/articles/PMC8024424
http://europepmc.org/articles/PMC8024424
Autor:
Xuecheng Yang, Jiangbing Du, Jiacheng Liu, Xinyi Wang, Ningfeng Tang, Yingchun Shang, Zuyuan He
Publikováno v:
Asia Communications and Photonics Conference 2021.
A proof-of-concept 2.5D optoelectronic integration solution for CPO applications is demonstrated, based on organic substrate, silicon photonics, flip-chip. Pre-FEC BER under 0.02 is obtained for 56Gbps OOK signals, indicating feasibility for 400G.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::34f806330ebfa4ac002954df37db49e0
https://doi.org/10.1364/acpc.2021.t2d.2
https://doi.org/10.1364/acpc.2021.t2d.2
Publikováno v:
Pediatric research. 89(6)
The mechanism of bilirubin neurotoxicity is poorly understood. We hypothesize that bilirubin inhibits the function of lipid rafts (LR), microdomains of the plasma membrane critical for signal transduction. To test this hypothesis, we measured the eff
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f2e6be022b7cf3f8a38a2d09c0ab6005
https://pubmed.ncbi.nlm.nih.gov/32937649
https://pubmed.ncbi.nlm.nih.gov/32937649
Publikováno v:
Birth Defects Res
Background Exposure to ethanol during pregnancy is the cause of fetal alcohol spectrum disorder. The function of L1 cell adhesion molecule (L1), critical for proper brain development, is dependent on detergent-resistant membrane microdomains (DRM). E
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::84d31262071b36ae03520fc56b505129
https://europepmc.org/articles/PMC9741483/
https://europepmc.org/articles/PMC9741483/
Publikováno v:
Pediatric research
Background The impact of bilirubin in preterm infants is poorly understood. An animal model would assist in improving understanding. The Gunn rat lacks uridine diphosphate-glucuronylsyl transferase 1 and can be made acutely hyperbilirubinemic by inje
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::77cec3f33b09e8a0381dfe2ae970028e
https://pubmed.ncbi.nlm.nih.gov/31578041
https://pubmed.ncbi.nlm.nih.gov/31578041
Autor:
Ningfeng Tang, Min He, Kimberly White, Cynthia F. Bearer, Natalie L. Davis, Julia A. Sabatino
Publikováno v:
Pediatric research
Background Prenatal toluene exposure can cause neurodevelopmental disabilities similar to fetal alcohol syndrome. Both share neuroanatomic pathologies similar to children with mutations in L1 cell adhesion molecule (L1). L1 mediates neurite outgrowth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::92b16efc8932316b24140bd3a902adf3
http://europepmc.org/articles/PMC4929035
http://europepmc.org/articles/PMC4929035
Publikováno v:
The Cerebellum. 14:413-420
Fetal alcohol spectrum disorder (FASD) is estimated to occur in 1 % of all live births. The developing cerebellum is vulnerable to the toxic effects of alcohol. People with FASD have cerebellar hypoplasia and developmental deficits associated with ce
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bb7f36d61857d91760292b0b8c12b2d5
https://doi.org/10.1007/s12311-015-0691-7
https://doi.org/10.1007/s12311-015-0691-7
Publikováno v:
Alcoholism: Clinical and Experimental Research. 37:383-389
Background Fetal alcohol spectrum disorder is an immense public health problem. In vitro studies support the hypothesis that L1 cell adhesion molecule (L1) is a target for ethanol (EtOH) developmental neurotoxicity. L1 is critical for the development
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::279e70235ac9a5aad591fbb74f2b1582
https://doi.org/10.1111/j.1530-0277.2012.01944.x
https://doi.org/10.1111/j.1530-0277.2012.01944.x
Autor:
Stephanie Fox, Benjamin L. Farah, Min He, Cynthia F. Bearer, Alfred T. Malouf, Ningfeng Tang, Yoav Littner
Publikováno v:
Journal of Neurochemistry. 119:859-867
Fetal alcohol spectrum disorder is estimated to affect 1% of live births. The similarities between children with fetal alcohol syndrome and those with mutations in the gene encoding L1 cell adhesion molecule (L1) implicates L1 as a target of ethanol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::fd5d337ee0fcb0abbad128dfdf111b14
https://doi.org/10.1111/j.1471-4159.2011.07467.x
https://doi.org/10.1111/j.1471-4159.2011.07467.x