Zobrazeno 1 - 10
of 48
pro vyhledávání: '"Ning-hua Cui"'
Publikováno v:
Redox Biology, Vol 72, Iss , Pp 103146- (2024)
Although platelet bioenergetic dysfunction is evident early in the pathogenesis of diabetic macrovascular complications, the bioenergetic characteristics in type 2 diabetic patients who developed coronary in-stent restenosis (ISR) and their effects o
Externí odkaz:
https://doaj.org/article/e8935c8aef9d48fa8ee1445d828a07e2
Publikováno v:
Cardiovascular Diabetology, Vol 21, Iss 1, Pp 1-15 (2022)
Abstract Background Outcome prediction tools for patients with type 2 diabetes mellitus (T2DM) undergoing percutaneous coronary intervention (PCI) are lacking. Here, we developed a machine learning-based metabolite classifier for predicting 1-year ma
Externí odkaz:
https://doaj.org/article/ea0b008a4df0417f83f4043375c6274c
Publikováno v:
Redox Biology, Vol 57, Iss , Pp 102507- (2022)
Type 2 diabetes mellitus (T2DM) is a strong indicator of late stent thrombosis (LST). Platelet bioenergetic dysfunction, although critical to the pathogenesis of diabetic macrovascular complications, remains uncharacterized in T2DM patients who devel
Externí odkaz:
https://doaj.org/article/8124a245a5304691b12ee9fcc2028f4d
Publikováno v:
Cardiovascular Diabetology, Vol 19, Iss 1, Pp 1-15 (2020)
Abstract Background Little is known about whether mitochondria 8-hydroxy-2′-deoxyguanosine (8-OHdG), a biomarker of mitochondrial DNA (mtDNA) oxidative damage, contributes to the development of coronary artery disease (CAD) in diabetic patients. He
Externí odkaz:
https://doaj.org/article/d2dd7fe688764a82ba0b9865dfdc5bec
Publikováno v:
Frontiers in Endocrinology, Vol 10 (2019)
Background: Mitochondrial DNA copy number (mtDNA-CN) may contribute to the development of various cancer types in a tumor-specific manner. However, little is known about whether leukocyte mtDNA content confers susceptibility to thyroid cancer (TC). T
Externí odkaz:
https://doaj.org/article/fe7625dcf71c40638cab68bd01082dc3
Autor:
Xue-bin Wang, Ning-hua Cui, Xia-nan Liu, Jun-fen Ma, Qing-hua Zhu, Shu-ren Guo, Jun-wei Zhao, Liang Ming
Publikováno v:
Frontiers in Genetics, Vol 9 (2018)
Background: Promoter hypermethylation in death-associated protein kinase 1 (DAPK1) gene has been long linked to cervical neoplasia, but the established results remained controversial. Here, we performed a meta-analysis to assess the associations of D
Externí odkaz:
https://doaj.org/article/87146038e67747b2a6f08722d184026f
Publikováno v:
Frontiers in Physiology, Vol 8 (2017)
Inhibition of poly(ADP-ribose) polymerase (PARP) may protect against coronary artery disease (CAD) in animal models, and rs1136410, a non-synonymous single nucleotide polymorphism (SNP) in PARP-1, has a potential impact on PARP activities in vitro. T
Externí odkaz:
https://doaj.org/article/1882478a8a5c4d1699a8e93e6c3b843c
Publikováno v:
Clinical Nutrition. 39:3771-3778
Summary Background & aims The 4977-bp mitochondrial deletion (mtDNA4977 deletion), as a hallmark of mitochondrial oxidative damage, may play an important role in coronary artery disease (CAD), but its interaction with folate deficiency among diabetic
Autor:
Xue-Bin Wang, Ya-di Han, Shrestha Sabina, Ning-Hua Cui, Shuai Zhang, Ze-Jin Liu, Cong Li, Fang Zheng
Publikováno v:
PLoS ONE, Vol 11, Iss 8, p e0160449 (2016)
A previous genome-wide association study showed that a single nucleotide polymorphism (SNP) rs2107595 in histone deacetylase 9 (HDAC9) gene was associated with large artery stroke (LAS) in Caucasians. Based on the similar atherosclerotic pathogenesis
Externí odkaz:
https://doaj.org/article/7c0ea9261b2a49f3b6a75231456a85fa
Autor:
Xue-bin Wang, Chen Qiao, Li Wei, Ya-di Han, Ning-hua Cui, Zhu-liang Huang, Zu-hua Li, Fang Zheng, Ming Yan
Publikováno v:
PLoS ONE, Vol 10, Iss 12, p e0145581 (2015)
Homocysteine (Hcy) is a potential risk factor for age-related cataract (ARC). Methylenetetrahydrofolate reductase (MTHFR) is the key enzyme for Hcy metabolism, and variants of MTHFR may affect MTHFR enzyme activity. This study mainly evaluated the as
Externí odkaz:
https://doaj.org/article/278ab8b18ad94f14bf3e360a91bf0d16