Zobrazeno 1 - 10
of 20
pro vyhledávání: '"Nina E. Landvik"'
Publikováno v:
Regulatory Toxicology and Pharmacology. 95:270-279
With the emergence of nanotechnology the number of manufactured nanomaterials (MNM) in production and use is constantly increasing. Exposure of workers to MNM is of concern, because still much is unknown about health effects. MNM may have different p
Functional effect of polymorphisms in 15q25 locus on CHRNA5 mRNA, bulky DNA adducts andTP53mutations
Publikováno v:
International Journal of Cancer. 132:1811-1820
Genome-wide association studies have demonstrated that genetic polymorphisms influence the risk of developing lung cancer. Nicotinic acetylcholine receptor alpha3, alpha5 and beta4 genes (CHRNA3, CHRNA5 and CHRNB4) cluster at the 15q25.1 lung cancer
Publikováno v:
Molecular Carcinogenesis. 51:E168-E175
Interleukin-1β is a key pro-inflammatory cytokine that has been associated with chronic inflammation and inflammation-related cancer initiation and progression. There are inter-individual differences in IL1B expression which may be due to single nuc
Autor:
David Ryberg, David H. Phillips, Nina E. Landvik, Aage Haugen, Shanbeh Zienolddiny, Richard S. Houlston, Vidar Skaug
Publikováno v:
Carcinogenesis. 30:1368-1371
Genome-wide association studies have provided evidence that common variation at 5p15.33 [telomerase reverse transcriptase (TERT)-cleft lip and palate transmembrane 1-like (CLPTM1L)], 6p21.33 and 15q25.1 (CHRNA5-CHRNA3) influences lung cancer risk and
Publikováno v:
Carcinogenesis. 30:1186-1192
Epidemiological evidence suggests a relationship between chronic inflammation and lung cancer. Inflammation in the lung may be modulated by host genetic factors such as polymorphisms in inflammatory genes. Identification of polymorphisms in inflammat
Autor:
Anita Solhaug, Nana Asare, Dominique Lagadic-Gossmann, Nina E. Landvik, Morgane Gorria, Volker M. Arlt, Jørn A. Holme
Publikováno v:
Toxicology
Toxicology, Elsevier, 2007, 231 (2-3), pp.159-74. ⟨10.1016/j.tox.2006.12.009⟩
Toxicology, 2007, 231 (2-3), pp.159-74. ⟨10.1016/j.tox.2006.12.009⟩
Toxicology, Elsevier, 2007, 231 (2-3), pp.159-74. ⟨10.1016/j.tox.2006.12.009⟩
Toxicology, 2007, 231 (2-3), pp.159-74. ⟨10.1016/j.tox.2006.12.009⟩
Nitrated-polycyclic aromatic hydrocarbons (nitro-PAHs) and diesel exhaust particle extracts (DEPE) induced apoptosis in Hepa1c1c7 cells with the following potency: 1,3-dinitropyrene (1,3-DNP)>1-nitropyrene (1-NP) >> DEPE >> 1,8-dinitropyrene (1,8-DNP
Publikováno v:
Journal of human genetics. 57(11)
Interleukin-1β (IL-1β), encoded by the IL1B gene, is a cytokine important in regulation of the inflammatory response. Elevated levels of IL1B expression have been associated with risk of gastric and lung cancer. We previously reported that a certai
Autor:
Aage Haugen, Kristine H. Anmarkud, Nina E. Landvik, Vidar Skaug, Shanbeh Zienolddiny, Xavier Tekpli
Publikováno v:
Cancer immunology, immunotherapy : CII. 62(2)
Epidemiologic and experimental evidences support the concept that inflammation promotes the development and progression of cancers. Interleukins (ILs) regulate the expression of several molecules and signaling pathways involved in inflammation. High
Publikováno v:
BMC Proceedings
Active smoking accounts for 90% of lung cancer deaths, but only 15% of smokers develop lung cancer. Screening for single-nucleotide polymorphisms (SNPs) in candidate pathway genes is an approach to defining high-risk subgroup. Polymorphisms have been
Autor:
Shan Zienolddiny, Nina Iversen, Grethe Skretting, Nina E. Landvik, Benedicte Stavik, Per Morten Sandset, Christiane Filion Myklebust
Publikováno v:
Biochemical and biophysical research communications. 397(1)
Tissue factor pathway inhibitor (TFPI) is the primary physiological inhibitor of tissue factor (TF) induced coagulation. Low plasma TFPI levels have been shown to be associated with increased risk of arterial and venous thrombosis. Several clinical s