Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Nikos Trakas"'
Autor:
Hai-Feng Li, Jone Furlund Owe, Nikos Trakas, Christos Stergiou, Yu Hong, Yao-Xian Yue, Geir Olve Skeie, Fredrik Romi, Katerina Karagiorgou, Nils Erik Gilhus, Hong-Jun Hao, Paraskevi Zisimopoulou, Xiang Gao, Xian-Jun Zhang, Socrates J. Tzartos, Qi Wang
Publikováno v:
European Journal of Neurology. 24:844-850
Background and purpose Myasthenia gravis (MG) is an autoimmune disease caused by antibody mediated impairment in the neuromuscular junction. Seronegative MG (SNMG) without antibodies against acetylcholine receptor (AChR) and muscle-specific kinase (M
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::edf367a405eb359e9a6d3dc9cfb1b58d
https://doi.org/10.1111/ene.13300
https://doi.org/10.1111/ene.13300
Autor:
Erdem Tüzün, Socrates J. Tzartos, Fred D. Finkelman, Premkumar Christadoss, Nikos Trakas, Abdullah Yilmaz, Richard T. Strait, Lamprini Skriapa, Ruksana Huda, Paraskevi Zisimopoulou, Melike Küçükerden, Sevil Kabadayi
Publikováno v:
Journal of neuroimmunology.
Sera of myasthenia gravis (MG) patients with muscle-specific receptor kinase-antibody (MuSK-Ab) predominantly display the non-complement fixing IgG4 isotype. Similarly, mouse IgG1, which is the analog of human IgG4, is the predominant isotype in mice
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::49cd14534e32217f94ef1432997b54a0
https://pubmed.ncbi.nlm.nih.gov/27235354
https://pubmed.ncbi.nlm.nih.gov/27235354
Autor:
Konstantinos Lazaridis, Socrates J. Tzartos, Panagiota Evangelakou, George Lagoumintzis, Paraskevi Zisimopoulou, Labrini Skriapa, Eirini Grapsa, Ioannis Kanelopoulos, Kostas Poulas, Nikos Trakas
Publikováno v:
Annals of the New York Academy of Sciences. 1275:7-12
Myasthenia gravis (MG) is an autoimmune disorder affecting the neuromuscular junction, usually caused by autoantibodies against the acetylcholine receptor (AChR) or the muscle-specific kinase (MuSK). Our aim is the development of a therapy based on t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e42901765d9790fe63c4ab0b78912c01
https://doi.org/10.1111/j.1749-6632.2012.06788.x
https://doi.org/10.1111/j.1749-6632.2012.06788.x
Autor:
Canan Ulusoy, Erdem Tüzün, Socrates J. Tzartos, Güher Saruhan-Direskeneli, Premkumar Christadoss, Fred D. Finkelman, Ruksana Huda, Konstantinos Poulas, Selin Turan, Eunmi Kim, Vuslat Yilmaz, Richard T. Strait, Nikos Trakas, Athanasios Niarchos, Lamprini Skriapa, Paraskevi Zisimopoulou
Publikováno v:
Clinical immunology (Orlando, Fla.). 151(2)
Myasthenia gravis (MG) is an autoimmune disease characterized by muscle weakness associated with acetylcholine receptor (AChR), muscle-specific receptor kinase (MuSK) or low-density lipoprotein receptor-related protein 4 (LRP4)-antibodies. MuSK-antib
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9d1a90f5e3ba30857405e20bc82689a2
https://pubmed.ncbi.nlm.nih.gov/24589747
https://pubmed.ncbi.nlm.nih.gov/24589747
Autor:
Konstantinos, Lazaridis, Paraskevi, Zisimopoulou, George, Lagoumintzis, Labrini, Skriapa, Nikos, Trakas, Panagiota, Evangelakou, Ioannis, Kanelopoulos, Eirini, Grapsa, Kostas, Poulas, Socrates, Tzartos
Publikováno v:
Annals of the New York Academy of Sciences. 1275
Myasthenia gravis (MG) is an autoimmune disorder affecting the neuromuscular junction, usually caused by autoantibodies against the acetylcholine receptor (AChR) or the muscle-specific kinase (MuSK). Our aim is the development of a therapy based on t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::8b7c3c6127dc28d8ac94638afddb680d
https://pubmed.ncbi.nlm.nih.gov/23278571
https://pubmed.ncbi.nlm.nih.gov/23278571
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1180:304-312
The structural and quantitative characteristics of many fibroblast-secreted proteins are modified during the in vitro cellular ageing. Here we report that three polypeptides (80, 84 and 87 kDa) absent from late passage fibroblast cultures, are consti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::30348f854da72e2e3f42bcee1e1026b8
https://doi.org/10.1016/0925-4439(93)90055-6
https://doi.org/10.1016/0925-4439(93)90055-6
Autor:
Kalliopi Bitzopoulou, Kostantinos Poulas, Leslie Jacobson, Ira Gavra, Alexandros Sotiriadis, Kalliopi Kostelidou, Orestis Lazos, Paraskevi Zisimopoulou, Sotiris Sideris, Grigoris Kordas, Socrates J. Tzartos, George Lagoumintzis, Nikos Trakas
Publikováno v:
Annals of the New York Academy of Sciences. 1132
Myasthenia gravis (MG) is usually caused by autoantibodies against muscle nicotinic acetylcholine receptor (AChR), which is composed of five subunits (alpha(2)betagammadelta or alpha(2)betaepsilondelta). Current treatments, including plasmapheresis,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b961f472d70524cdb159e9e98e412f7b
https://pubmed.ncbi.nlm.nih.gov/18567880
https://pubmed.ncbi.nlm.nih.gov/18567880
Publikováno v:
Journal of neuroimmunology. 159(1-2)
Antibodies against the acetylcholine receptor (AChR) are the main pathogenic factor in myasthenia gravis (MG). Clinical improvement correlates well with a reduction in levels of circulating anti-AChR antibodies, and plasmapheresis is an efficient sho
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::623b51fae4ed145815c1711457331131
https://pubmed.ncbi.nlm.nih.gov/15652418
https://pubmed.ncbi.nlm.nih.gov/15652418
Publikováno v:
European journal of immunology. 23(8)
Monoclonal antibodies (mAb) against the main immunogenic region (MIR) of the acetylcholine receptor (AChR) are very potent in inducing antigenic modulation of the AChR in animals and in muscle cell cultures. A recombinant antibody fragment of the rat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bd11d48098fced86e7b729265544e0de
https://pubmed.ncbi.nlm.nih.gov/8344344
https://pubmed.ncbi.nlm.nih.gov/8344344
Publikováno v:
Mutation research. 256(2-6)
Fibroblast secreted proteins participate in the formation of extracellular matrix. Extracellular matrix affects growth factor action, mediates cell adhesion and supports cell growth. Structural and quantitative characteristics of secreted proteins ar
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::79bc94117317443e288519a72bd6f176
https://pubmed.ncbi.nlm.nih.gov/1722005
https://pubmed.ncbi.nlm.nih.gov/1722005