Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Nicole L. Glosson-Byers"'
Autor:
Naiara Naiana Dejani, Stephanie L. Brandt, Young Min Son, C. Henrique Serezani, Nicole L. Glosson-Byers, Annie R. Piñeros, Alexandra Ivo de Medeiros, Sue Wang
Publikováno v:
Diabetes
People with diabetes are more prone to Staphylococcus aureus skin infection than healthy individuals. Control of S. aureus infection depends on dendritic cell (DC)–induced T-helper 17 (Th17)–mediated neutrophil recruitment and bacterial clearance
Autor:
Sonia Jancar, Charles S. Dela Cruz, Flavia Sisti, Soujuan Wang, Sarah M. Sturgeon, C. Henrique Serezani, Hector R. Wong, Young Min Son, Nathaniel Andrews, José C. Alves-Filho, Lindsey D. Mayo, Fernando Q. Cunha, Stephanie L. Brandt, Nicole L. Glosson-Byers, Luciano Filgueiras
Publikováno v:
Sci Signal
Sepsis-induced organ damage is caused by systemic inflammatory response syndrome (SIRS), which results in substantial comorbidities. Therefore, it is of medical importance to identify molecular brakes that can be exploited to dampen inflammation and
Autor:
Gretta L. Stritesky, Olufolakemi Awe, Sarita Sehra, Heather A. Bruns, Duy Pham, Mark H. Kaplan, Qing Yu, Nicole L. Glosson-Byers
Publikováno v:
The Journal of Immunology. 193:2631-2640
Th17 cells are critical for the clearance of extracellular bacteria and fungi, but also contribute to the pathology of autoimmune diseases and allergic inflammation. After exposure to an appropriate cytokine environment, Th17 cells can acquire a Th1-
Autor:
Hector R. Wong, Sarah M. Sturgeon, Annie Rocio Piñeros Alvarez, Soujuan Wang, C. Henrique Serezani, Paul R. Territo, Stephanie L. Brandt, José C. Alves-Filho, Brian P. McCarthy, Nicole L. Glosson-Byers
Publikováno v:
Repositório Institucional da USP (Biblioteca Digital da Produção Intelectual)
Universidade de São Paulo (USP)
instacron:USP
Universidade de São Paulo (USP)
instacron:USP
Sepsis can induce an overwhelming systemic inflammatory response, resulting in organ damage and death. Suppressor of cytokine signaling 1 (SOCS1) negatively regulates signaling by cytokine receptors and Toll-like receptors (TLRs). However, the cellul
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8a35f0e96e3bcb8ddf8e7444902da7f1
Publikováno v:
JAK-STAT. 3(3)
STAT4 is a critical mediator of inflammatory immunity and is required for all known IL-12 biological responses, including the induction of IFN-γ and development of Th1 cells. We demonstrate that IL-23, an IL-12-related cytokine, also requires STAT4
Autor:
Baohua Zhou, Mark H. Kaplan, Evelyn T. Nguyen, Nahid Akhtar, Sarita Sehra, Weiguo Yao, Nicole L. Glosson-Byers
Publikováno v:
Journal of Allergy and Clinical Immunology. 136:433-440.e1
Background IL-9 is important for the growth and survival of mast cells. IL-9 is produced by T cells, natural killer T cells, mast cells, eosinophils, and innate lymphoid cells, although the cells required for mast cell accumulation during allergic in