Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Nicole Croci"'
Autor:
Eleni Siopi, Mehrnaz Jafarian-Tehrani, Angelo H. Cho, Michel Plotkine, Nicole Croci, Catherine Marchand-Leroux, Shadi Homsi
Publikováno v:
Journal of Neurotrauma. 28:2135-2143
Traumatic brain injury (TBI) induces both focal and diffuse lesions that are concurrently responsible for the ensuing morbidity and mortality and for which no established treatment is available. It has been recently reported that an endogenous neurop
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6aefc8b6b152982a0614d0004b157bab
https://doi.org/10.1089/neu.2010.1738
https://doi.org/10.1089/neu.2010.1738
Autor:
Michel Plotkine, Mehrnaz Jafarian-Tehrani, Catherine Marchand-Leroux, Fabiola Federico, Nicole Croci, Bruno Palmier, Shadi Homsi
Publikováno v:
Brain Research. 1291:122-132
One of the severe complications following traumatic brain injury (TBI) is cerebral edema and its effective treatment is of great interest to prevent further brain damage. This study investigated the effects of minocycline, known for its anti-inflamma
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::91c8e1f6ded10d4a5f098eae901b16f7
https://doi.org/10.1016/j.brainres.2009.07.031
https://doi.org/10.1016/j.brainres.2009.07.031
Autor:
Isabelle Margaill, Bruno Palmier, Michel Plotkine, Alexia Paucard, Fanny Taillieu, Nicole Croci
Publikováno v:
European Journal of Pharmacology. 483:259-265
The present study examined caspase activation and its modulation by nitric oxide (NO) in a model of oxidative stress induced by injection of malonate (3 micromol), a mitochondrial toxin, into rat striatum. Caspase-3-like enzymatic activity was maxima
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f76e65f9eabf24ce316e9f868aa08f6b
https://doi.org/10.1016/j.ejphar.2003.10.038
https://doi.org/10.1016/j.ejphar.2003.10.038
Publikováno v:
Experimental Neurology. 184:973-980
Poly(ADP-ribose) polymerase (PARP) was shown to be detrimental in cerebral ischemia but the mechanisms whereby PARP is deleterious have yet to be determined. They may include a role in neutrophil infiltration known to aggravate ischemic damage. In th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cce6eba384b827ab40e4b409433ed97d
https://doi.org/10.1016/s0014-4886(03)00367-4
https://doi.org/10.1016/s0014-4886(03)00367-4
Publikováno v:
Brain Research. 987:32-38
The present work examined whether polymorphonuclear neutrophil (PMN) infiltration contributes to cortical and striatal brain damage and oxidative stress in a model of transient focal cerebral ischemia. A 2-h occlusion of the left middle cerebral arte
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5b9bbd48393e9fdb3c6e7bf332a02538
https://doi.org/10.1016/s0006-8993(03)03224-4
https://doi.org/10.1016/s0006-8993(03)03224-4
Autor:
Bruno Palmier, Nicole Croci, Catherine Marchand-Verrecchia, Michel Plotkine, Pierre-Etienne Chabrier, Li Ding-Zhou, Isabelle Margaill
Publikováno v:
Journal of Pharmacology and Experimental Therapeutics. 306:588-594
Nitric oxide (NO) and reactive oxygen species are both implicated in neuronal death due to cerebral ischemia. BN 80933, an original compound associating an inhibitor of neuronal NO synthase with an antioxidant, has been shown to reduce functional and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::693efc104c7b2b43652cfbc9f2f5dcb7
https://doi.org/10.1124/jpet.103.051490
https://doi.org/10.1124/jpet.103.051490
Autor:
Li Ding-Zhou, Catherine Marchand-Verrecchia, Isabelle Margaill, Michel Plotkine, Nicole Croci
Publikováno v:
European Journal of Pharmacology. 457:137-146
The role of nitric oxide (NO) in the development of post-ischemic cerebral infarction has been extensively examined, but fewer studies have investigated its role in other outcomes. In the present study, we first determined the temporal evolution of i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ddff2d761444daa6a1ad1239d370ea6d
https://doi.org/10.1016/s0014-2999(02)02686-9
https://doi.org/10.1016/s0014-2999(02)02686-9
Autor:
Bruno Palmier, Catherine Marchand-Leroux, Mathieu Soustrat, Michel Plotkine, Haymen Girgis, Nicole Croci
Publikováno v:
Brain research. 1491
The implication of cyclooxygenase (COX) type 2 in post-traumatic consequences is so far controversial. In experimental models of traumatic brain injury (TBI), genetic disruption or pharmacological inhibition of COX-2 has been shown to be neuroprotect
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::85269e3edf8735e3d1b1570b90627e46
https://pubmed.ncbi.nlm.nih.gov/23122881
https://pubmed.ncbi.nlm.nih.gov/23122881
Autor:
Mehrnaz Jafarian-Tehrani, Catherine Marchand-Leroux, Tomaso Piaggio, Michel Plotkine, Florence Noble, Shadi Homsi, Nicole Croci
Publikováno v:
Journal of neurotrauma. 27(5)
Traumatic brain injury (TBI) causes a wide spectrum of consequences, such as microglial activation, cerebral inflammation, and focal and diffuse brain injury, as well as functional impairment. In this study we aimed to investigate the effects of acut
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e2d91d8c05cd59ddec37a033df4ccf6b
https://pubmed.ncbi.nlm.nih.gov/20166806
https://pubmed.ncbi.nlm.nih.gov/20166806
Autor:
Virginie Escriou, Hervé Rhinn, Daniel Scherman, Catherine Marchand-Leroux, Nicole Croci, Michel Plotkine
Publikováno v:
BMC Molecular Biology
BMC Molecular Biology, Vol 9, Iss 1, p 62 (2008)
BMC Molecular Biology, Vol 9, Iss 1, p 62 (2008)
Background Traumatic brain injury models are widely studied, especially through gene expression, either to further understand implied biological mechanisms or to assess the efficiency of potential therapies. A large number of biological pathways are
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8a57a8e52d79260e6da25e6ba31d7968
https://pubmed.ncbi.nlm.nih.gov/18611280
https://pubmed.ncbi.nlm.nih.gov/18611280