Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Nichole L. Dudek"'
Publikováno v:
Brain Pathology. 20:176-189
Huntington's disease (HD) is a neurodegenerative disease caused by mutant huntingtin protein containing an expanded polyglutamine tract, which may cause abnormal protein-protein interactions such as increased association with calmodulin (CaM). We pre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::29c71e0b002155fa37efc9ffa7e95958
https://doi.org/10.1111/j.1750-3639.2008.00258.x
https://doi.org/10.1111/j.1750-3639.2008.00258.x
Publikováno v:
The Journal of Neuroscience. 29:11550-11559
Huntington's disease (HD) is an autosomal dominant neurodegenerative disorder, caused by a polyglutamine expansion in the huntingtin protein (htt). Increasing evidence suggests that transglutaminase (TGase) plays a critical role in the pathophysiolog
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ec04002cbc8ac919db8c39e305274f50
https://doi.org/10.1523/jneurosci.3307-09.2009
https://doi.org/10.1523/jneurosci.3307-09.2009
Publikováno v:
Journal of Pharmacology and Experimental Therapeutics. 326:153-162
Transglutaminase (TGase)-induced activation of small G proteins via 5-hydroxytryptamine (HT)(2A) receptor signaling leads to platelet aggregation (Cell 115:851-862, 2003). We hypothesize that stimulation of 5-HT(2A) receptors in neurons activates TGa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::975b0fecc92267221b7a71de07e8c393
https://doi.org/10.1124/jpet.107.135046
https://doi.org/10.1124/jpet.107.135046
Publikováno v:
The FASEB Journal. 29
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5edd91c9232f91a11ef290981294e30d
https://doi.org/10.1096/fasebj.29.1_supplement.860.8
https://doi.org/10.1096/fasebj.29.1_supplement.860.8
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 309(2)
The role of serotonin in the hemodynamic response to blood loss remains controversial. Caudal raphe serotonin neurons are activated during hypotensive hemorrhage, and their destruction attenuates sympathetic increases following blood loss in unanesth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bce809fe7e96e5ee6e4e3aae54572e7c
https://pubmed.ncbi.nlm.nih.gov/25980022
https://pubmed.ncbi.nlm.nih.gov/25980022
Publikováno v:
Journal of Neuropathology & Experimental Neurology. 64:58-65
The most prominent neuropathologic hallmarks of Huntington disease (HD) are cortical and striatal perinuclear cytoplasmic aggregates and intranuclear inclusions of mutant huntingtin. Our laboratory previously demonstrated that huntingtin protein colo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e6d93a7e37c38063343e123c7d06107b
https://doi.org/10.1093/jnen/64.1.58
https://doi.org/10.1093/jnen/64.1.58
Publikováno v:
The FASEB Journal. 26
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2710c104d243ff19bf012acaf7ab8a96
https://doi.org/10.1096/fasebj.26.1_supplement.1080.3
https://doi.org/10.1096/fasebj.26.1_supplement.1080.3
Publikováno v:
The FASEB Journal. 24
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0c91871fa0107903b61c260babf34bf3
https://doi.org/10.1096/fasebj.24.1_supplement.578.7
https://doi.org/10.1096/fasebj.24.1_supplement.578.7
Publikováno v:
Psychopharmacology. 213(2-3)
Serotonin and especially serotonin 2A (5-HT(2A)) receptor signaling are important in the etiology and treatment of schizophrenia and affective disorders. We previously reported a novel 5-HT(2A) receptor effector, increased transglutaminase (TGase)-ca
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7528e203245b6a9f9e354aef6197d35e
https://pubmed.ncbi.nlm.nih.gov/20717650
https://pubmed.ncbi.nlm.nih.gov/20717650
Publikováno v:
Brain Pathology (Zurich, Switzerland)
Huntington's disease (HD) is a neurodegenerative disease caused by mutant huntingtin protein containing an expanded polyglutamine tract, which may cause abnormal protein-protein interactions such as increased association with calmodulin (CaM). We pre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::f3548b782b8a55e925fe28dd762e4008
https://pubmed.ncbi.nlm.nih.gov/19338577
https://pubmed.ncbi.nlm.nih.gov/19338577