Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Nicholas Bodkin"'
Publikováno v:
Genome Biology, Vol 25, Iss 1, Pp 1-43 (2024)
Abstract Background Cells and tissues have a remarkable ability to adapt to genetic perturbations via a variety of molecular mechanisms. Nonsense-induced transcriptional compensation, a form of transcriptional adaptation, has recently emerged as one
Externí odkaz:
https://doaj.org/article/372d7f36ae9c4c009d428352f3921309
Autor:
Nicholas Bodkin, Melissa Ross, Micah T. McClain, Emily R. Ko, Christopher W. Woods, Geoffrey S. Ginsburg, Ricardo Henao, Ephraim L. Tsalik
Publikováno v:
Genome Medicine, Vol 14, Iss 1, Pp 1-14 (2022)
Abstract Background Measuring host gene expression is a promising diagnostic strategy to discriminate bacterial and viral infections. Multiple signatures of varying size, complexity, and target populations have been described. However, there is littl
Externí odkaz:
https://doaj.org/article/4bd42597d1644ff6a26c2412f38deb1c
Autor:
Syed Faaiz Enam, Sajidur Rahman Kader, Nicholas Bodkin, Johnathan G. Lyon, Mark Calhoun, Cesar Azrak, Pooja Munnilal Tiwari, Daryll Vanover, Haichen Wang, Philip J. Santangelo, Ravi Venkat Bellamkonda
Publikováno v:
Journal of Neuroinflammation, Vol 17, Iss 1, Pp 1-24 (2020)
Abstract Background Appropriately modulating inflammation after traumatic brain injury (TBI) may prevent disabilities for the millions of those inflicted annually. In TBI, cellular mediators of inflammation, including macrophages and microglia, posse
Externí odkaz:
https://doaj.org/article/12bd5e2f3a6341a8ac7135fd9d2a035c
Autor:
Cesar Azrak, Haichen Wang, Mark Calhoun, Daryll Vanover, Syed Faaiz Enam, Johnathan G. Lyon, Nicholas Bodkin, Philip J. Santangelo, Pooja M. Tiwari, Ravi V. Bellamkonda, Sajidur Rahman Kader
Publikováno v:
Journal of Neuroinflammation, Vol 17, Iss 1, Pp 1-24 (2020)
Journal of Neuroinflammation
Journal of Neuroinflammation
BackgroundAppropriately modulating inflammation after traumatic brain injury (TBI) may prevent disabilities for the millions of those inflicted annually. In TBI, cellular mediators of inflammation, including macrophages and microglia, possess a range
Publikováno v:
Open Forum Infectious Diseases
Background Host gene expression has emerged as a promising diagnostic strategy to discriminate bacterial and viral infection. Multiple gene signatures of varying size and complexity have been developed in various clinical populations. However, there