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pro vyhledávání: '"Nicholas A. Ingoglia"'
Autor:
Nicholas A. Ingoglia
Publikováno v:
Methods in Molecular Biology ISBN: 9781071629413
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d419397a0892b07a9d5ce41eb5eea810
https://doi.org/10.1007/978-1-0716-2942-0_4
https://doi.org/10.1007/978-1-0716-2942-0_4
Autor:
Nicholas A Ingoglia, Binta Jalloh
Publikováno v:
Neural Regeneration Research, Vol 11, Iss 3, Pp 390-391 (2016)
Externí odkaz:
https://doaj.org/article/3df32a36be334d6cb08bc07fddc03463
Autor:
Nicholas A. Ingoglia
Publikováno v:
Academic Medicine. 84:464-467
Most graduate schools associated with medical schools offer programs leading to the PhD degree but pay little attention to master's programs. This is unfortunate because many university graduates who are interested specifically in biomedical rather t
Autor:
Nicholas A, Ingoglia
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 1337
Transfer RNA-mediated posttranslational protein modification by arginine has been demonstrated in vitro in axoplasm extruded from the giant axons of squid and in injured and regenerating vertebrate nerves. In nerve and axoplasm, the highest activity
Autor:
Nicholas A. Ingoglia
Publikováno v:
Methods in Molecular Biology ISBN: 9781493929344
Transfer RNA-mediated posttranslational protein modification by arginine has been demonstrated in vitro in axoplasm extruded from the giant axons of squid and in injured and regenerating vertebrate nerves. In nerve and axoplasm, the highest activity
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ab5acb17d7eaeff1c84900623cad6ac8
https://doi.org/10.1007/978-1-4939-2935-1_4
https://doi.org/10.1007/978-1-4939-2935-1_4
Publikováno v:
Experimental Neurology. 167:451-455
Injury to axons in the CNS leads to little regenerative repair and loss of function. Conversely, injury to axons in the PNS results in vigorous regrowth of severed axons, usually with restoration of function. This difference is generally attributed t
Publikováno v:
Journal of Neuroscience Methods. 96:19-23
Synaptic terminals are sites of high metabolic activity and thus are particularly vulnerable to oxidative stress. Oxidative damage to proteins can be toxic to neurons and may cause irreversible cell damage and neurodegeneration. A neuroprotective mec
Publikováno v:
Neurochemical Research. 25:51-58
The N-terminal, posttranslational arginylation of proteins is ubiquitous in eukaryotic cells. Previous experiments, using purified components of the reaction incubated in the presence of exogenous substrates, have shown that only those proteins conta
Publikováno v:
Neurochemical Research. 23:1411-1420
While the posttranslational N-terminal arginylation of proteins has been demonstrated in a variety of eukaryotic cells including neurons and their axons, the targets of the reaction are poorly understood. Several lines of evidence suggest that arginy
Publikováno v:
Neurochemical Research. 19:105-110
The tRNA mediated, posttranslational, N-terminal arginylation of proteins occurs in all eukaryotic cells. In nervous tissue, these reactions can be inhibited by endogenous molecules with a molecular weight of between one thousand and five thousand. I