Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Nga Thi, Trinh"'
Autor:
Kyung Eun Lee, In Ah Choi, Ju-Yang Jung, Ha Rim Yeon, Hyoun-Ah Kim, Nga Thi Trinh, Hyun Jeong Kim, Joohee Kim, Woorim Kim
Publikováno v:
Pharmacogenetics and Genomics. 32:10-15
Objectives Nuclear factor of activated T cells C2 (NFATC2) is known as a member of the transcription family and enhances tumor necrosis factor-alpha (TNF-α) synthesis in human T cells at the gene transcription level. Although NFATC2 has a potential
Autor:
Sang Oh Kang, Hyun Jeong Kim, Hyoun-Ah Kim, Ju-Yang Jung, Nga Thi Trinh, In Ah Choi, Joohee Kim, Yunjeong Choi, Kyung Eun Lee, Woorim Kim, Kyung Hyun Min
Publikováno v:
Pharmacogenomics and Personalized Medicine. 13:81-88
Purpose TNF-α is a transmembrane protein which requires cleavage by ADAM17 in order to act systemically. The activation of ADAM17 to generate soluble TNF‑α results in an increased inflammatory activity. We hypothesized that variants spanning the
Autor:
Woorim, Kim, Soo, Jin Oh, Nga, Thi Trinh, Jin, Yeon Gil, In, Ah Choi, Ji, Hyoun Kim, Joo, Hee Kim, Ju-Yang, Jung, Jinhyun, Kim, Hyoun-Ah, Kim, Kyung, Eun Lee
Publikováno v:
International Immunopharmacology. 111:109094
This study was designed to investigate the effects of polymorphisms in RETN on remission in RA patients receiving TNF-α inhibitors. In addition, machine learning algorithms were trained to predict remission. Ten single-nucleotide polymorphisms were
Autor:
Bomi Jeong, Kyung Eun Lee, Myung Koo Lee, Tae Young Heo, Hyun Jeong Kim, Young Sook Ku, Nga Thi Trinh, Ju Young Kim, Woorim Kim
Publikováno v:
Diabetes Research and Clinical Practice. 154:116-123
Aim To examine the effect of oral diabetes medication on the risk of dementia in an elderly cohort with type 2 diabetes. Methods This was a population-based cohort study using the Korean National Health Insurance claims data from 2002 to 2013. Elderl
Autor:
Ju-Yang Jung, Woorim Kim, Kyung Hyun Min, Hyun Jeong Kim, Sang Oh Kang, Ha Rim Yeon, Kyung Eun Lee, Nga Thi Trinh, Joohee Kim, Hyoun-Ah Kim, In Ah Choi
Background: Despite the improvement from the introduction of tumor necrosis factor inhibitors (TNFi) in the rheumatoid arthritis (RA), TNFi therapy fails for more than 30% or results in a partial response. Thus, we aimed to explore treatment marker b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a73d898074a91b6c4f9d77cff6243ed9
https://doi.org/10.21203/rs.2.24442/v1
https://doi.org/10.21203/rs.2.24442/v1
Autor:
Hyun Jeong, Kim, Nga Thi, Trinh, Yunjeong, Choi, Woorim, Kim, Kyung Hyun, Min, Sang Oh, Kang, Joo Hee, Kim, Hyoun-Ah, Kim, Ju-Yang, Jung, In Ah, Choi, Kyung Eun, Lee
Publikováno v:
Pharmacogenomics and Personalized Medicine
Purpose TNF-α is a transmembrane protein which requires cleavage by ADAM17 in order to act systemically. The activation of ADAM17 to generate soluble TNF‑α results in an increased inflammatory activity. We hypothesized that variants spanning the
Akademický článek
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