Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Newshan Behrangi"'
Autor:
Luisa Müller, Nicole Power Guerra, Anna Schildt, Tobias Lindner, Jan Stenzel, Newshan Behrangi, Carina Bergner, Teresa Alberts, Daniel Bühler, Jens Kurth, Bernd Joachim Krause, Deborah Janowitz, Stefan Teipel, Brigitte Vollmar, Angela Kuhla
Publikováno v:
Biomolecules, Vol 13, Iss 5, p 769 (2023)
Obesity is characterized by immoderate fat accumulation leading to an elevated risk of neurodegenerative disorders, along with a host of metabolic disturbances. Chronic neuroinflammation is a main factor linking obesity and the propensity for neurode
Externí odkaz:
https://doaj.org/article/3becc8f178f141b2869b43c9334151e6
Autor:
Julia Nedelcu, Christin Reinbach, Philipp Riedler, Matthias Brendel, Axel Rominger, Joel Kaye, Newshan Behrangi, Zhan Jiangshan, Christoph Schmitz, Markus Kipp
Publikováno v:
Neurobiology of Disease, Vol 134, Iss , Pp - (2020)
Accumulating evidence suggests that a degenerative processes within the brain can trigger the formation of new, focal inflammatory lesions in Multiple Sclerosis (MS). Here, we used a novel pre-clinical MS animal model to test whether the amelioration
Externí odkaz:
https://doaj.org/article/f5504117da3e49749314902ce6884ca3
Publikováno v:
Cells, Vol 9, Iss 4, p 843 (2020)
Multiple sclerosis (MS) is a chronic inflammatory demyelinating disease of the central nervous system. Various pre-clinical models with different specific features of the disease are available to study MS pathogenesis and to develop new therapeutic o
Externí odkaz:
https://doaj.org/article/b06286385cb44f1fba30e72cbf5b9844
Autor:
Newshan Behrangi, Nabiyollah Namvar, Mitra Ataei, Sakineh Dizaji, Golshid Javdani, Mahommad Hossein Sanati
Publikováno v:
Acta Medica Iranica, Vol 55, Iss 9 (2017)
Matrix metalloproteinase-9 (GELB) as a member of gelatinases plays key role in the destruction of blood-brain barrier (BBB), T cells migration into the CNS, and demyelination induction. Considering remyelination induction in response to tart cherry e
Externí odkaz:
https://doaj.org/article/a4d637bf415e47ff861eaa2e130b5f84
Publikováno v:
Cells, Vol 8, Iss 1, p 24 (2019)
Multiple sclerosis (MS) is a neuroinflammatory disorder of the central nervous system (CNS), and represents one of the main causes of disability in young adults. On the histopathological level, the disease is characterized by inflammatory demyelinati
Externí odkaz:
https://doaj.org/article/1190a2dbda374d6ea9cb1f2f8faaee48
Autor:
Newshan Behrangi, Leo Heinig, Linda Frintrop, Emily Santrau, Jens Kurth, Bernd Krause, Dimitrinka Atanasova, Tim Clarner, Athanassios Fragoulis, Markus Joksch, Henrik Rudolf, Sven G. Meuth, Sarah Joost, Markus Kipp
Publikováno v:
Proceedings of the National Academy of Sciences. 119
Multiple sclerosis (MS), an autoimmune-driven, inflammatory demyelinating disease of the central nervous system (CNS), causes irreversible accumulation of neurological deficits to a variable extent. Although there are potent disease-modifying agents
Publikováno v:
Journal of Molecular Neuroscience
Oligodendrocytes, the myelinating cells of the central nervous system, orchestrate several key cellular functions in the brain and spinal cord, including axon insulation, energy transfer to neurons, and, eventually, modulation of immune responses. Th
Autor:
Joel Kaye, Julia Nedelcu, Zhan Jiangshan, Christoph Schmitz, Axel Rominger, Philipp Riedler, Christin Reinbach, Matthias Brendel, Markus Kipp, Newshan Behrangi
Publikováno v:
Neurobiology of Disease, Vol 134, Iss, Pp-(2020)
Nedelcu, Julia; Reinbach, Christin; Riedler, Philipp; Brendel, Matthias; Rominger, Axel; Kaye, Joel; Behrangi, Newshan; Jiangshan, Zhan; Schmitz, Christoph; Kipp, Markus (2020). Laquinimod ameliorates secondary brain inflammation. Neurobiology of disease, 134, p. 104675. Elsevier 10.1016/j.nbd.2019.104675
Nedelcu, Julia; Reinbach, Christin; Riedler, Philipp; Brendel, Matthias; Rominger, Axel; Kaye, Joel; Behrangi, Newshan; Jiangshan, Zhan; Schmitz, Christoph; Kipp, Markus (2020). Laquinimod ameliorates secondary brain inflammation. Neurobiology of disease, 134, p. 104675. Elsevier 10.1016/j.nbd.2019.104675
Accumulating evidence suggests that a degenerative processes within the brain can trigger the formation of new, focal inflammatory lesions in Multiple Sclerosis (MS). Here, we used a novel pre-clinical MS animal model to test whether the amelioration
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6ad17cfbe433cd34d307102053d38271
Autor:
Anja K. E. Horn, Markus Kipp, Felix Schweiger, Vladislav Yakimov, Christoph Schmitz, Tanja Hochstrasser, Jiangshan Zhan, Newshan Behrangi
Publikováno v:
Histochemistry and cell biology. 152(2)
Oligodendrocyte degeneration is a hallmark of multiple sclerosis pathology, and protecting oligodendrocytes and myelin is likely to be of clinical relevance. Traditionally, oligodendrocyte and myelin degeneration are viewed as a direct consequence of