Zobrazeno 1 - 10 of 11 pro vyhledávání: '"Neurotoxins/metabolism"'
1
Detection of VAMP Proteolysis by Tetanus and Botulinum Neurotoxin Type B In Vivo with a Cleavage-Specific Antibody
Autor: Federico Fabris, Petra Šoštarić, Ivica Matak, Thomas Binz, Anna Toffan, Morena Simonato, Cesare Montecucco, Marco Pirazzini, Ornella Rossetto
Publikováno v: International Journal of Molecular Sciences; Volume 23; Issue 8; Pages: 4355
Tetanus and Botulinum type B neurotoxins are bacterial metalloproteases that specifically cleave the vesicle-associated membrane protein VAMP at an identical peptide bond, resulting in inhibition of neuroexocytosis. The minute amounts of these neurot
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3f417d9e71a169194ace92f128b5a4c7
https://pubmed.ncbi.nlm.nih.gov/35457172
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2
The dynamics of autism spectrum disorders
Publikováno v: International Journal of Environmental Research and Public Health. 10(8):3384-3408
In recent years concern has risen about the increasing prevalence of Autism Spectrum Disorders (ASD). Accumulating evidence shows that exposure to neurotoxic compounds is related to ASD. Neurotransmitters might play a key role, as research has indica
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______4612::318170448c42034a4d8b1c1a43ccfe79
https://research.vu.nl/en/publications/791a5dc9-f9c6-4fa5-bdfd-20d839ca5963
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3
The Translocation Domain of Botulinum Neurotoxin A Moderates the Propensity of the Catalytic Domain to Interact with Membranes at Acidic pH
Autor: Patrick England, Julien Barbier, Sylvain Pichard, Anne Araye, Javier Pérez, Alexandre Chenal, Bruno Baron, Amélie Goudet, Daniel Gillet, Sophie Zinn-Justin
Publikováno v: PLoS ONE
PLoS ONE, 2016, 11 (4), pp.e0153401. ⟨10.1371/journal.pone.0153401⟩
PLoS ONE, Vol 11, Iss 4, p e0153401 (2016)
PLoS ONE, Public Library of Science, 2016, 11 (4), pp.e0153401. ⟨10.1371/journal.pone.0153401⟩
International audience; Botulinum neurotoxin A (BoNT/A) is composed of three domains: a catalytic domain (LC), a translocation domain (H N) and a receptor-binding domain (H C). Like most bacterial toxins BoNT/A is an amphitropic protein, produced in
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::54fbe07a82d025638b54118c5f0f0e50
https://hal-pasteur.archives-ouvertes.fr/pasteur-01378747
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4
Maurotoxin, a four disulfide bridge toxin fromScorpio maurusvenom: purification, structure and action on potassium channels
Autor: R. Kharrat, Hervé Rochat, M. El Ayeb, Marcel Crest, J. Van Rietschoten, F. Sampieri, R. Oughideni, Marie-France Martin-Eauclaire, Pascal Mansuelle
Publikováno v: FEBS Letters
FEBS Letters, 1997, 406 (3), pp.284-290. ⟨10.1016/S0014-5793(97)00285-8⟩
International audience; A new toxin acting on K+ channels, maurotoxin (MTX), has been purified to homogeneity from the venom of the chactoid scorpion Scorpio maurus. MTX is a basic single chain 34 amino acid residue polypeptide, amidated at its C ter
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::db8bd65da81e46041a5b7794fcfc6635
https://doi.org/10.1016/s0014-5793(97)00285-8
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5
The dynamics of autism spectrum disorders: how neurotoxic compounds and neurotransmitters interact
Autor: Madeleine R. Brouns, Ilona Quaak, Margot van de Bor
Publikováno v: International Journal of Environmental Research and Public Health
International Journal of Environmental Research and Public Health, Vol 10, Iss 8, Pp 3384-3408 (2013)
Quaak, I, Brouns, M R & Van de Bor, M 2013, ' The dynamics of autism spectrum disorders : how neurotoxic compounds and neurotransmitters interact ', International Journal of Environmental Research and Public Health, vol. 10, no. 8, pp. 3384-408 . https://doi.org/10.3390/ijerph10083384
In recent years concern has risen about the increasing prevalence of Autism Spectrum Disorders (ASD). Accumulating evidence shows that exposure to neurotoxic compounds is related to ASD. Neurotransmitters might play a key role, as research has indica
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::abd54bb276b2de5157475a1e181747cb
https://pubmed.ncbi.nlm.nih.gov/27918486
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6
CA150 expression delays striatal cell death in overexpression and knock-in conditions for mutant huntingtin neurotoxicity
Autor: Ashwani Kumar Thakur, Emmanuel Brouillet, Christian Neri, James Pearson, Patrick Aebischer, Margarita Arango, Sébastien Holbert, Etienne Régulier, Dania Zala, Nicole Déglon, Ronald Wetzel
Publikováno v: Journal of Neuroscience
Journal of Neuroscience, 2006, 26 (17), pp.4649-4659. ⟨10.1523/JNEUROSCI.5409-05.2006⟩
Journal of Neuroscience, Society for Neuroscience, 2006, 26 (17), pp.4649-4659. ⟨10.1523/JNEUROSCI.5409-05.2006⟩
Transcriptional dysregulation caused by expanded polyglutamines (polyGlns) in huntingtin (htt) may be central to cell-autonomous mechanisms for neuronal cell death in Huntington's disease (HD) pathogenesis. We hypothesized that these mechanisms may i
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c07f4e24a47f4a28df9ca507139174fb
https://hal.inrae.fr/hal-02666883
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9
Neurotoxicity and neuroprotection in Parkinson's disease
Autor: Lange, Klaus W., Youdim, M. B., Riederer, Peter
Recent findings of impaired mitochondrial function, altered iron metabolism and increased lipid peroxidation in the substantia nigra in Parkinson's disease emphasize the significance of oxidative stress and free radical formation in the pathogenesis
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::25ee464ad67403000c9b7047fbeefed6
https://epub.uni-regensburg.de/25435/
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