Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Nestor Morales-Peza"'
Autor:
Victoria Juarez, Prasert Auewarakul, Alejandro García-Carrancá, Nestor Morales-Peza, Angel Cid-Arregui
Publikováno v:
Virology. 294(1):135-140
Human papillomavirus type 18 is a causative agent of epithelial cancers in the uterine cervix. We show here that estrogen and progesterone activate β-galactosidase expression from the early promoter of this virus in the genital epithelia of transgen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9f58d217e51de8f069d7421a528ff5cc
Autor:
Cristina Fernandez-Mejia, Nestor Morales-Peza, Vicente Díaz-Sánchez, Guillermo Romero-Navarro, Sumiko Morimoto
Publikováno v:
Endocrinology. 142:1442-1447
Coexistence of hyperinsulinemia and hyperandrogenism in women has been frequently described. Most of the studies addressing this issue have focused on the mechanisms by which insulin produces hyperandrogenism. In the present study, we analyzed the ef
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a8b7ab9f71e5b6b29b81f3a178c4c3ed
https://doi.org/10.1210/endo.142.4.8069
https://doi.org/10.1210/endo.142.4.8069
Autor:
Olga Medina-Martínez, Françoise Thierry, Nestor Morales-Peza, Moshe Yaniv, Alejandro García-Carrancá
Publikováno v:
Virology. 217(1):392-396
We determined that the human papillomavirus type 18 (HPV18) regulatory region contains one functional GRE sequence that interacts with the glucocorticoid receptor. This sequence conferred a moderate hormonal activation to the HPV18 P 105 promoter. Tw
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a7cde462d1e85e715438a093ddeea866
Autor:
Patricio Gariglio, Rita Villanueva, Angel Cid-Arregui, Esther López-Bayghen, Irma Castelán-Sánchez, Rocio Tapia, Nestor Morales-Peza, Alejandro García-Carrancá, Enrique García-Villa
Publikováno v:
BMC Cancer, Vol 6, Iss 1, p 218 (2006)
BMC Cancer
BMC Cancer
BackgroundHigh risk HPVs are causative agents of anogenital cancers. Viral E6 and E7 genes are continuously expressed and are largely responsible for the oncogenic activity of these viruses. Transcription of the E6 and E7 genes is controlled by the v
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0f20102f4657cc02a3ec911673ecd632