Zobrazeno 1 - 10
of 5 921
pro vyhledávání: '"Neointimal hyperplasia"'
Publikováno v:
陆军军医大学学报, Vol 46, Iss 12, Pp 1403-1409 (2024)
Objective To explore the role of sulodexide (SDX) in neointimal hyperplasia of arteriovenous fistulas (AVFs) in chronic kidney disease (CKD) rats and its possible mechanism. Methods A total of 18 male rats (weighing 300±50 g) were randomly and equal
Externí odkaz:
https://doaj.org/article/bce9ed08bbe84d71856df2b0eed134f1
Autor:
Yin Wang, Xiao-mei Huang, Yi Zhang, Jingjing Li, Jun Li, Zheng Ye, Yu Peng, Xian-jin Zhang, Na Tang, Wen-wen Qiu, Li Xu
Publikováno v:
Renal Failure, Vol 46, Iss 1 (2024)
This study aimed to investigate ultrasound features of arteriovenous fistula stenosis and their relationship with primary patency after percutaneous transluminal angioplasty (post-intervention primary patency) and compare this classification with tha
Externí odkaz:
https://doaj.org/article/f96756e0cc38419da69abca0f0f1e818
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 177, Iss , Pp 117022- (2024)
Background: The transplantation of endothelial progenitor cells (EPCs) has been shown to reduce neointimal hyperplasia following arterial injury. However, the efficacy of this approach is hampered by limited homing of EPCs to the injury site. Additio
Externí odkaz:
https://doaj.org/article/0f4f096695e9496392155a3985cfa015
Publikováno v:
Materials Today Bio, Vol 27, Iss , Pp 101133- (2024)
In-stent restenosis (ISR), caused by aggressive vascular smooth muscle cell (VSMC) proliferation, is a serious complication of stenting. Therefore, developing therapeutic approaches that target VSMC inhibition is imperative. Our previous study showed
Externí odkaz:
https://doaj.org/article/6aea5ac12f9d4b7f9f753f136a1d783a
Publikováno v:
Cellular & Molecular Biology Letters, Vol 29, Iss 1, Pp 1-19 (2024)
Abstract Neointimal hyperplasia is a pathological vascular remodeling caused by abnormal proliferation and migration of subintimal vascular smooth muscle cells (VSMCs) following intimal injury. There is increasing evidence that tRNA-derived small RNA
Externí odkaz:
https://doaj.org/article/ca6338f3bee14f67b6944fd2a1e2e8e1
Publikováno v:
Frontiers in Cardiovascular Medicine, Vol 11 (2024)
We have reviewed the development and current status of therapies based on exposure to non-ionizing radiation (with a photon energy less than 10 eV) aimed at suppressing the venous neointimal hyperplasia, and consequentially at avoiding stenosis in ar
Externí odkaz:
https://doaj.org/article/f1cdf005e4b04ac4bcf6fbc1d6a6c1d4
Publikováno v:
Biochemistry and Biophysics Reports, Vol 36, Iss , Pp 101567- (2023)
Background: Ischemia‒reperfusion (IR) is a pathological process that causes secondary damage to blood vessels. However, whether IR can further worsen neointima formation after balloon injury and the detailed mechanism are unclear. Methods: An in vi
Externí odkaz:
https://doaj.org/article/2040d1073b6c4e0aa67c323e203b743e
Autor:
Ke Peng, Mingliang Wang, Jun Wang, Qiang Wang, De Li, Xiongshan Sun, Yongjian Yang, Dachun Yang
Publikováno v:
Clinical and Experimental Hypertension, Vol 45, Iss 1 (2023)
Background In-stent restenosis hardly limits the therapeutic effect of the percutaneous vascular intervention. Although the restenosis is significantly ameliorated after the application of new drug-eluting stents, the incidence of restenosis remains
Externí odkaz:
https://doaj.org/article/436eaee6b549408ab17ca11455c472d9
Publikováno v:
Clinical and Experimental Hypertension, Vol 45, Iss 1 (2023)
Objective Neointimal hyperplasia is the primary mechanism underlying atherosclerosis and restenosis after percutaneous coronary intervention. Ketogenic diet (KD) exerts beneficial effects in various diseases, but whether it could serve as non-drug th
Externí odkaz:
https://doaj.org/article/38155f080a234612a30a48e4c5e080f3
Autor:
Guangzheng Shi, Xinran Tong, Weihong Sun, Zilong Fang, Wendong Chen, Gonghao Jiang, Peili Zhang, Qun Li
Publikováno v:
Frontiers in Bioscience-Landmark, Vol 29, Iss 5, p 173 (2024)
Background: Neointimal hyperplasia (NIH) is the pathological basis of vascular injury disease. Vascular cells are the dominant cells in the process of NIH, but the extent of heterogeneity amongst them is still unclear. Methods: A mouse model of NIH w
Externí odkaz:
https://doaj.org/article/3f1ae9ba98284d139a5fa590eaf4e18a