Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Neila Benromdhane"'
Autor:
Asma Labidi, Héla Baccouche, Monia Fekih, Sonia Mahjoub, Nadia BenMustapha, Meriem Serghini, Neila BenRomdhane, Jalel Boubaker
Publikováno v:
Annals of Hepatology, Vol 18, Iss 4, Pp 627-632 (2019)
Introduction and Objectives: For long, bleeding in cirrhotic patients has been associated with acquired coagulation disorders. The aim of our study was to investigate the impact of acquired coagulation disorders on bleeding risk in cirrhotic patients
Externí odkaz:
https://doaj.org/article/496402f55b2a4f22baf375f8363902bd
Autor:
H. Baccouche, Meriem Serghini, Nadia BenMustapha, Neila Benromdhane, Monia Fekih, A. Labidi, Sonia Mahjoub, Jalel Boubaker
Publikováno v:
Annals of Hepatology, Vol 18, Iss 4, Pp 627-632 (2019)
Introduction and Objectives For long, bleeding in cirrhotic patients has been associated with acquired coagulation disorders. The aim of our study was to investigate the impact of acquired coagulation disorders on bleeding risk in cirrhotic patients.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0ac21c6d8ee961489e2776e86ae50e98
https://pubmed.ncbi.nlm.nih.gov/31097395
https://pubmed.ncbi.nlm.nih.gov/31097395
Autor:
Imen Moumni, Amine Zorai, Neila Benromdhane, Salem Abbes, Dorra Chaouechi, Sonia Mahjoub, Ikbel Mosbahi
Publikováno v:
Hemoglobin. 38:88-90
We describe a new δ-globin variant, Hb A2-Tunis [δ46(CD5)Gly → Glu; HBD: c.140G>A]. This hemoglobin (Hb) variant displayed a faster electrophoretic mobility than normal Hb A2 and was expressed at 3.2%. The molecular defect was characterized by DN
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::59d0e79004149f1325a11b0477510b25
https://doi.org/10.3109/03630269.2013.872123
https://doi.org/10.3109/03630269.2013.872123
Autor:
Imen, Moumni, Amine, Zorai, Sonia, Mahjoub, Ikbel, Mosbahi, Dorra, Chaouechi, Neila, Benromdhane, Salem, Abbes
Publikováno v:
Hemoglobin. 38(2)
We describe a new δ-globin variant, Hb A2-Tunis [δ46(CD5)Gly → Glu; HBD: c.140GA]. This hemoglobin (Hb) variant displayed a faster electrophoretic mobility than normal Hb A2 and was expressed at 3.2%. The molecular defect was characterized by DNA
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::cf5cab602c4e99d7267244ecc2457639
https://pubmed.ncbi.nlm.nih.gov/24471655
https://pubmed.ncbi.nlm.nih.gov/24471655