Zobrazeno 1 - 10
of 27
pro vyhledávání: '"Neil C. Davis"'
Autor:
Neil C. Davis, Emil L. Smith
Publikováno v:
Methods of Biochemical Analysis, Volume 2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8b39b85e786d6aa7e97ea94d54ca4ce6
https://doi.org/10.1002/9780470110188.ch8
https://doi.org/10.1002/9780470110188.ch8
Autor:
Neil C. Davis, Emil L. Smith
Publikováno v:
Journal of Biological Chemistry. 200:373-384
Publikováno v:
Journal of Clinical Investigation. 38:14-20
Publikováno v:
Journal of Clinical Investigation. 52:896-904
Membranoproliferative nephritis in children is frequently associated with a hypocomplementemia produced at least in part by C3 breakdown mediated by a circulating anticomplementary factor known as C3 nephritic factor (C3NeF). C3 breakdown by this fac
Autor:
Emil L. Smith, Neil C. Davis
Publikováno v:
Journal of Biological Chemistry. 224:261-275
Publikováno v:
The Journal of Pediatrics. 67:1089-9112
Of 24 patients with nephritis of duration over one year, 17, consistently, over long periods, had normal levels of serum β 1C -globulin and 7, consistently, had reduced levels. Levels of serum complement were normal or reduced in parallel with the
Publikováno v:
The Journal of Immunology. 96:650-658
Summary Four distinct antigenic determinant groups have been found to be associated with β1C-globulin. Antigen A is present on β1A and antigen D is on a second breakdown product of β1C, designated α2D. Both are also present on β1C. Another antig
Publikováno v:
Kidney International. 3(2):116-125
Although Ounn [1] in 1914 first observed the low serum complement levels in acute post-streptococcal nephritis, it was Kellett [2] who in 1935 first hypothesized that the bypocomplementemia was the result of an antigen-antibody reaction occurring in
Publikováno v:
Journal of Clinical Investigation. 46:539-548
Evidence has been obtained for the presence in vivo of alpha(2D)-globulin, a breakdown product of serum beta(1C)-globulin, in patients with acute and persistent hypocomplementemic glomerulonephritis. The protein has been identified by immunoelectroph